Vascular adhesion molecules in atherosclerosis.
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[1] G. Getz,et al. Site Specificity of Atherosclerosis: Site-Selective Responses to Atherosclerotic Modulators , 2004, Arteriosclerosis, thrombosis, and vascular biology.
[2] L. Lindbom,et al. Importance of Primary Capture and L-Selectin–Dependent Secondary Capture in Leukocyte Accumulation in Inflammation and Atherosclerosis in Vivo , 2001, The Journal of experimental medicine.
[3] D. Broide,et al. IL-5 links adaptive and natural immunity specific for epitopes of oxidized LDL and protects from atherosclerosis. , 2004, The Journal of clinical investigation.
[4] R. Hynes,et al. Lymphoid cells recognize an alternatively spliced segment of fibronectin via the integrin receptor alpha 4 beta 1. , 1990, Cell.
[5] W. Daniel,et al. Emergence of dendritic cells in rupture-prone regions of vulnerable carotid plaques. , 2004, Atherosclerosis.
[6] J. Witztum,et al. Innate and acquired immunity in atherogenesis , 2002, Nature Medicine.
[7] A. Freemont,et al. A Comparative Study of Neovascularisation in Atherosclerotic Plaques Using CD31, CD105 and TGFβ1 , 2006, Pathobiology.
[8] Andreas Schober,et al. Circulating activated platelets exacerbate atherosclerosis in mice deficient in apolipoprotein E , 2003, Nature Medicine.
[9] Thomas Christen,et al. Connexin37 protects against atherosclerosis by regulating monocyte adhesion , 2006, Nature Medicine.
[10] Hansson,et al. Detection of B Cells and Proinflammatory Cytokines in Atherosclerotic Plaques of Hypercholesterolaemic Apolipoprotein E Knockout Mice , 1999, Scandinavian journal of immunology.
[11] K. Preissner,et al. The Junctional Adhesion Molecule-C Promotes Neutrophil Transendothelial Migration in Vitro and in Vivo* , 2004, Journal of Biological Chemistry.
[12] C. Cabañas,et al. Minimally modified low-density lipoprotein induces monocyte adhesion to endothelial connecting segment-1 by activating beta1 integrin. , 1999, The Journal of clinical investigation.
[13] G. Hansson,et al. Adhesion of Activated T Lymphocytes to Vascular Smooth Muscle Cells and Dermal Fibroblasts is Mediated by β1‐ and β2‐Integrins , 1992 .
[14] K. Ley,et al. Direct demonstration of P-selectin- and VCAM-1-dependent mononuclear cell rolling in early atherosclerotic lesions of apolipoprotein E-deficient mice. , 1999, Circulation research.
[15] P. Burger,et al. Platelet P-selectin facilitates atherosclerotic lesion development. , 2003, Blood.
[16] Peihong Ma,et al. Endothelial expression of E-selectin is induced by the platelet-specific chemokine platelet factor 4 through LRP in an NF-kappaB-dependent manner. , 2005, Blood.
[17] R. McEver. Selectins: lectins that initiate cell adhesion under flow. , 2002, Current opinion in cell biology.
[18] T. Kupper,et al. CD43 is a ligand for E-selectin on CLA+ human T cells. , 2006, Blood.
[19] Elena Galkina,et al. Lymphocyte recruitment into the aortic wall before and during development of atherosclerosis is partially L-selectin dependent , 2006, The Journal of experimental medicine.
[20] T. Kita,et al. P-selectin and vascular cell adhesion molecule-1 are focally expressed in aortas of hypercholesterolemic rabbits before intimal accumulation of macrophages and T lymphocytes. , 1997, Arteriosclerosis, thrombosis, and vascular biology.
[21] K. Ramana,et al. Inhibition of aldose reductase attenuates TNF‐α‐induced expression of adhesion molecules in endothelial cells , 2004, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[22] Aldons J. Lusis,et al. Atherosclerosis : Vascular biology , 2000 .
[23] 松本 真典. CD43 functions as a ligand for E-selectin on activated T cells , 2007 .
[24] Y. Bobryshev,et al. Ultrastructural recognition of cells with dendritic cell morphology in human aortic intima. Contacting interactions of Vascular Dendritic Cells in athero-resistant and athero-prone areas of the normal aorta. , 1995, Archives of histology and cytology.
[25] P. Libby,et al. Apolipoprotein CIII Induces Expression of Vascular Cell Adhesion Molecule-1 in Vascular Endothelial Cells and Increases Adhesion of Monocytic Cells , 2006, Circulation.
