P53 mediates ceramide-induced apoptosis in SKN-SH cells
暂无分享,去创建一个
Y. Suh | J. Bonventre | Myoung-Woo Lee | H. Chae | J. Bach | K. Kim | W. Lee | Sung Su Kim | Young-Min Jung
[1] M. Nikolova-Karakashian,et al. Activation of sphingolipid turnover and chronic generation of ceramide and sphingosine in liver during aging , 2000, Mechanisms of Ageing and Development.
[2] E. Yonish-Rouach,et al. Mechanisms of p53-induced apoptosis: in search of genes which are regulated during p53-mediated cell death. , 1998, Toxicology letters.
[3] G. Dbaibo,et al. Distinct sites of action of Bcl-2 and Bcl-xL in the ceramide pathway of apoptosis. , 1998, The Biochemical journal.
[4] A. Khar,et al. Selective involvement of caspase‐3 in ceramide induced apoptosis in AK‐5 tumor cells , 1998, FEBS letters.
[5] R. Kolesnick,et al. Signal transduction of stress via ceramide. , 1998, The Biochemical journal.
[6] G. Dbaibo,et al. p53-dependent ceramide response to genotoxic stress. , 1998, The Journal of clinical investigation.
[7] A. W. Murray,et al. Neurotrophic Factors Prevent Ceramide‐Induced Apoptosis Downstream of c‐Jun N‐Terminal Kinase Activation in PC12 Cells , 1998, Journal of neurochemistry.
[8] Y Agid,et al. Mitochondrial Free Radical Signal in Ceramide‐Dependent Apoptosis: A Putative Mechanism for Neuronal Death in Parkinson's Disease , 1997, Journal of neurochemistry.
[9] S. Kim,et al. Sphingomyelinase activity is enhanced in cerebral cortex of senescence-accelerated mouse-P/10 with advancing age. , 1997, Biochemical and biophysical research communications.
[10] J. Coggins,et al. The study of cell-death proteins in the outer mitochondrial membrane by chemical cross-linking. , 1997, The Biochemical journal.
[11] Y. Hannun,et al. Bcl‐2 antagonizes apoptotic cell death induced by two new ceramide analogues , 1997, FEBS letters.
[12] John Calvin Reed. Double identity for proteins of the Bcl-2 family , 1997, Nature.
[13] G. Laurent,et al. Influence of Bcl-2 overexpression on the ceramide pathway in daunorubicin-induced apoptosis of leukemic cells , 1997, Oncogene.
[14] M. Moskowitz,et al. Expression of a Dominant Negative Mutant of Interleukin-1β Converting Enzyme in Transgenic Mice Prevents Neuronal Cell Death Induced by Trophic Factor Withdrawal and Ischemic Brain Injury , 1997, The Journal of experimental medicine.
[15] A. Levine. p53, the Cellular Gatekeeper for Growth and Division , 1997, Cell.
[16] A. W. Murray,et al. Ceramide induces apoptosis in PC12 cells , 1997, FEBS letters.
[17] N. Miyasaka,et al. Ceramide induces apoptosis via CPP32 activation , 1996, FEBS letters.
[18] Junying Yuan,et al. Human ICE/CED-3 Protease Nomenclature , 1996, Cell.
[19] C. Borner,et al. Bcl-2 interrupts the ceramide-mediated pathway of cell death. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[20] M. Oren,et al. Cell type‐specific inhibition of p53‐mediated apoptosis by mdm2. , 1996, The EMBO journal.
[21] John Calvin Reed,et al. Proapoptotic Protein Bax Heterodimerizes with Bcl-2 and Homodimerizes with Bax via a Novel Domain (BH3) Distinct from BH1 and BH2 (*) , 1996, The Journal of Biological Chemistry.
[22] E. White,et al. The E1B 19K protein blocks apoptosis by interacting with and inhibiting the p53-inducible and death-promoting Bax protein. , 1996, Genes & development.
[23] Y. Agid,et al. Ceramide Induces Apoptosis in Cultured Mesencephalic Neurons , 1996, Journal of neurochemistry.
[24] A. Bielawska,et al. Role of Ceramide in Cellular Senescence (*) , 1995, The Journal of Biological Chemistry.
[25] S. Korsmeyer,et al. Bax-Deficient Mice with Lymphoid Hyperplasia and Male Germ Cell Death , 1995, Science.
[26] Z. Oltvai,et al. Multiple Bcl-2 family members demonstrate selective dimerizations with Bax. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[27] Seamus J. Martin,et al. Protease activation during apoptosis: Death by a thousand cuts? , 1995, Cell.
[28] V. Rotter,et al. Direct involvement of p53 in programmed cell death of oligodendrocytes. , 1995, The EMBO journal.
[29] S. Korsmeyer,et al. Bad, a heterodimeric partner for Bcl-xL and Bcl-2, displaces bax and promotes cell death , 1995, Cell.
[30] T. Graeber,et al. Hypoxia induces accumulation of p53 protein, but activation of a G1-phase checkpoint by low-oxygen conditions is independent of p53 status , 1994, Molecular and cellular biology.
[31] S. Korsmeyer,et al. Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell death , 1993, Cell.
[32] C. Purdie,et al. Thymocyte apoptosis induced by p53-dependent and independent pathways , 1993, Nature.
[33] P. W. Mesner,et al. Nerve growth factor withdrawal-induced cell death in neuronal PC12 cells resembles that in sympathetic neurons , 1992, The Journal of cell biology.
[34] M. M. Bradford. A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. , 1976, Analytical biochemistry.
[35] U. K. Laemmli,et al. Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4 , 1970, Nature.
[36] N. Thornberry. The caspase family of cysteine proteases. , 1997, British medical bulletin.
[37] T. Deckwerth,et al. Molecular mechanisms of developmental neuronal death. , 1993, Annual review of neuroscience.