PLZF promotes the development of asthma tolerance via affecting memory phenotypes of immune cells.

[1]  Dongchi Zhao,et al.  Respiratory syncytial virus nonstructural protein 1 breaks immune tolerance in mice by downregulating Tregs through TSLP-OX40/OX40L-mTOR axis. , 2021, Molecular immunology.

[2]  Ying Zhao,et al.  ZBTB Transcription Factors: Key Regulators of the Development, Differentiation and Effector Function of T Cells , 2021, Frontiers in Immunology.

[3]  Z. Jie,et al.  Th17/Treg cell imbalance played an important role in respiratory syncytial virus infection compromising asthma tolerance In mice. , 2021, Microbial pathogenesis.

[4]  A. Abed,et al.  Immune signature of CCR7 + central memory T cells associates with disease severity and Immunoglobulin E in bronchial asthma. , 2020, European annals of allergy and clinical immunology.

[5]  W. Phipatanakul,et al.  Prevention of asthma: targets for intervention. , 2020, Chest.

[6]  R. Sadreyev,et al.  Distinct functions of tissue-resident and circulating memory Th2 cells in allergic airway disease , 2020, bioRxiv.

[7]  Z. Jie,et al.  iNKT cells with high PLZF expression are recruited into the lung via CCL21‐CCR7 signaling to facilitate the development of asthma tolerance in mice , 2020, European journal of immunology.

[8]  Tangchun Wu,et al.  Prevalence, risk factors, and management of asthma in China: a national cross-sectional study , 2019, The Lancet.

[9]  L. Anderson,et al.  Infant Viral Respiratory Infection Nasal Immune‐Response Patterns and Their Association with Subsequent Childhood Recurrent Wheeze , 2018, American journal of respiratory and critical care medicine.

[10]  F. Takei,et al.  ILC2 memory: Recollection of previous activation , 2018, Immunological reviews.

[11]  L. Tordesillas,et al.  Mechanisms of Oral Tolerance , 2018, Clinical Reviews in Allergy & Immunology.

[12]  S. Jameson,et al.  Understanding Subset Diversity in T Cell Memory. , 2018, Immunity.

[13]  D. Meyerholz,et al.  Memory CD8 T cells mediate severe immunopathology following respiratory syncytial virus infection , 2018, PLoS pathogens.

[14]  C. Akdis,et al.  Treatment for food allergy , 2018, The Journal of allergy and clinical immunology.

[15]  Wei Sun,et al.  Respiratory Syncytial virus infection compromises asthma tolerance by recruiting interleukin‐17A‐producing cells via CCR6‐CCL20 signaling , 2017, Molecular immunology.

[16]  T. Nakayama,et al.  Epigenetic regulation of T‐helper cell differentiation, memory, and plasticity in allergic asthma , 2017, Immunological reviews.

[17]  E. Simões,et al.  Palivizumab Prophylaxis in Preterm Infants and Subsequent Recurrent Wheezing. Six‐Year Follow‐up Study , 2017, American journal of respiratory and critical care medicine.

[18]  C. Camargo,et al.  Cohort Study of Severe Bronchiolitis during Infancy and Risk of Asthma by Age 5 Years. , 2017, The journal of allergy and clinical immunology. In practice.

[19]  R. Xavier,et al.  Trained immunity: A program of innate immune memory in health and disease , 2016, Science.

[20]  Byung Hyun Kang,et al.  PLZF+ Innate T Cells Support the TGF-β-Dependent Generation of Activated/Memory-Like Regulatory T Cells , 2016, Molecules and cells.

[21]  B. Lim,et al.  Concise Review: Balancing Stem Cell Self‐Renewal and Differentiation with PLZF , 2015, Stem cells.

[22]  Ping Li,et al.  Invariant NKT Cells Act as an Adjuvant to Enhance Th2 Inflammatory Response in an OVA-Induced Mouse Model of Asthma , 2015, PloS one.

[23]  Y. Okamoto,et al.  The interleukin-33-p38 kinase axis confers memory T helper 2 cell pathogenicity in the airway. , 2015, Immunity.