[26] P. Newman,et al. Signal transduction pathways mediated by PECAM-1: new roles for an old molecule in platelet and vascular cell biology. , 2003, Arteriosclerosis, thrombosis, and vascular biology.
[27] E. Yeh,et al. Inhibition of alpha4 integrin and ICAM-1 markedly attenuate macrophage homing to atherosclerotic plaques in ApoE-deficient mice. , 1998, Circulation.
[28] F. Luscinskas,et al. The 130-kDa glycoform of CD43 functions as an E-selectin ligand for activated Th1 cells in vitro and in delayed-type hypersensitivity reactions in vivo. , 2007, The Journal of investigative dermatology.
[29] G. Hansson,et al. T lymphocytes in human atherosclerotic plaques are memory cells expressing CD45RO and the integrin VLA-1. , 1992, Arteriosclerosis and thrombosis : a journal of vascular biology.
[30] Timothy A. Springer,et al. Adhesion receptors of the immune system , 1990, Nature.
[31] J. Witztum,et al. The role of oxidized LDL in atherosclerosis. , 1991, Advances in experimental medicine and biology.
[32] D. Haskard,et al. TNF-α, IL-4, and IFN-γ Regulate Differential Expression of P- and E-Selectin Expression by Porcine Aortic Endothelial Cells1 , 2000, The Journal of Immunology.
[33] M. Schaller,et al. Platelets Recruit Human Dendritic Cells Via Mac-1/JAM-C Interaction and Modulate Dendritic Cell Function In Vitro , 2007, Arteriosclerosis, thrombosis, and vascular biology.
[34] E. Butcher,et al. Lymphocyte trafficking and regional immunity. , 1999, Advances in immunology.
[35] M. Lenter,et al. The E-selectin-ligand ESL-1 is a variant of a receptor for fibroblast growth factor , 1995, Nature.
[36] M. Cybulsky,et al. A major role for VCAM-1, but not ICAM-1, in early atherosclerosis. , 2001, The Journal of clinical investigation.
[37] David A. Schultz,et al. A mechanosensory complex that mediates the endothelial cell response to fluid shear stress , 2005, Nature.
[38] U. Andersson,et al. Cytokine expression in advanced human atherosclerotic plaques: dominance of pro-inflammatory (Th1) and macrophage-stimulating cytokines. , 1999, Atherosclerosis.
[39] R. Germain,et al. A novel lymphocyte function-associated antigen (LFA-1): cellular distribution, quantitative expression, and structure. , 1981, Journal of immunology.
[40] Peter Libby,et al. The immune response in atherosclerosis: a double-edged sword , 2006, Nature Reviews Immunology.
[41] J. Gorski,et al. PECAM-1 (CD31) cloning and relation to adhesion molecules of the immunoglobulin gene superfamily. , 1990, Science.
[42] J. Mehta,et al. Transforming Growth Factor-β1 Modulates Oxidatively Modified LDL–Induced Expression of Adhesion Molecules: Role of LOX-1 , 2001 .
[43] J. Borén,et al. Subendothelial retention of atherogenic lipoproteins in early atherosclerosis , 2002, Nature.
[44] M. Elices,et al. VCAM-1 on activated endothelium interacts with the leukocyte integrin VLA-4 at a site distinct from the VLA-4/Fibronectin binding site , 1990, Cell.
[45] C. Parkos,et al. The JAM family of proteins. , 2005, Advanced drug delivery reviews.
[46] J. Phillips,et al. α4β1 Integrin (VLA-4) Blockade Attenuates both Early and Late Leukocyte Recruitment and Neointimal Growth following Carotid Injury in Apolipoprotein E (–/–) Mice , 2004, Journal of Vascular Research.
[47] Elisabetta Dejana,et al. Importance of junctional adhesion molecule-A for neointimal lesion formation and infiltration in atherosclerosis-prone mice. , 2005, Arteriosclerosis, thrombosis, and vascular biology.
[48] R. Lal,et al. Gap junctional communication between vascular cells. Induction of connexin43 messenger RNA in macrophage foam cells of atherosclerotic lesions. , 1993, The American journal of pathology.
[49] Y. Bobryshev,et al. Monocyte recruitment and foam cell formation in atherosclerosis. , 2006, Micron.
