Participation of Presenilin 2 in Apoptosis: Enhanced Basal Activity Conferred by an Alzheimer Mutation
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T. Sunderland | P. Vito | B. Zhao | W. Wasco | B. Wolozin | Boyu Zhao | B. Wolozin | L. D’Adamio | T Sunderland | K. Iwasaki | J. Ganjei | E. Lacaná | J. Kusiak | J W Kusiak | W Wasco | L D'Adamio | B Wolozin | K Iwasaki | P Vito | J K Ganjei | E Lacanà | B Zhao | K. Iwasaki
[1] J. Hardy. Framing β–amyloid , 1992, Nature Genetics.
[2] B. Ghetti,et al. A mutation in the amyloid precursor protein associated with hereditary Alzheimer's disease. , 1991, Science.
[3] G. Forloni,et al. Apoptosis mediated neurotoxicity induced by chronic application of beta amyloid fragment 25-35. , 1993, Neuroreport.
[4] B. Hyman,et al. Alzheimer–associated presenilins 1 and 2 : Neuronal expression in brain and localization to intracellular membranes in mammalian cells , 1996, Nature Medicine.
[5] G. Schellenberg,et al. Candidate gene for the chromosome 1 familial Alzheimer's disease locus , 1995, Science.
[6] Seamus J. Martin,et al. Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas , 1995, Nature.
[7] A. LeBlanc,et al. Increased production of 4 kDa amyloid beta peptide in serum deprived human primary neuron cultures: possible involvement of apoptosis , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[8] S. Sisodia,et al. Altered processing of a mutant amyloid precursor protein in neuronal and endothelial cells , 1995, Journal of neuroscience research.
[9] T. Minegishi,et al. A constitutively activating mutation of the luteinizing hormone receptor in familial male precocious puberty , 1993, Nature.
[10] S. Ju,et al. Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation , 1995, Nature.
[11] P. Krammer,et al. Autocrine T-cell suicide mediated by APO-1/(Fas/CD95) , 1995, Nature.
[12] D. Kirschner,et al. Neurotrophic and neurotoxic effects of amyloid beta protein: reversal by tachykinin neuropeptides. , 1990, Science.
[13] G. Huber,et al. Apoptotic Cell Death Induced by β‐Amyloid1–42 Peptide Is Cell Type Dependent , 1995 .
[14] J. Rommens,et al. Familial Alzheimer's disease in kindreds with missense mutations in a gene on chromosome 1 related to the Alzheimer's disease type 3 gene , 1995, Nature.
[15] D. Pollen,et al. Cloning of a gene bearing missense mutations in early-onset familial Alzheimer's disease , 1995, Nature.
[16] P. W. Mesner,et al. A timetable of events during programmed cell death induced by trophic factor withdrawal from neuronal PC12 cells , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[17] I. Wilson. Another Twist to MHC-Peptide Recognition , 1996, Science.
[18] S. Singer,et al. Genetic Clues to Alzheimer's Disease , 1996, Science.
[19] S. Ben‐Sasson,et al. Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation , 1992, The Journal of cell biology.
[20] J. Hardy,et al. Early-onset Alzheimer's disease caused by mutations at codon 717 of the β-amyloid precursor protein gene , 1991, Nature.
[21] J. Haines,et al. Molecular and prospective phenotypic characterization of a pedigree with familial Alzheimer's disease and a missense mutation in codon 717 of the β‐amyloid precursor protein gene , 1992, Neurology.
[22] S. Takeda,et al. G Protein-Mediated Neuronal DNA Fragmentation Induced by Familial Alzheimer's Disease-Associated Mutants of APP , 1996, Science.
[23] C. Cotman,et al. Apoptosis is induced by beta-amyloid in cultured central nervous system neurons. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[24] J. McGillis,et al. Heat shock alters Alzheimer's β amyloid precursor protein expression in human endothelial cells , 1994, Journal of neuroscience research.
[25] P. Vito,et al. Requirement of the Familial Alzheimer's Disease Gene PS2 for Apoptosis , 1996, The Journal of Biological Chemistry.
[26] M. Pericak-Vance,et al. Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's disease , 1991, Nature.