[24]  Dakang Xu,et al.  BTB-ZF transcriptional regulator PLZF modifies chromatin to restrain inflammatory signaling programs , 2015, Proceedings of the National Academy of Sciences.

[25]  S. Jameson,et al.  Steady-state production of IL-4 modulates immunity in mouse strains and is determined by lineage diversity of iNKT cells , 2013, Nature Immunology.

[26]  Thomas Gebhardt,et al.  Memory T cell subsets, migration patterns, and tissue residence. , 2013, Annual review of immunology.

[27]  P. Brennan,et al.  Invariant natural killer T cells: an innate activation scheme linked to diverse effector functions , 2013, Nature Reviews Immunology.

[28]  S. Wenzel,et al.  Early infection with respiratory syncytial virus impairs regulatory T cell function and increases susceptibility to allergic asthma , 2012, Nature Medicine.

[29]  J. Wolchok,et al.  Altered Development of NKT Cells, γδ T Cells, CD8 T Cells and NK Cells in a PLZF Deficient Patient , 2011, PloS one.

[30]  Nathan E. Hnatiuk,et al.  A CD1d-Dependent Antagonist Inhibits the Activation of Invariant NKT Cells and Prevents Development of Allergen-Induced Airway Hyperreactivity , 2010, The Journal of Immunology.

[31]  D. Chaplin Overview of the immune response. , 2003, The Journal of allergy and clinical immunology.

[32]  J. Kohlmeier,et al.  Migration, maintenance and recall of memory T cells in peripheral tissues , 2009, Nature Reviews Immunology.

[33]  P. Pandolfi,et al.  The BTB–zinc finger transcriptional regulator PLZF controls the development of invariant natural killer T cell effector functions , 2008, Nature Immunology.

[34]  S. Oshima,et al.  FTY720 suppresses CD4+CD44highCD62L- effector memory T cell-mediated colitis. , 2006, American journal of physiology. Gastrointestinal and liver physiology.

[35]  H. Weiner,et al.  Oral Tolerance: Therapeutic Implications for Autoimmune Diseases , 2006, Clinical & developmental immunology.

[36]  G. Berry,et al.  Glycolipid activation of invariant T cell receptor+ NK T cells is sufficient to induce airway hyperreactivity independent of conventional CD4+ T cells. , 2006, Proceedings of the National Academy of Sciences of the United States of America.

[37]  P. Gustafsson,et al.  Severe respiratory syncytial virus bronchiolitis in infancy and asthma and allergy at age 13. , 2005, American journal of respiratory and critical care medicine.

[38]  R. Proia,et al.  Lymphocyte egress from thymus and peripheral lymphoid organs is dependent on S1P receptor 1 , 2004, Nature.

[39]  D. Kemeny,et al.  Inhibition of Th1- and Th2-Mediated Airway Inflammation by the Sphingosine 1-Phosphate Receptor Agonist FTY720 , 2003, The Journal of Immunology.

[40]  D. Umetsu,et al.  Essential role of NKT cells producing IL-4 and IL-13 in the development of allergen-induced airway hyperreactivity , 2003, Nature Medicine.

[41]  G. Dekan,et al.  Long-Lived Th2 Memory in Experimental Allergic Asthma1 , 2002, The Journal of Immunology.

[42]  S. Tsuzuki,et al.  Interactions of GATA-2 with the promyelocytic leukemia zinc finger (PLZF) protein, its homologue FAZF, and the t(11;17)-generated PLZF-retinoic acid receptor alpha oncoprotein. , 2002, Blood.

[43]  I. Ho,et al.  ROG, repressor of GATA, regulates the expression of cytokine genes. , 2000, Immunity.

[44]  A. Zelent,et al.  Fusion between a novel Krüppel‐like zinc finger gene and the retinoic acid receptor‐alpha locus due to a variant t(11;17) translocation associated with acute promyelocytic leukaemia. , 1993, The EMBO journal.