Metabotropic glutamate receptor-dependent long-term potentiation
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[1] Mark T. Harnett,et al. Burst-Timing-Dependent Plasticity of NMDA Receptor-Mediated Transmission in Midbrain Dopamine Neurons , 2009, Neuron.
[2] W. Abraham,et al. Mechanisms of group I mGluR-dependent long-term depression of NMDA receptor-mediated transmission at Schaffer collateral-CA1 synapses. , 2009, Journal of neurophysiology.
[3] G. Barrionuevo,et al. Bidirectional Hebbian Plasticity at Hippocampal Mossy Fiber Synapses on CA3 Interneurons , 2008, The Journal of Neuroscience.
[4] R. Anwyl,et al. Extrasynaptic NR2D-Containing NMDARs Are Recruited to the Synapse during LTP of NMDAR-EPSCs , 2008, The Journal of Neuroscience.
[5] R. Anwyl,et al. Involvement of group I mGluRs in LTP induced by strong high frequency stimulation in the dentate gyrus in vitro , 2008, Neuroscience Letters.
[6] K. Chergui,et al. Long‐term potentiation in the nucleus accumbens requires both NR2A‐ and NR2B‐containing N‐methyl‐d‐aspartate receptors , 2008, The European journal of neuroscience.
[7] K. Chergui,et al. Dopamine D1 receptors and group I metabotropic glutamate receptors contribute to the induction of long-term potentiation in the nucleus accumbens , 2008, Neuropharmacology.
[8] J. Kerr,et al. Dopamine Receptor Activation Is Required for Corticostriatal Spike-Timing-Dependent Plasticity , 2008, The Journal of Neuroscience.
[9] D. Manahan‐Vaughan,et al. Metabotropic glutamate receptor 1 (mGluR1) and 5 (mGluR5) regulate late phases of LTP and LTD in the hippocampal CA1 region in vitro , 2008, The European journal of neuroscience.
[10] T. Tsumoto,et al. Metabotropic Glutamate Receptor Type 5-Dependent Long-Term Potentiation of Excitatory Synapses on Fast-Spiking GABAergic Neurons in Mouse Visual Cortex , 2008, The Journal of Neuroscience.
[11] R. Cunha,et al. Adenosine A2A Receptors Are Essential for Long-Term Potentiation of NMDA-EPSCs at Hippocampal Mossy Fiber Synapses , 2008, Neuron.
[12] Hyung-Bae Kwon,et al. Long-Term Potentiation Selectively Expressed by NMDA Receptors at Hippocampal Mossy Fiber Synapses , 2008, Neuron.
[13] J. Lacaille,et al. Selective induction of metabotropic glutamate receptor 1– and metabotropic glutamate receptor 5–dependent chemical long-term potentiation at oriens/alveus interneuron synapses of mouse hippocampus , 2008, Neuroscience.
[14] D. Purpura,et al. NMDA receptor trafficking in synaptic plasticity and neuropsychiatric disorders , 2007, Nature Reviews Neuroscience.
[15] Peter Somogyi,et al. Anti-Hebbian Long-Term Potentiation in the Hippocampal Feedback Inhibitory Circuit , 2007, Science.
[16] C. L. Cox,et al. Absence of metabotropic glutamate receptor-mediated plasticity in the neocortex of fragile X mice , 2007, Proceedings of the National Academy of Sciences.
[17] R. Anwyl,et al. Nicotinic receptor‐mediated enhancement of long‐term potentiation involves activation of metabotropic glutamate receptors and ryanodine‐sensitive calcium stores in the dentate gyrus , 2006, The European journal of neuroscience.
[18] J. Lacaille,et al. mGluR1/5 subtype‐specific calcium signalling and induction of long‐term potentiation in rat hippocampal oriens/alveus interneurones , 2006, The Journal of physiology.
[19] Kevin Fox,et al. The Role of Nitric Oxide and GluR1 in Presynaptic and Postsynaptic Components of Neocortical Potentiation , 2006, The Journal of Neuroscience.
[20] R. Anwyl,et al. Long-term potentiation is mediated by multiple kinase cascades involving CaMKII or either PKA or p42/44 MAPK in the adult rat dentate gyrus in vitro. , 2006, Journal of neurophysiology.
[21] R. Anwyl,et al. Long-Term Depression of NMDA Receptor-Mediated Synaptic Transmission Is Dependent on Activation of Metabotropic Glutamate Receptors and Is Altered to Long-Term Potentiation by Low Intracellular Calcium Buffering , 2006, The Journal of Neuroscience.
[22] G. Collingridge,et al. The regulation of hippocampal LTP by the molecular switch, a form of metaplasticity, requires mGlu5 receptors , 2005, Neuropharmacology.
[23] Dietmar Schmitz,et al. Synaptic plasticity at hippocampal mossy fibre synapses , 2005, Nature Reviews Neuroscience.
[24] Wolf Singer,et al. Synaptic plasticity in the absence of backpropagating spikes of layer II inputs to layer V pyramidal cells in rat visual cortex , 2005, The European journal of neuroscience.
[25] K. Hsu,et al. Characterization of long-term potentiation of primary afferent transmission at trigeminal synapses of juvenile rats: essential role of subtype 5 metabotropic glutamate receptors , 2005, Pain.
