Blood Groups and Red Cell Antigens

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[14]  A. Koch,et al.  A Novel Function for a Glucose Analog of Blood Group H Antigen as a Mediator of Leukocyte-Endothelial Adhesion via Intracellular Adhesion Molecule 1* , 2003, Journal of Biological Chemistry.

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[23]  J. Chang,et al.  Hydrops foetalis caused by anti‐Mur in first pregnancy – a case report , 2002, Transfusion medicine.

[24]  J. Cartron,et al.  Antigenic and Functional Properties of the Human Red Blood Cell Urea Transporter hUT-B1* , 2002, The Journal of Biological Chemistry.

[25]  N. Eicher,et al.  Novel alleles at the JK blood group locus explain the absence of the erythrocyte urea transporter in European families * , 2002, British journal of haematology.

[26]  L. Cunha-Ribeiro,et al.  Blood group antigen profile predicted by molecular biology— use of real-time polymerase chain reaction to genotype important KEL, JK, RHD, and RHCE alleles , 2002, Immunohematology.

[27]  M. Laffan,et al.  The relationship between ABO histo‐blood group, factor VIII and von Willebrand factor , 2001, Transfusion medicine.

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[29]  M. Reid,et al.  First Example of Hemolytic Disease of the Newborn Caused by Anti-Or and Confirmation of the Molecular Basis of Or , 2000, Vox Sanguinis.

[30]  Y. Brossard,et al.  Quantitative determination of anti‐K (KEL1) IgG and IgG subclasses in the serum of severely alloimmunized pregnant women by ELISA , 2000, Transfusion.

[31]  P. Sistonen,et al.  Molecular heterogeneity of the Jk(null) phenotype: expression analysis of the Jk(S291P) mutation found in Finns. , 2000, Blood.

[32]  K. Geissler,et al.  Kell is not restricted to the erythropoietic lineage but is also expressed on myeloid progenitor cells , 2000, British journal of haematology.

[33]  W. Flegel,et al.  RHD gene deletion occurred in the Rhesus box. , 2000, Blood.

[34]  A. Pogo,et al.  Deletion of the Murine Duffy Gene (Dfy) Reveals that the Duffy Receptor Is Functionally Redundant , 2000, Molecular and Cellular Biology.

[35]  A. Pogo,et al.  The Duffy protein: a malarial and chemokine receptor. , 2000, Seminars in hematology.

[36]  S. Urbaniak,et al.  RhD haemolytic disease of the fetus and the newborn. , 2000, Blood reviews.

[37]  Reid,et al.  New genotypes in Fy(a− b−) individuals: nonsense mutations (Trp to stop) in the coding sequence of either FY A or FY B , 2000, British journal of haematology.

[38]  J. Poole,et al.  The MNS Blood Group Antigens, Vr (MNS12) and Mta (MNS14), Each Arise from an Amino Acid Substitution on Glycophorin A , 2000, Vox Sanguinis.

[39]  N. Avent,et al.  The Rh blood group system: a review. , 2000, Blood.

[40]  S. Henry,et al.  Genomic characterization of the Kidd blood group gene:different molecular basis of the Jk(a–b–) phenotypein Polynesians and Finns , 2000, Transfusion.

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[44]  M. Olsson,et al.  Genomic typing of the Kidd blood group locus by a single‐tube allele‐specific primer PCR technique , 1998, British journal of haematology.

[45]  L. Svensson,et al.  Point Mutations and Deletion Responsible for the Bombay H null and the Reunion H weak Blood Groups , 1998, Vox Sanguinis.

[46]  I. Roberts,et al.  Inhibition of erythroid progenitor cells by anti-Kell antibodies in fetal alloimmune anemia. , 1998, The New England journal of medicine.

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[49]  Y. Koda,et al.  Missense mutation of FUT1 and deletion of FUT2 are responsible for Indian Bombay phenotype of ABO blood group system. , 1997, Biochemical and biophysical research communications.

