Role of tissue plasminogen activator and plasminogen activator inhibitor polymorphism in myocardial infarction
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Ahmed Sadeque | N. Ahmed | M. Azam | R. Qamar | M. Ajmal | W. Ahmed | I. Saeed | Umar M Kaleem | Meera Malik | Amina Khan
[1] K. Nakashima,et al. [The Rotterdam study]. , 2011, Nihon rinsho. Japanese journal of clinical medicine.
[2] Jianjun Zhang,et al. Association of deoxyribonuclease I genetic polymorphisms with myocardial infarction in Han Chinese , 2009, Molecular Biology Reports.
[3] Scott M. Williams,et al. Male–female differences in the genetic regulation of t-PA and PAI-1 levels in a Ghanaian population , 2008, Human Genetics.
[4] R. Macko,et al. Microvascular tissue plasminogen activator is reduced in diabetic neuropathy , 2007, Neurology.
[5] S. Yusuf,et al. Risk factors for early myocardial infarction in South Asians compared with individuals in other countries. , 2007, JAMA.
[6] D. Newby,et al. Tissue plasminogen activator genetic polymorphisms do not influence tissue plasminogen activator release in patients with coronary heart disease , 2006, Journal of thrombosis and haemostasis : JTH.
[7] M. Castellano. Geographic ancestry, angiotensinogen gene polymorphism, and cardiovascular risk. , 2006, Hypertension.
[8] N. Chaturvedi,et al. Risk factors for non-fatal myocardial infarction in young South Asian adults , 2004, Heart.
[9] B. Peterlin,et al. 4G4G Genotype of PAI-1 Gene Promoter Polymorphism Is Not Associated with Myocardial Infarction in Caucasians with Type-2 Diabetes , 2003, Cardiology.
[10] M. Margaglione,et al. The plasminogen activator inhibitor‐1 −675 4G/5G genotype influences the risk of myocardial infarction associated with elevated plasma proinsulin and insulin concentrations in men from Europe: the HIFMECH Study , 2003, Journal of thrombosis and haemostasis : JTH.
[11] A. Sharrett,et al. Lack of association of the plasminogen activator inhibitor‐1 4G/5G promoter polymorphism with cardiovascular disease in the elderly , 2003, Journal of thrombosis and haemostasis : JTH.
[12] H. Izawa,et al. Prediction of the risk of myocardial infarction from polymorphisms in candidate genes. , 2002, The New England journal of medicine.
[13] K. Lewandrowski,et al. Cardiac markers for myocardial infarction. A brief review. , 2002, American journal of clinical pathology.
[14] H. Wolfe,et al. Kinetic profiles by topographic compartments in muscle-invasive transitional cell carcinomas of the bladder: role of TP53 and NF1 genes. , 2002, American journal of clinical pathology.
[15] T. Nakajima,et al. Linkage disequilibrium and haplotype analysis among eight novel single-nucleotide polymorphisms in the human tissue-type plasminogen activator (t-PA) gene , 2001, Journal of Human Genetics.
[16] J. Sambrook,et al. Molecular Cloning: A Laboratory Manual , 2001 .
[17] T. Spector,et al. The genetics of haemostasis: a twin study , 2001, The Lancet.
[18] C. Glueck,et al. The 4G/4G polymorphism of the hypofibrinolytic plasminogen activator inhibitor type 1 gene: an independent risk factor for serious pregnancy complications. , 2000, Metabolism: clinical and experimental.
[19] N. Wenger,et al. The role of the t-PA I/D and PAI-1 4G/5G polymorphisms in African-American adults with a diagnosis of myocardial infarction or venous thromboembolism. , 2000, Thrombosis research.
[20] J. Carlquist,et al. Lack of association of a common polymorphism of the plasminogen activator inhibitor-1 gene with coronary artery disease and myocardial infarction. , 1999, Journal of the American College of Cardiology.
[21] E Falk,et al. Stable versus unstable atherosclerosis: clinical aspects. , 1999, American heart journal.
[22] P. Reitsma,et al. The 4G/5G Polymorphism in the Plasminogen Activator Inhibitor-1 Gene Is not Associated with Myocardial Infarction , 1999, Thrombosis and Haemostasis.
[23] N. Samani,et al. Distribution of Tissue Plasminogen Activator Insertion/Deletion Polymorphism in Myocardial Infarction and Control Subjects , 1998, Thrombosis and Haemostasis.
[24] M. Margaglione,et al. The PAI-1 gene locus 4G/5G polymorphism is associated with a family history of coronary artery disease. , 1998, Arteriosclerosis, thrombosis, and vascular biology.
[25] P. Ridker,et al. Alu-repeat polymorphism in the gene coding for tissue-type plasminogen activator (t-PA) and risks of myocardial infarction among middle-aged men. , 1997, Arteriosclerosis, thrombosis, and vascular biology.
[26] A. Hofman,et al. Tissue plasminogen activator and risk of myocardial infarction. The Rotterdam Study. , 1997, Circulation.
[27] A. Hamsten,et al. Allele-specific increase in basal transcription of the plasminogen-activator inhibitor 1 gene is associated with myocardial infarction. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[28] S E Humphries,et al. Genetic determinants of arterial thrombosis. , 1994, Bailliere's best practice & research. Clinical haematology.
[29] R. Rauramaa,et al. HDL, HDL2, and HDL3 Subfractions, and the Risk of Acute Myocardial Infarction: A Prospective Population Study in Eastern Finnish Men , 1991, Circulation.
[30] K. Stowman. World health statistics. , 1949, The Milbank Memorial Fund quarterly.
[31] M. Bai,et al. Association of LT-α Ala252Gly gene polymorphism and the genetic predisposition of coronary heart disease in Chinese , 2009, Molecular Biology Reports.
[32] M. Ishaq,et al. PROFILE OF ACUTE MYOCARDIAL INFARCTION (AMI) IN PAKISTAN , 2007 .
[33] R. Bernat,et al. Impact of FXIII-A Val34Leu polymorphism on coronary artery disease in Croatian patients , 2007, Molecular Biology Reports.
[34] R. Rauramaa,et al. HDL, HDL_2, and HDL_3 subfractions, and the risk of acute myocardial infarction , 1991 .