Lipopolysaccharide-induced hemolysis: Evidence for direct membrane interactions
暂无分享,去创建一个
Katharina Effenberger-Neidnicht | J. Peters | H. de Groot | Jürgen Peters | M. Hartmann | K. Effenberger-Neidnicht | C. Mayer | Matthias Hartmann | Stephan Brauckmann | Herbert de Groot | Michael Nagel | Christian Mayer | S. Brauckmann | Michael Nagel
[1] G. Decavalas,et al. Severe Sepsis and Septic Shock , 2018 .
[2] C. Mayer,et al. Impact of bacterial endotoxin on the structure of DMPC membranes. , 2015, Biochimica et biophysica acta.
[3] C Briggs,et al. ICSH recommendations for the standardization of nomenclature and grading of peripheral blood cell morphological features , 2015, International journal of laboratory hematology.
[4] G. Bernard,et al. Randomized, Placebo-Controlled Trial of Acetaminophen for the Reduction of Oxidative Injury in Severe Sepsis: The Acetaminophen for the Reduction of Oxidative Injury in Severe Sepsis Trial* , 2015, Critical care medicine.
[5] M. Tomita,et al. Polymyxin B-immobilized fi ber column hemoperfusion removes endotoxin throughout a 24-hour treatment period ☆ , 2014 .
[6] G. Bernard,et al. Association between haptoglobin, hemopexin and mortality in adults with sepsis , 2013, Critical Care.
[7] N. Webster,et al. Role of Microparticles in Sepsis , 2013 .
[8] V. Santos-Ebinuma,et al. LPS–protein aggregation influences protein partitioning in aqueous two-phase micellar systems , 2013, Applied Microbiology and Biotechnology.
[9] J. Vincent,et al. Effect of eritoran, an antagonist of MD2-TLR4, on mortality in patients with severe sepsis: the ACCESS randomized trial. , 2013, JAMA.
[10] J. Peterson,et al. Association Between Cell-Free Hemoglobin, Acetaminophen, and Mortality in Patients With Sepsis: An Observational Study* , 2013, Critical care medicine.
[11] P. Buehler,et al. Hemolysis and free hemoglobin revisited: exploring hemoglobin and hemin scavengers as a novel class of therapeutic proteins. , 2013, Blood.
[12] F. Petrat,et al. Free hemoglobin concentration in severe sepsis: methods of measurement and prediction of outcome , 2012, Critical Care.
[13] B. Bonev,et al. Receptor-Independent Interaction of Bacterial Lipopolysaccharide with Lipid and Lymphocyte Membranes; the Role of Cholesterol , 2012, PloS one.
[14] S.‐H. Lee,et al. ICSH recommendations for identification, diagnostic value, and quantitation of schistocytes , 2012, International Journal of Laboratory Hematology.
[15] P. Knuefermann,et al. In Vivo Toll-Like Receptor 4 Antagonism Restores Cardiac Function During Endotoxemia , 2011, Shock.
[16] S. Barnum,et al. Red blood cell age and potentiation of transfusion‐related pathology in trauma patients , 2011, Transfusion.
[17] Angelina Iniguez,et al. Discovery and characterization of a unique mycobacterial heme acquisition system , 2011, Proceedings of the National Academy of Sciences.
[18] M. Yamazaki,et al. Spontaneous insertion of lipopolysaccharide into lipid membranes from aqueous solution. , 2011, Chemistry and physics of lipids.
[19] James E Crowe,et al. Specificity for human hemoglobin enhances Staphylococcus aureus infection. , 2010, Cell host & microbe.
[20] N. Sepúlveda,et al. A Central Role for Free Heme in the Pathogenesis of Severe Sepsis , 2010, Science Translational Medicine.
[21] G. Vercellotti,et al. Heme degradation and vascular injury. , 2010, Antioxidants & redox signaling.
[22] Mark T Gladwin,et al. Lactate dehydrogenase as a biomarker of hemolysis-associated nitric oxide resistance, priapism, leg ulceration, pulmonary hypertension, and death in patients with sickle cell disease. , 2005, Blood.
[23] M. Gladwin,et al. The clinical sequelae of intravascular hemolysis and extracellular plasma hemoglobin: a novel mechanism of human disease. , 2005, JAMA.
[24] D. Rossignol,et al. Inhibition of endotoxin response by synthetic TLR4 antagonists. , 2004, Current topics in medicinal chemistry.
[25] O. Linderkamp,et al. Endotoxin binding to erythrocyte membrane and erythrocyte deformability in human sepsis and in vitro , 2003, Critical care medicine.
[26] D. Su,et al. Hemoglobin infusion augments the tumor necrosis factor response to bacterial endotoxin (lipopolysaccharide) in mice. , 1999, Critical Care Medicine.
[27] K. Tracey,et al. HYPOPHYSECTOMY, HIGH TUMOR NECROSIS FACTOR LEVELS, AND HEMOGLOBINEMIA IN LETHAL ENDOTOXEMIC SHOCK , 1998, Shock.
[28] G. Hill. Cardiopulmonary bypass-induced inflammation: is it important? , 1998, Journal of cardiothoracic and vascular anesthesia.
[29] D. Mollitt,et al. Sepsis-induced alterations in the erythrocyte membrane. , 1994, The American surgeon.