[50]  M. Kaneko,et al.  Wide variety of point mutations in the H gene of Bombay and para-Bombay individuals that inactivate H enzyme. , 1997, Blood.

[51]  J. Todd,et al.  The molecular basis of the Kidd blood group polymorphism and its lack of association with type 1 diabetes susceptibility. , 1997, Human molecular genetics.

[52]  W. Flegel,et al.  Polymorphism of the h allele and the population frequency of sporadic nonfunctional alleles , 1997, Transfusion.

[53]  R. Artal,et al.  Female Alloimmunization With Antibodies Known to Cause Hemolytic Disease , 1997, Obstetrics and gynecology.

[54]  Martin L. Olsson,et al.  The Blood Group Antigen Factsbook , 1997 .

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[56]  C. van Krimpen,et al.  Haemolytic disease of the newborn due to anti-K antibodies. , 1996, European journal of obstetrics, gynecology, and reproductive biology.

[57]  I. Bromilow,et al.  Haemolytic Disease of the Newborn due to Anti‐M , 1995, Vox sanguinis.

[58]  A. Waheed,et al.  Severe hemolytic disease of the newborn due to anti-Js(b). , 1995 .

[59]  C. Rodeck,et al.  Erythropoietic suppression in fetal anemia because of Kell alloimmunization. , 1994, American journal of obstetrics and gynecology.

[60]  J. Watchko,et al.  Prevalence and lack of clinical significance of blood group incompatibility in mothers with blood type A or B. , 1994, The Journal of pediatrics.

[61]  J. Lowe,et al.  Molecular basis for H blood group deficiency in Bombay (Oh) and para-Bombay individuals. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[62]  M. Uchikawa,et al.  First Case of Hemolytic Disease of the Newborn due to Anti‐Ula Antibodies , 1994, Vox sanguinis.

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[64]  N. Mohandas,et al.  Red blood cell glycophorins. , 1992, Blood.

[65]  M. Telen,et al.  What is a blood group antigen? , 1992, Transfusion.

[66]  R. Gunn,et al.  Urinary concentrating ability in patients with Jk(a-b-) blood type who lack carrier-mediated urea transport. , 1992, Journal of the American Society of Nephrology : JASN.

[67]  G. Kusnierz-Alejska,et al.  Haemolytic Disease of the Newborn due to Anti‐Dia and Incidence of the Dia Antigen in Poland , 1992, Vox sanguinis.

[68]  R. Gunn,et al.  Urea transport deficiency in Jk(a-b-) erythrocytes. , 1991, The American journal of physiology.

[69]  S. Buck,et al.  The differentiation of delayed serologic and delayed hemolytic transfusion reactions: incidence, long‐term serologic findings, and clinical significance , 1990, Transfusion.

[70]  K. Sazama,et al.  Reports of 355 transfusion‐associated deaths: 1976 through 1985 , 1990, Transfusion.

[71]  M. Reid,et al.  Associations between human red cell blood group antigens and disease. , 1990, Transfusion medicine reviews.

[72]  J. Bowman,et al.  Erythroblastosis Fetalis Produced by Anti‐k , 1989, Vox sanguinis.

[73]  D. Bowen,et al.  Delayed Haemolytic Transfusion Reaction Due to Simultaneous Appearance of Anti‐Fya and Anti‐Fy5 , 1988, Vox sanguinis.

[74]  V. Shah,et al.  Hydrops Fetalis Due to ABO Incompatibility , 1988, Clinical pediatrics.

[75]  P. Pennec,et al.  Study of Anti‐Fya in Five Black Fy(a‐b‐) Patients , 1987, Vox sanguinis.

[76]  T. Lane,et al.  B antigen acquired by normal A1 red cells exposed to a patient's serum , 1987, Transfusion.

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[78]  S. Dopp,et al.  Anti‐U and hemolytic disease of the newborn , 1983, Transfusion.

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[97]  R. Sanger,et al.  A "new" blood group character related to the ABO system. , 1952, Lancet.

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