Resistance exercise enhances the molecular signaling of mitochondrial biogenesis induced by endurance exercise in human skeletal muscle.
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Henrik Mascher | Eva Blomstrand | Li Wang | K. Sahlin | H. Mascher | E. Blomstrand | N. Psilander | Niklas Psilander | Kent Sahlin | Li Wang
[1] A. Jeukendrup,et al. The effects of replacing a portion of endurance training by explosive strength training on performance in trained cyclists , 2001, European Journal of Applied Physiology.
[2] L. Goodyear,et al. Exercise, MAPK, and NF-κB signaling in skeletal muscle , 2007 .
[3] P. Aagaard,et al. Effects of strength training on endurance capacity in top‐level endurance athletes , 2010, Scandinavian journal of medicine & science in sports.
[4] M. Hall,et al. TOR Signaling in Growth and Metabolism , 2006, Cell.
[5] R. Scarpulla,et al. PGC-1-Related Coactivator, a Novel, Serum-Inducible Coactivator of Nuclear Respiratory Factor 1-Dependent Transcription in Mammalian Cells , 2001, Molecular and Cellular Biology.
[6] L. Goodyear,et al. Exercise, MAPK, and NF-kappaB signaling in skeletal muscle. , 2007, Journal of applied physiology.
[7] D. Bishop,et al. The effects of strength training on endurance performance and muscle characteristics. , 1999, Medicine and science in sports and exercise.
[8] C. Foster,et al. Potential for strength and endurance training to amplify endurance performance. , 1988, Journal of applied physiology.
[9] A. Bigard,et al. Interaction between signalling pathways involved in skeletal muscle responses to endurance exercise , 2006, Pflügers Archiv.
[10] A. Bonen,et al. PGC-1alpha-mediated regulation of gene expression and metabolism: implications for nutrition and exercise prescriptions. , 2008, Applied physiology, nutrition, and metabolism = Physiologie appliquee, nutrition et metabolisme.
[11] G. Dimitriadis,et al. Acute and chronic effects of strenuous exercise on glucose metabolism in isolated, incubated soleus muscle of exercise-trained rats. , 1989, Acta physiologica Scandinavica.
[12] D. Chinkes,et al. Resistance exercise increases AMPK activity and reduces 4E‐BP1 phosphorylation and protein synthesis in human skeletal muscle , 2006, The Journal of physiology.
[13] S. Chien,et al. Mechanical stimuli and nutrients regulate rapamycin‐sensitive signaling through distinct mechanisms in skeletal muscle , 2006, Journal of cellular biochemistry.
[14] K. Sahlin,et al. Erratum to: Mitochondrial gene expression in elite cyclists: effects of high-intensity interval exercise , 2010, European Journal of Applied Physiology.
[15] K. Häkkinen,et al. Explosive‐strength training improves 5‐km running time by improving running economy and muscle power , 1999, Journal of applied physiology.
[16] Ronald J. Gutmann,et al. Recombination processes in doubly capped antimonide-based quaternary thin films , 1999 .
[17] S. B. Wilkinson,et al. Differential effects of resistance and endurance exercise in the fed state on signalling molecule phosphorylation and protein synthesis in human muscle , 2008, The Journal of physiology.
[18] W. Kraus,et al. PGC-1α mRNA expression is influenced by metabolic perturbation in exercising human skeletal muscle , 2004 .
[19] P. Neufer,et al. Substrate availability and transcriptional regulation of metabolic genes in human skeletal muscle during recovery from exercise. , 2005, Metabolism: clinical and experimental.
[20] G. Shulman,et al. The role of AMP‐activated protein kinase in mitochondrial biogenesis , 2006, The Journal of physiology.
[21] J. Hawley,et al. Consecutive bouts of diverse contractile activity alter acute responses in human skeletal muscle. , 2009, Journal of applied physiology.
[22] K. Sahlin,et al. Similar expression of oxidative genes after interval and continuous exercise. , 2009, Medicine and science in sports and exercise.
