A dominant-negative mutation in the TRESK potassium channel is linked to familial migraine with aura
暂无分享,去创建一个
Marie-Pierre Dubé | Olaf Ansorge | Namrata Gupta | Sreeram Ramagopalan | M. Z. Cader | Jorge Sequeiros | Ronald G Lafrenière | Guy A Rouleau | B. Brais | L. Griffiths | N. Gupta | M. Dubé | G. Ebers | G. Rouleau | R. Lafreniére | S. Tucker | J. Sequeiros | J. Pereira-Monteiro | S. Ramagopalan | O. Ansorge | Lyn R Griffiths | J. Poulin | Martin M. Marcinkiewicz | Martin M Marcinkiewicz | George Ebers | M Zameel Cader | Jean-François Poulin | Isabelle Andres-Enguix | Maryse Simoneau | Karine Boisvert | François Lafrenière | Shannon McLaughlan | Bernard Brais | Jose Maria Pereira-Monteiro | Stephen J Tucker | I. Andres-Enguix | M. Simoneau | S. Ramagopalan | Karine Boisvert | François Lafrenière | S. McLaughlan | J. Pereira‐Monteiro
[1] Dan Levy,et al. Activation of Meningeal Nociceptors by Cortical Spreading Depression: Implications for Migraine with Aura , 2010, The Journal of Neuroscience.
[2] Péter Enyedi,et al. Molecular Background of Leak K (cid:1) Currents: Two-Pore Domain Potassium Channels , 2010 .
[3] C. Yost,et al. Regional expression of the anesthetic-activated potassium channel TRESK in the rat nervous system , 2009, Neuroscience Letters.
[4] Howard S. Smith,et al. Calcineurin as a nociceptor modulator. , 2009, Pain physician.
[5] M. Ferrari,et al. Molecular genetics of migraine , 2009, Human Genetics.
[6] D. Mendonça,et al. Familial Clustering of Migraine: Further Evidence From a Portuguese Study , 2009, Headache.
[7] R. Ofir,et al. Maternally inherited Birk Barel mental retardation dysmorphism syndrome caused by a mutation in the genomically imprinted potassium channel KCNK9. , 2008, American journal of human genetics.
[8] Dongyue Huang,et al. Roles of TRESK, a novel two-pore domain K+ channel, in pain pathway and general anesthesia , 2008, Neuroscience Bulletin.
[9] T. Licher. Two-Pore Domain Potassium Channels , 2008 .
[10] E. Wischmeyer,et al. TRESK two‐pore‐domain K+ channels constitute a significant component of background potassium currents in murine dorsal root ganglion neurones , 2007, The Journal of physiology.
[11] H. Lerche,et al. Peripheral nerve hyperexcitability due to dominant-negative KCNQ2 mutations , 2007, Neurology.
[12] A. Mathie. Neuronal two‐pore‐domain potassium channels and their regulation by G protein‐coupled receptors , 2007, The Journal of physiology.
[13] G. Czirják,et al. Targeting of Calcineurin to an NFAT-like Docking Site Is Required for the Calcium-dependent Activation of the Background K+ Channel, TRESK* , 2006, Journal of Biological Chemistry.
[14] T. Strom,et al. Mutation in the neuronal voltage-gated sodium channel SCN1A in familial hemiplegic migraine , 2005, The Lancet.
[15] A. Straube,et al. Calcineurin Inhibitor‐Induced Headache: Clinical Characteristics and Possible Mechanisms , 2005, Headache.
[16] C. Hancock. The long and winding road , 2005, Nature Reviews Drug Discovery.
[17] R. Lea,et al. Investigation of hormone receptor genes in migraine , 2005, Neurogenetics.
[18] Joseph F. Cotten,et al. Potent Activation of the Human Tandem Pore Domain K Channel TRESK with Clinical Concentrations of Volatile Anesthetics , 2004, Anesthesia and analgesia.
[19] Z. Tóth,et al. The Two-pore Domain K+ Channel, TRESK, Is Activated by the Cytoplasmic Calcium Signal through Calcineurin* , 2004, Journal of Biological Chemistry.
[20] D. Labuda,et al. Dynamic allele-specific oligonucleotide hybridization on solid support. , 2004, Analytical biochemistry.
[21] Jes Olesen,et al. The International Classification of Headache Disorders: 2nd edition. , 2004, Cephalalgia : an international journal of headache.
[22] S. Cherny,et al. Significant linkage to migraine with aura on chromosome 11q24. , 2003, Human molecular genetics.
[23] Kohei Inamura,et al. A Novel Two-pore Domain K+ Channel, TRESK, Is Localized in the Spinal Cord* , 2003, Journal of Biological Chemistry.
[24] A. Ballabio,et al. Haploinsufficiency of ATP1A2 encoding the Na+/K+ pump α2 subunit associated with familial hemiplegic migraine type 2 , 2003, Nature Genetics.
[25] Kelly L Rogers,et al. Molecular Mechanisms of Migraine , 2003, American journal of pharmacogenomics : genomics-related research in drug development and clinical practice.
[26] C. Iadecola,et al. From CSD to headache: A long and winding road , 2002, Nature Medicine.
[27] Andrew K. Dunn,et al. Intrinsic brain activity triggers trigeminal meningeal afferents in a migraine model , 2002, Nature Medicine.
[28] G. Abecasis,et al. Merlin—rapid analysis of dense genetic maps using sparse gene flow trees , 2002, Nature Genetics.
[29] R. Lipton,et al. Prevalence and Burden of Migraine in the United States: Data From the American Migraine Study II , 2001, Headache.
[30] J. Adelman,et al. Episodic Ataxia Mutations in Kv1.1 Alter Potassium Channel Function by Dominant Negative Effects or Haploinsufficiency , 1998, The Journal of Neuroscience.
[31] G. Mitchell,et al. Long QT and ventricular arrhythmias in transgenic mice expressing the N terminus and first transmembrane segment of a voltage-gated potassium channel. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[32] Dennis E Bulman,et al. Familial Hemiplegic Migraine and Episodic Ataxia Type-2 Are Caused by Mutations in the Ca2+ Channel Gene CACNL1A4 , 1996, Cell.
[33] G. Lambert,et al. Inhalational Anesthetics Inhibit Spreading Depression: Relevance to Migraine , 1996, Cephalalgia : an international journal of headache.