[50] A. Zernecke,et al. Involvement of JAM-A in Mononuclear Cell Recruitment on Inflamed or Atherosclerotic Endothelium: Inhibition by Soluble JAM-A , 2005, Arteriosclerosis, thrombosis, and vascular biology.
[51] A. Zalewski,et al. Lipoprotein-associated phospholipase A2 as a target of therapy , 2005, Current opinion in lipidology.
[52] David Zurakowski,et al. Inhibition of plaque neovascularization reduces macrophage accumulation and progression of advanced atherosclerosis , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[53] M. Aurrand-Lions,et al. Adhesion mechanisms regulating the migration of monocytes , 2004, Nature Reviews Immunology.
[54] Qingbo Xu,et al. Atherosclerosis, autoimmunity, and vascular‐associated lymphoid tissue , 1997, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[55] Richard O. Hynes,et al. Lymphoid cells recognize an alternatively spliced segment of fibronectin via the integrin receptor α 4 β 1 , 1990, Cell.
[56] R. Alon,et al. Immune cell migration in inflammation: present and future therapeutic targets , 2005, Nature Immunology.
[57] A. Zernecke,et al. JAM-1 is a ligand of the β2 integrin LFA-1 involved in transendothelial migration of leukocytes , 2002, Nature Immunology.
[58] M. Davies,et al. The expression of the adhesion molecules ICAM‐1, VCAM‐1, PECAM, and E‐selectin in human atherosclerosis , 1993, The Journal of pathology.
[59] B. Zinselmeyer,et al. Images in cardiovascular medicine. Multiphoton microscopy for 3-dimensional imaging of lymphocyte recruitment into apolipoprotein-E-deficient mouse carotid artery. , 2007, Circulation.
[60] T. Springer. Traffic signals for lymphocyte recirculation and leukocyte emigration: The multistep paradigm , 1994, Cell.
[61] M. Cybulsky,et al. Targeted disruption of the murine VCAM1 gene: essential role of VCAM-1 in chorioallantoic fusion and placentation. , 1995, Genes & development.
[62] W. H. Evans,et al. Gap junctions: structure and function (Review) , 2002, Molecular membrane biology.
[63] M. Salifu,et al. The F11 receptor (F11R/JAM-A) in atherothrombosis: Overexpression of F11R in atherosclerotic plaques , 2007, Thrombosis and Haemostasis.
[64] M. Cybulsky,et al. Adhesion of Monocytes to Arterial Endothelium and Initiation of Atherosclerosis Are Critically Dependent on Vascular Cell Adhesion Molecule-1 Gene Dosage , 2001, Arteriosclerosis, thrombosis, and vascular biology.
[65] K. Ley,et al. P-selectin Glycoprotein Ligand-1 Mediates L-Selectin–dependent Leukocyte Rolling in Venules , 2003, The Journal of experimental medicine.
[66] V. Orlova,et al. Junctional adhesion molecule-C regulates vascular endothelial permeability by modulating VE-cadherin–mediated cell–cell contacts , 2006, The Journal of experimental medicine.
[67] F. Mach,et al. Leukocyte recruitment in atherosclerosis: Potential targets for therapeutic approaches? , 2006, Cellular and Molecular Life Sciences CMLS.
[68] K. Ley,et al. Role of vascular cell adhesion molecule-1 and fibronectin connecting segment-1 in monocyte rolling and adhesion on early atherosclerotic lesions. , 2000, Circulation research.
[69] K. Williams,et al. Atherosclerosis--an inflammatory disease. , 1999, The New England journal of medicine.
[70] T. Kinashi. Integrin regulation of lymphocyte trafficking: lessons from structural and signaling studies. , 2007, Advances in immunology.
[71] K. Ley,et al. Spleen tyrosine kinase Syk is necessary for E-selectin-induced alpha(L)beta(2) integrin-mediated rolling on intercellular adhesion molecule-1. , 2007, Immunity.
[72] C. Alpers,et al. Neovascular expression of E-selectin, intercellular adhesion molecule-1, and vascular cell adhesion molecule-1 in human atherosclerosis and their relation to intimal leukocyte content. , 1996, Circulation.
[73] G. Garin,et al. Flow-mediated signaling modulates endothelial cell phenotype. , 2006, Endothelium : journal of endothelial cell research.
[74] T. Mayadas,et al. Absence of P-selectin delays fatty streak formation in mice. , 1997, The Journal of clinical investigation.
