Nanostructure of DNA repair foci revealed by superresolution microscopy
暂无分享,去创建一个
Sören Doose | Markus Sauer | Simon Memmel | Dmitri Sisario | Michael Flentje | M. Sauer | M. Flentje | S. Doose | V. Sukhorukov | C. Djuzenova | J. Neubauer | Dmitri Sisario | S. Memmel | Heiko Zimmermann | Heiko Zimmermann | Julia Neubauer | Cholpon S. Djuzenova | Vladimir L. Sukhorukov | Simon Memmel
[1] D. Chan,et al. The DNA-dependent Protein Kinase Is Inactivated by Autophosphorylation of the Catalytic Subunit (*) , 1996, The Journal of Biological Chemistry.
[2] E. Rogakou,et al. Megabase Chromatin Domains Involved in DNA Double-Strand Breaks in Vivo , 1999, The Journal of cell biology.
[3] S. Nakajima,et al. Differential phosphorylation of DNA-PKcs regulates the interplay between end-processing and end-ligation during nonhomologous end-joining. , 2015, Molecular cell.
[4] R. Fietkau,et al. Distinct increased outliers among 136 rectal cancer patients assessed by γH2AX , 2015, Radiation Oncology.
[5] J. Hancock,et al. On the use of Ripley's K-function and its derivatives to analyze domain size. , 2009, Biophysical journal.
[6] M. Svetlova,et al. Mechanism of elimination of phosphorylated histone H2AX from chromatin after repair of DNA double-strand breaks. , 2010, Mutation research.
[7] D. Rhodes. Chromatin structure: The nucleosome core all wrapped up , 1997, Nature.
[8] Mike Heilemann,et al. Super-resolution fluorescence imaging of chromosomal DNA. , 2012, Journal of structural biology.
[9] S. Jackson,et al. A new method for high-resolution imaging of Ku foci to decipher mechanisms of DNA double-strand break repair , 2013, The Journal of cell biology.
[10] W. Bonner,et al. γ-H2AX in Cancer Cells: A Potential Biomarker for Cancer Diagnostics, Prediction and Recurrence , 2006, Cell cycle.
[11] Sarah Aufmkolk,et al. Investigating cellular structures at the nanoscale with organic fluorophores. , 2013, Chemistry & biology.
[12] E. Rogakou,et al. DNA Double-stranded Breaks Induce Histone H2AX Phosphorylation on Serine 139* , 1998, The Journal of Biological Chemistry.
[13] M. Heilemann,et al. Subdiffraction-resolution fluorescence imaging with conventional fluorescent probes. , 2008, Angewandte Chemie.
[14] Mike Heilemann,et al. Live-cell super-resolution imaging with trimethoprim conjugates , 2010, Nature Methods.
[15] Low level phosphorylation of histone H2AX on serine 139 (γH2AX) is not associated with DNA double-strand breaks , 2016, Oncotarget.
[16] J. Turchi,et al. Unraveling the Complexities of DNA-Dependent Protein Kinase Autophosphorylation , 2014, Molecular and Cellular Biology.
[17] M. Flentje,et al. Cell Surface Area and Membrane Folding in Glioblastoma Cell Lines Differing in PTEN and p53 Status , 2014, PLoS ONE.
[18] S. Lees-Miller,et al. Autophosphorylation of DNA-Dependent Protein Kinase Regulates DNA End Processing and May Also Alter Double-Strand Break Repair Pathway Choice , 2005, Molecular and Cellular Biology.
[19] Takeo Ohnishi,et al. Does γH2AX foci formation depend on the presence of DNA double strand breaks , 2005 .
[20] Michel Nussenzweig,et al. H2AX: the histone guardian of the genome. , 2004, DNA repair.
[21] V. Bezrookove,et al. A minority of foci or pan-nuclear apoptotic staining of γH2AX in the S phase after UV damage contain DNA double-strand breaks , 2010, Proceedings of the National Academy of Sciences.
[22] K. McManus,et al. ATM-dependent DNA damage-independent mitotic phosphorylation of H2AX in normally growing mammalian cells. , 2005, Molecular biology of the cell.
[23] M. Durante,et al. Identification of the elementary structural units of the DNA damage response , 2017, Nature Communications.
[24] Yves Pommier,et al. The complexity of phosphorylated H2AX foci formation and DNA repair assembly at DNA double-strand breaks , 2010, Cell cycle.
[25] B. Ripley. Modelling Spatial Patterns , 1977 .
[26] E. Soutoglou,et al. DNA damage response in the absence of DNA lesions continued… , 2009, Cell cycle.
[27] E. Stelzer,et al. A macrodomain-containing histone rearranges chromatin upon sensing PARP1 activation , 2009, Nature Structural &Molecular Biology.
[28] Anthony W. Parker,et al. The dynamics of Ku70/80 and DNA-PKcs at DSBs induced by ionizing radiation is dependent on the complexity of damage , 2012, Nucleic acids research.
[29] M. Sauer,et al. rapidSTORM: accurate, fast open-source software for localization microscopy , 2012, Nature Methods.