[23] Anthony Shield,et al. Early signaling responses to divergent exercise stimuli in skeletal muscle from well‐trained humans , 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[24] Jiandie D. Lin,et al. Suppression of Reactive Oxygen Species and Neurodegeneration by the PGC-1 Transcriptional Coactivators , 2006, Cell.
[25] I. Vogiatzis,et al. Resistance exercise-induced increase in muscle mass correlates with p70S6 kinase phosphorylation in human subjects , 2007, European Journal of Applied Physiology.
[26] J. Babraj,et al. Selective activation of AMPK‐PGC‐1α or PKB‐TSC2‐mTOR signaling can explain specific adaptive responses to endurance or resistance training‐like electrical muscle stimulation , 2005, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[27] Psilander Niklas,et al. Mitochondrial gene expression in elite cyclists: effects of high-intensity interval exercise , 2010, European Journal of Applied Physiology.
[28] D. O'Gorman,et al. Exercise intensity‐dependent regulation of peroxisome proliferator‐activated receptor γ coactivator‐1α mRNA abundance is associated with differential activation of upstream signalling kinases in human skeletal muscle , 2010, The Journal of physiology.
[29] J. P. McCoy,et al. The Mammalian Target of Rapamycin (mTOR) Pathway Regulates Mitochondrial Oxygen Consumption and Oxidative Capacity* , 2006, Journal of Biological Chemistry.
[30] W. Kraus,et al. PGC-1alpha mRNA expression is influenced by metabolic perturbation in exercising human skeletal muscle. , 2004, Journal of applied physiology.
[31] V. Mootha,et al. mTOR controls mitochondrial oxidative function through a YY1–PGC-1α transcriptional complex , 2007, Nature.
[32] P. Laursen,et al. Effect of Concurrent Resistance and Endurance Training on Physiologic and Performance Parameters of Well-Trained Endurance Cyclists , 2009, Journal of strength and conditioning research.
[33] J. Hawley. Molecular responses to strength and endurance training: are they incompatible? , 2009, Applied physiology, nutrition, and metabolism = Physiologie appliquee, nutrition et metabolisme.
[34] George A. Brooks,et al. Lactate sensitive transcription factor network in L6 cells: activation of MCT1 and mitochondrial biogenesis , 2007, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[35] F. Kadi,et al. The biology of satellite cells and telomeres in human skeletal muscle: effects of aging and physical activity , 2010, Scandinavian journal of medicine & science in sports.
[36] C. Lundby,et al. Relative workload determines exercise-induced increases in PGC-1alpha mRNA. , 2010, Medicine and science in sports and exercise.
[37] M. Gibala,et al. Brief intense interval exercise activates AMPK and p38 MAPK signaling and increases the expression of PGC-1alpha in human skeletal muscle. , 2009, Journal of applied physiology.
[38] J. Helgerud,et al. Maximal strength training improves running economy in distance runners. , 2008, Medicine and science in sports and exercise.
[39] D G Sale,et al. Interaction between concurrent strength and endurance training. , 1990, Journal of applied physiology.
[40] Y. Hellsten,et al. Calcium signalling in the regulation of PGC-1α, PDK4 and HKII mRNA expression , 2007, Biological chemistry.
[41] A. de Haan,et al. The muscle fiber type–fiber size paradox: hypertrophy or oxidative metabolism? , 2010, European Journal of Applied Physiology.
[42] K. Baar. Training for endurance and strength: lessons from cell signaling. , 2006, Medicine and science in sports and exercise.
[43] B. Spiegelman,et al. Transducer of regulated CREB-binding proteins (TORCs) induce PGC-1α transcription and mitochondrial biogenesis in muscle cells , 2006, Proceedings of the National Academy of Sciences.
[44] J. Blenis,et al. Inactivation of the Tuberous Sclerosis Complex-1 and -2 Gene Products Occurs by Phosphoinositide 3-Kinase/Akt-dependent and -independent Phosphorylation of Tuberin* , 2003, Journal of Biological Chemistry.
[45] J. Bergstrom. Percutaneous needle biopsy of skeletal muscle in physiological and clinical research. , 1975, Scandinavian journal of clinical and laboratory investigation.