[75] A. Beaudet,et al. P-Selectin or Intercellular Adhesion Molecule (Icam)-1 Deficiency Substantially Protects against Atherosclerosis in Apolipoprotein E–Deficient Mice , 2000, The Journal of experimental medicine.
[76] M. Cybulsky,et al. Getting to the site of inflammation: the leukocyte adhesion cascade updated , 2007, Nature Reviews Immunology.
[77] D. Wagner,et al. Prominent role of P-selectin in the development of advanced atherosclerosis in ApoE-deficient mice. , 2000, Circulation.
[78] Elisabetta Dejana,et al. Endothelial cell-to-cell junctions: molecular organization and role in vascular homeostasis. , 2004, Physiological reviews.
[79] D. Haskard,et al. TNF-alpha, IL-4, and IFN-gamma regulate differential expression of P- and E-selectin expression by porcine aortic endothelial cells. , 2000, Journal of immunology.
[80] K. Ley,et al. Role of platelets in the development of atherosclerosis. , 2004, Trends in cardiovascular medicine.
[81] E. Butcher. Leukocyte-endothelial cell recognition: Three (or more) steps to specificity and diversity , 1991, Cell.
[82] N. Leitinger,et al. Oxidized Phospholipids Alter Vascular Connexin Expression, Phosphorylation, and Heterocellular Communication , 2006, Arteriosclerosis, thrombosis, and vascular biology.
[83] Steffen Jung,et al. Blood monocytes consist of two principal subsets with distinct migratory properties. , 2003, Immunity.
[84] N. Hogg,et al. Mechanisms contributing to the activity of integrins on leukocytes , 2002, Immunological reviews.
[85] A. Beaudet,et al. Absence of P-Selectin, but Not Intercellular Adhesion Molecule-1, Attenuates Neointimal Growth After Arterial Injury in Apolipoprotein E–Deficient Mice , 2001, Circulation.
[86] F. Tacke,et al. Monocyte subsets differentially employ CCR2, CCR5, and CX3CR1 to accumulate within atherosclerotic plaques. , 2007, The Journal of clinical investigation.
[87] C. Weber,et al. Platelets as Immune Cells: Bridging Inflammation and Cardiovascular Disease , 2007, Circulation research.
[88] P. Frenette,et al. Complete identification of E-selectin ligands on neutrophils reveals distinct functions of PSGL-1, ESL-1, and CD44. , 2007, Immunity.
[89] C. Gleissner,et al. Effects of Native and Modified Low-Density Lipoproteins on Monocyte Recruitment in Atherosclerosis , 2007, Hypertension.
[90] María Yáñez-Mó,et al. Dynamic interaction of VCAM-1 and ICAM-1 with moesin and ezrin in a novel endothelial docking structure for adherent leukocytes , 2002, The Journal of cell biology.
[91] P. Libby,et al. An atherogenic diet rapidly induces VCAM-1, a cytokine-regulatable mononuclear leukocyte adhesion molecule, in rabbit aortic endothelium. , 1993, Arteriosclerosis and thrombosis : a journal of vascular biology.
[92] W. Muller. Leukocyte-endothelial-cell interactions in leukocyte transmigration and the inflammatory response. , 2003, Trends in immunology.
[93] J. Bonnet,et al. Regulation of vascular cell adhesion molecule-1 and intercellular adhesion molecule-1 in human vascular smooth muscle cells. , 1994, Circulation research.
[94] S. Cunningham,et al. JAM2 interacts with alpha4beta1. Facilitation by JAM3. , 2002, The Journal of biological chemistry.
[95] R. Vollandt,et al. The Lamina Adventitia Is the Major Site of Immune Cell Accumulation in Standard Chow-Fed Apolipoprotein E–Deficient Mice , 2005, Arteriosclerosis, thrombosis, and vascular biology.
[96] P. Lelkes,et al. Gene expression profiling of human aortic endothelial cells exposed to disturbed flow and steady laminar flow. , 2002, Physiological genomics.
[97] M. Cybulsky,et al. Endothelial expression of a mononuclear leukocyte adhesion molecule during atherogenesis. , 1991, Science.
[98] P. Frenette,et al. CD44 is a physiological E-selectin ligand on neutrophils , 2005, The Journal of experimental medicine.
[99] G. Bondjers,et al. Regional Accumulations of T Cells, Macrophages, and Smooth Muscle Cells in the Human Atherosclerotic Plaque , 1986, Arteriosclerosis.