[30] S. Hartmann,et al. Actin cytoskeleton organization, cell surface modification and invasion rate of 5 glioblastoma cell lines differing in PTEN and p53 status. , 2015, Experimental cell research.
[31] Y. Shiloh,et al. Chromatin relaxation in response to DNA double-strand breaks is modulated by a novel ATM- and KAP-1 dependent pathway , 2006, Nature Cell Biology.
[32] Louise Fairall,et al. EM measurements define the dimensions of the "30-nm" chromatin fiber: evidence for a compact, interdigitated structure. , 2006, Proceedings of the National Academy of Sciences of the United States of America.
[33] Christian Rübe,et al. DNA repair in the context of chromatin: new molecular insights by the nanoscale detection of DNA repair complexes using transmission electron microscopy. , 2011, DNA repair.
[34] M. Sauer,et al. Photometry unlocks 3D information from 2D localization microscopy data , 2016, Nature Methods.
[35] J. Reindl,et al. Chromatin organization revealed by nanostructure of irradiation induced γH2AX, 53BP1 and Rad51 foci , 2017, Scientific Reports.
[36] Mingzhu Wang,et al. Cryo-EM Study of the Chromatin Fiber Reveals a Double Helix Twisted by Tetranucleosomal Units , 2014, Science.
[37] A. Gudkov,et al. Pseudo-DNA damage response in senescent cells , 2009, Cell cycle.
[38] J. Bewersdorf,et al. H2AX chromatin structures and their response to DNA damage revealed by 4Pi microscopy , 2006, Proceedings of the National Academy of Sciences.
[39] V. Yamazaki,et al. A critical role for histone H2AX in recruitment of repair factors to nuclear foci after DNA damage , 2000, Current Biology.
[40] Burkhard Jakob,et al. Autophosphorylation of DNA-PKCS regulates its dynamics at DNA double-strand breaks , 2007, The Journal of cell biology.
[41] A. Shibata,et al. SETDB1, HP1 and SUV39 promote repositioning of 53BP1 to extend resection during homologous recombination in G2 cells , 2015, Nucleic acids research.
[42] Sabrina Rossberger,et al. Superresolution light microscopy shows nanostructure of carbon ion radiation‐induced DNA double‐strand break repair foci , 2016, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[43] T. Richmond,et al. The structure of DNA in the nucleosome core , 2003, Nature.
[44] M. Sung,et al. A Macrohistone Variant Links Dynamic Chromatin Compaction to BRCA1-Dependent Genome Maintenance , 2014, Cell reports.
[45] Markus Sauer,et al. Migration pattern, actin cytoskeleton organization and response to PI3K-, mTOR-, and Hsp90-inhibition of glioblastoma cells with different invasive capacities , 2017, Oncotarget.
[46] R. Kanaar,et al. Analysis of ionizing radiation-induced foci of DNA damage repair proteins. , 2005, Mutation research.
[47] Sebastian van de Linde,et al. Live-cell dSTORM with SNAP-tag fusion proteins. , 2011, Nature methods.
[48] E. Thompson,et al. Compromized DNA repair as a basis for identification of cancer radiotherapy patients with extreme radiosensitivity. , 2016, Cancer letters.
[49] A. El-Osta,et al. Evaluation of the efficacy of radiation-modifying compounds using γH2AX as a molecular marker of DNA double-strand breaks , 2011, Genome Integrity.
[50] P. Jeggo,et al. The life and death of DNA-PK , 2005, Oncogene.
[51] Christophe E. Redon,et al. Characteristics of γ-H2AX foci at DNA double-strand breaks sites , 2003 .
[52] M. Stuschke,et al. Use of γH2AX and other biomarkers of double-strand breaks during radiotherapy. , 2010, Seminars in radiation oncology.
[53] Michael Scholz,et al. Modeling Cell Survival after Photon Irradiation Based on Double-Strand Break Clustering in Megabase Pair Chromatin Loops , 2012, Radiation research.
[54] David J. Chen,et al. Repair of HZE-Particle-Induced DNA Double-Strand Breaks in Normal Human Fibroblasts , 2008, Radiation research.
[55] Qi Ding,et al. Autophosphorylation-dependent remodeling of the DNA-dependent protein kinase catalytic subunit regulates ligation of DNA ends. , 2004, Nucleic acids research.
[56] B. Salles,et al. Ionizing-radiation induced DNA double-strand breaks: a direct and indirect lighting up. , 2013, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.
[57] J. Cleaver,et al. Phosphorylated H2Ax is not an unambiguous marker for DNA double-strand breaks , 2011, Cell cycle.
[58] L. Wiesmüller,et al. In vitro model for DNA double‐strand break repair analysis in breast cancer reveals cell type–specific associations with age and prognosis , 2016, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[59] S. Lees-Miller,et al. The DNA-dependent protein kinase: A multifunctional protein kinase with roles in DNA double strand break repair and mitosis. , 2015, Progress in biophysics and molecular biology.