TRAIL pathway components and their putative role in granulosa cell apoptosis in the human ovary.

[1]  D. Mruk,et al.  Blood‐testis barrier dynamics are regulated by testosterone and cytokines via their differential effects on the kinetics of protein endocytosis and recycling in Sertoli cells , 2008, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[2]  A. Johnson,et al.  Cisplatin-mediated sensitivity to TRAIL-induced cell death in human granulosa tumor cells. , 2008, Gynecologic oncology.

[3]  A. Johnson,et al.  Inhibition of proteasome activity sensitizes human granulosa tumor cells to TRAIL-induced cell death. , 2008, Cancer letters.

[4]  A. Johnson,et al.  Tumor necrosis factor-related apoptosis inducing ligand expression and activity in hen granulosa cells. , 2007, Reproduction.

[5]  P. Secchiero,et al.  The role of the TRAIL/TRAIL receptors system in hematopoiesis and endothelial cell biology. , 2006, Cytokine & growth factor reviews.

[6]  G. Screaton,et al.  Preligand assembly domain-mediated ligand-independent association between TRAIL receptor 4 (TR4) and TR2 regulates TRAIL-induced apoptosis. , 2005, Proceedings of the National Academy of Sciences of the United States of America.

[7]  G. Screaton,et al.  Expression of TRAIL and TRAIL receptors in normal and malignant tissues , 2005, Cell Research.

[8]  T. Miyano,et al.  Regulation mechanism of selective atresia in porcine follicles: regulation of granulosa cell apoptosis during atresia. , 2004, The Journal of reproduction and development.

[9]  B. Nickoloff,et al.  Low‐dose UV‐radiation sensitizes keratinocytes to TRAIL‐induced apoptosis , 2004, Journal of cellular physiology.

[10]  Boquan Jin,et al.  Localization and variation of TRAIL and its receptors in human placenta during gestation. , 2004, Life sciences.

[11]  P. Secchiero,et al.  TRAIL regulates normal erythroid maturation through an ERK-dependent pathway. , 2004, Blood.

[12]  N. Inoue,et al.  Roles of tumor necrosis factor-related apoptosis-inducing ligand signaling pathway in granulosa cell apoptosis during atresia in pig ovaries. , 2003, The Journal of reproduction and development.

[13]  P. Secchiero,et al.  TRAIL Promotes the Survival and Proliferation of Primary Human Vascular Endothelial Cells by Activating the Akt and ERK Pathways , 2003, Circulation.

[14]  P. Secchiero,et al.  Tumor Necrosis Factor‐Related Apoptosis‐Inducing Ligand (TRAIL) Sequentially Upregulates Nitric Oxide and Prostanoid Production in Primary Human Endothelial Cells , 2003, Circulation research.

[15]  T. Vaskivuo,et al.  Apoptosis in the human ovary. , 2003, Reproductive biomedicine online.

[16]  A. Ashkenazi,et al.  Apo2L/TRAIL and its death and decoy receptors , 2003, Cell Death and Differentiation.

[17]  B. Fisch,et al.  Preliminary studies on apoptosis in human fetal ovaries. , 2002, Fertility and sterility.

[18]  N. Manabe,et al.  TRAIL-decoy receptor 1 plays inhibitory role in apoptosis of granulosa cells from pig ovarian follicles. , 2002, The Journal of veterinary medical science.

[19]  A. Kraft,et al.  Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) triggers apoptosis in normal prostate epithelial cells , 2002, Oncogene.

[20]  T. Vaskivuo,et al.  Survival of human ovarian follicles from fetal to adult life: apoptosis, apoptosis-related proteins, and transcription factor GATA-4. , 2001, The Journal of clinical endocrinology and metabolism.

[21]  Y. Nishi,et al.  Printed in U.S.A. Copyright © 2001 by The Endocrine Society Establishment and Characterization of a Steroidogenic Human Granulosa-Like Tumor Cell Line, KGN, That Expresses Functional Follicle-Stimulating Hormone Receptor , 2000 .

[22]  E. Alnemri,et al.  TNF-related apoptosis-inducing ligand (TRAIL) as a negative regulator of normal human erythropoiesis. , 2000, Blood.

[23]  YOUHAI H. Chen,et al.  Tumor Necrosis Factor–Related Apoptosis-Inducing Ligand (Trail) Is an Inhibitor of Autoimmune Inflammation and Cell Cycle Progression , 2000, The Journal of experimental medicine.

[24]  G. Cohen,et al.  Active Caspases and Cleaved Cytokeratins Are Sequestered into Cytoplasmic Inclusions in Trail-Induced Apoptosis , 2000, The Journal of cell biology.

[25]  M. Ultsch,et al.  Triggering cell death: the crystal structure of Apo2L/TRAIL in a complex with death receptor 5. , 1999, Molecular cell.

[26]  T. Griffith,et al.  TRAIL: a molecule with multiple receptors and control mechanisms. , 1998, Current opinion in immunology.

[27]  Xiaodong Wang,et al.  Bid, a Bcl2 Interacting Protein, Mediates Cytochrome c Release from Mitochondria in Response to Activation of Cell Surface Death Receptors , 1998, Cell.

[28]  Junying Yuan,et al.  Cleavage of BID by Caspase 8 Mediates the Mitochondrial Damage in the Fas Pathway of Apoptosis , 1998, Cell.

[29]  A. Gurney,et al.  A novel receptor for Apo2L/TRAIL contains a truncated death domain , 1997, Current Biology.

[30]  Henning Walczak,et al.  TRAIL‐R2: a novel apoptosis‐mediating receptor for TRAIL , 1997, The EMBO journal.

[31]  R. Gentz,et al.  An antagonist decoy receptor and a death domain-containing receptor for TRAIL. , 1997, Science.

[32]  Arul M. Chinnaiyan,et al.  The Receptor for the Cytotoxic Ligand TRAIL , 1997, Science.

[33]  S. Marsters,et al.  Induction of Apoptosis by Apo-2 Ligand, a New Member of the Tumor Necrosis Factor Cytokine Family* , 1996, The Journal of Biological Chemistry.

[34]  Tilly Jl The molecular basis of ovarian cell death during germ cell attrition, follicular atresia, and luteolysis. , 1996 .

[35]  C A Smith,et al.  Identification and characterization of a new member of the TNF family that induces apoptosis. , 1995, Immunity.

[36]  D. Wilson,et al.  Localization of transcription factor GATA-4 to regions of the mouse embryo involved in cardiac development. , 1994, Developmental biology.

[37]  M. Faddy,et al.  Accelerated disappearance of ovarian follicles in mid-life: implications for forecasting menopause. , 1992, Human reproduction.

[38]  T.W.Sadler Langman's Medical Embryology , 1969 .

[39]  T. G. Baker A quantitative and cytological study of germ cells in human ovaries , 1963, Proceedings of the Royal Society of London. Series B. Biological Sciences.