[100] G. Owens,et al. Smooth Muscle–Targeted Knockout of Connexin43 Enhances Neointimal Formation in Response to Vascular Injury , 2007, Arteriosclerosis, thrombosis, and vascular biology.
[101] R. Poston,et al. Increase in the adhesion molecule P-selectin in endothelium overlying atherosclerotic plaques. Coexpression with intercellular adhesion molecule-1. , 1994, The American journal of pathology.
[102] F. Mach,et al. Reduced Connexin43 Expression Inhibits Atherosclerotic Lesion Formation in Low-Density Lipoprotein Receptor–Deficient Mice , 2003, Circulation.
[103] Richard O Hynes,et al. Integrins Bidirectional, Allosteric Signaling Machines , 2002, Cell.
[104] K. Ley,et al. Adhesion molecules and atherogenesis. , 2001, Acta physiologica Scandinavica.
[105] Peter Libby,et al. Innate and Adaptive Immunity in the Pathogenesis of Atherosclerosis , 2002, Circulation research.
[106] R. Alexander,et al. Modified low density lipoprotein and its constituents augment cytokine-activated vascular cell adhesion molecule-1 gene expression in human vascular endothelial cells. , 1995, The Journal of clinical investigation.
[107] C. Carman,et al. Structural basis of integrin regulation and signaling. , 2007, Annual review of immunology.
[108] A. Zwinderman,et al. A novel functional polymorphism in the PECAM-1 gene (53G>A) is associated with progression of atherosclerosis in the LOCAT and REGRESS studies. , 2003, Atherosclerosis.
[109] M. Cybulsky,et al. Low-grade chronic inflammation in regions of the normal mouse arterial intima predisposed to atherosclerosis , 2006, The Journal of experimental medicine.
[110] E. Yeh,et al. Inhibition of α4 Integrin and ICAM-1 Markedly Attenuate Macrophage Homing to Atherosclerotic Plaques in ApoE-Deficient Mice , 1998 .
[111] D. Parums,et al. Demonstration of immunoglobulin in the neighbourhood of advanced atherosclerotic plaques. , 1981, Atherosclerosis.
[112] G. le Naour,et al. Atherogenic properties of LDL particles modified by human group X secreted phospholipase A2 on human endothelial cell function , 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[113] S. Hemmerich,et al. Vascular Endothelial Junction-associated Molecule, a Novel Member of the Immunoglobulin Superfamily, Is Localized to Intercellular Boundaries of Endothelial Cells* , 2000, The Journal of Biological Chemistry.
[114] K. Pritchard,et al. Native LDL increases endothelial cell adhesiveness by inducing intercellular adhesion molecule-1. , 1996, Arteriosclerosis, thrombosis, and vascular biology.
[115] S. Rosen,et al. Sulfated L-selectin ligands as a therapeutic target in chronic inflammation. , 2006, Trends in immunology.
[116] E. Oviedo-Orta,et al. GAP JUNCTION INTERCELLULAR COMMUNICATION DURING LYMPHOCYTE TRANSENDOTHELIAL MIGRATION , 2002 .
[117] D. Wagner,et al. Leukocyte-endothelium adhesion molecules in atherosclerosis. , 1998, The Journal of laboratory and clinical medicine.
[118] V. Kuchroo,et al. TH-17 cells in the circle of immunity and autoimmunity , 2007, Nature Immunology.
[119] Kazuko Kobayashi,et al. Oxidative modification of low-density lipoprotein and immune regulation of atherosclerosis. , 2006, Progress in lipid research.
[120] K. Ley,et al. Selectins in T-cell recruitment to non-lymphoid tissues and sites of inflammation , 2004, Nature Reviews Immunology.
[121] E. Dejana,et al. Junctional Adhesion Molecule, a Novel Member of the Immunoglobulin Superfamily That Distributes at Intercellular Junctions and Modulates Monocyte Transmigration , 1998, The Journal of cell biology.
[122] R. Hynes,et al. The combined role of P- and E-selectins in atherosclerosis. , 1998, The Journal of clinical investigation.
[123] M. Stemerman,et al. In Vivo Low-Density Lipoprotein Exposure Induces Intercellular Adhesion Molecule-1 and Vascular Cell Adhesion Molecule-1 Correlated With Activator Protein-1 Expression , 2006, Arteriosclerosis, thrombosis, and vascular biology.
[124] E. Chavakis,et al. The role of junctional adhesion molecule‐C (JAM‐C) in oxidized LDL‐mediated leukocyte recruitment , 2005, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[125] T. Springer,et al. The structure of immunoglobulin superfamily domains 1 and 2 of MAdCAM-1 reveals novel features important for integrin recognition. , 1998, Structure.
[126] E. Rodríguez,et al. HUVECs from newborns with a strong family history of myocardial infarction overexpress adhesion molecules and react abnormally to stimulating agents , 2005, Clinical and experimental immunology.
[127] P. Frenette,et al. PSGL-1 participates in E-selectin-mediated progenitor homing to bone marrow: evidence for cooperation between E-selectin ligands and alpha4 integrin. , 2003, Blood.
[128] H. Jongsma,et al. TUMOUR NECROSIS FACTOR α ALTERS THE EXPRESSION OF CONNEXIN43, CONNEXIN40, AND CONNEXIN37, IN HUMAN UMBILICAL VEIN ENDOTHELIAL CELLS , 1998 .
[129] E. Eriksson. Leukocyte recruitment to atherosclerotic lesions, a complex web of dynamic cellular and molecular interactions. , 2003, Current drug targets. Cardiovascular & haematological disorders.
[130] P. Libby,et al. Ly-6Chi monocytes dominate hypercholesterolemia-associated monocytosis and give rise to macrophages in atheromata. , 2007, The Journal of clinical investigation.
[131] K. Ley,et al. Critical Role of Platelet P-Selectin in the Response to Arterial Injury in Apolipoprotein-E–Deficient Mice , 2004, Arteriosclerosis, thrombosis, and vascular biology.
[132] J. Goldstein,et al. Lowering LDL--Not Only How Low, But How Long? , 2006, Science.
[133] A. Beaudet,et al. Deficiency of inflammatory cell adhesion molecules protects against atherosclerosis in mice. , 1997, Arteriosclerosis, thrombosis, and vascular biology.
[134] N. Leitinger. Oxidized phospholipids as modulators of inflammation in atherosclerosis , 2003, Current opinion in lipidology.
[135] K. Moore,et al. Chemokine CXCL10 Promotes Atherogenesis by Modulating the Local Balance of Effector and Regulatory T Cells , 2006, Circulation.
[136] Gini Williams. Holding the Patient , 2001, Annals of the New York Academy of Sciences.
[137] T. Lüscher,et al. Reduced Connexin43 Expression Limits Neointima Formation After Balloon Distension Injury in Hypercholesterolemic Mice , 2006, Circulation.
[138] K. Moore,et al. Reduced atherosclerosis in MyD88-null mice links elevated serum cholesterol levels to activation of innate immunity signaling pathways , 2004, Nature Medicine.
[139] T. Christen,et al. Connexins in leukocytes: shuttling messages? , 2004, Cardiovascular research.
[140] Thomas N. Sato,et al. Increased DC trafficking to lymph nodes and contact hypersensitivity in junctional adhesion molecule-A-deficient mice. , 2004, The Journal of clinical investigation.
[141] A. Carr,et al. Oxidation of LDL by myeloperoxidase and reactive nitrogen species: reaction pathways and antioxidant protection. , 2000, Arteriosclerosis, thrombosis, and vascular biology.
[142] M. Aurrand-Lions,et al. Junctional adhesion molecule-2 (JAM-2) promotes lymphocyte transendothelial migration. , 2002, Blood.
[143] G. Frantz,et al. Vascular Endothelial-Junctional Adhesion Molecule (VE-JAM)/JAM 2 Interacts with T, NK, and Dendritic Cells Through JAM 3 , 2002, The Journal of Immunology.
[144] J. Bordet,et al. Oxidized low-density lipoprotein induces the expression of P-selectin (GMP140/PADGEM/CD62) on human endothelial cells. , 1995, The Biochemical journal.
[145] E. Kunkel,et al. Role of primary and secondary capture for leukocyte accumulation in vivo. , 1998, Circulation research.
[146] J. Cyster,et al. Detection of a sulfotransferase (HEC-GlcNAc6ST) in high endothelial venules of lymph nodes and in high endothelial venule-like vessels within ectopic lymphoid aggregates: relationship to the MECA-79 epitope. , 2004, The American journal of pathology.
[147] R. Ross,et al. Upregulation of VCAM-1 and ICAM-1 at atherosclerosis-prone sites on the endothelium in the ApoE-deficient mouse. , 1998, Arteriosclerosis, thrombosis, and vascular biology.