[26] J. Lacaille,et al. Differential Regulation of Metabotropic Glutamate Receptor- and AMPA Receptor-Mediated Dendritic Ca2+ Signals by Presynaptic and Postsynaptic Activity in Hippocampal Interneurons , 2005, The Journal of Neuroscience.
[27] P. Sanna,et al. The metabotropic glutamate receptor 5 is necessary for late-phase long-term potentiation in the hippocampal CA1 region , 2004, Brain Research.
[28] E. D’Angelo,et al. Increased neurotransmitter release during long‐term potentiation at mossy fibre–granule cell synapses in rat cerebellum , 2004, The Journal of physiology.
[29] Michael W. Salter,et al. Src kinases: a hub for NMDA receptor regulation , 2004, Nature Reviews Neuroscience.
[30] Stéphanie Ratté,et al. Synapse‐specific mGluR1‐dependent long‐term potentiation in interneurones regulates mouse hippocampal inhibition , 2004, The Journal of physiology.
[31] X. F. Wang,et al. Long term potentiation varies with layer in rat visual cortex , 2003, Brain Research.
[32] D. Chen,et al. Robust neural integration from retinal transplants in mice deficient in GFAP and vimentin , 2003, Nature Neuroscience.
[33] J. Roder,et al. Co-stimulation of mGluR5 and N-Methyl-D-aspartate Receptors Is Required for Potentiation of Excitatory Synaptic Transmission in Hippocampal Neurons* , 2003, Journal of Biological Chemistry.
[34] K. Fox,et al. Neocortical Long-Term Potentiation and Experience-Dependent Synaptic Plasticity Require α-Calcium/Calmodulin-Dependent Protein Kinase II Autophosphorylation , 2003, The Journal of Neuroscience.
[35] P. Calabresi,et al. Corticostriatal LTP requires combined mGluR1 and mGluR5 activation , 2003, Neuropharmacology.
[36] R. Anwyl,et al. Group I Metabotropic Glutamate Receptor (mGluR)-Dependent Long-Term Depression Mediated via p38 Mitogen-Activated Protein Kinase Is Inhibited by Previous High-Frequency Stimulation and Activation of mGluRs and Protein Kinase C in the Rat Dentate Gyrus In Vitro , 2002, The Journal of Neuroscience.
[37] U. Eysel,et al. Metabotropic glutamate receptors mediate expression of LTP in slices of rat visual cortex , 2002, The European journal of neuroscience.
[38] J. Lacaille,et al. A hebbian form of long-term potentiation dependent on mGluR1a in hippocampal inhibitory interneurons , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[39] P. Calabresi,et al. Dopaminergic control of synaptic plasticity in the dorsal striatum , 2001, The European journal of neuroscience.
[40] J. Wickens,et al. Dopamine D-1/D-5 receptor activation is required for long-term potentiation in the rat neostriatum in vitro. , 2001, Journal of neurophysiology.
[41] P. Greengard,et al. Dopamine and cAMP-Regulated Phosphoprotein 32 kDa Controls Both Striatal Long-Term Depression and Long-Term Potentiation, Opposing Forms of Synaptic Plasticity , 2000, The Journal of Neuroscience.
[42] W. Abraham,et al. Metabotropic Glutamate Receptors Trigger Homosynaptic Protein Synthesis to Prolong Long-Term Potentiation , 2000, The Journal of Neuroscience.
[43] P. Calabresi,et al. A Critical Role of the Nitric Oxide/cGMP Pathway in Corticostriatal Long-Term Depression , 1999, The Journal of Neuroscience.
[44] R. Anwyl. Metabotropic glutamate receptors: electrophysiological properties and role in plasticity , 1999, Brain Research Reviews.
[45] E. D’Angelo,et al. Evidence for NMDA and mGlu receptor-dependent long-term potentiation of mossy fiber-granule cell transmission in rat cerebellum. , 1999, Journal of neurophysiology.
[46] Z. Bortolotto,et al. The potent mGlu receptor antagonist LY341495 identifies roles for both cloned and novel mGlu receptors in hippocampal synaptic plasticity , 1998, Neuropharmacology.
[47] J. Roder,et al. Selective abolition of the NMDA component of long-term potentiation in mice lacking mGluR5. , 1998, Learning & memory.
[48] M. Bear,et al. Effects of the Metabotropic Glutamate Receptor Antagonist MCPG on Phosphoinositide Turnover and Synaptic Plasticity in Visual Cortex , 1998, The Journal of Neuroscience.
[49] L. Bindman,et al. Metabotropic glutamate receptors are involved in long-term potentiation in isolated slices of rat medial frontal cortex. , 1997, Journal of neurophysiology.
[50] R. Anwyl,et al. Tetanically induced LTP involves a similar increase in the AMPA and NMDA receptor components of the excitatory postsynaptic current: investigations of the involvement of mGlu receptors , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[51] J. Lacaille,et al. Mechanisms of selective long-term potentiation of excitatory synapses in stratum oriens/alveus interneurons of rat hippocampal slices. , 1995, Journal of neurophysiology.
[52] Michael J. Rowan,et al. Long-lasting enhancement of NMDA receptor-mediated synaptic transmission by metabotropic glutamate receptor activation , 1994, Nature.
[53] P. Somogyi,et al. The metabotropic glutamate receptor (mGluRlα) is concentrated at perisynaptic membrane of neuronal subpopulations as detected by immunogold reaction , 1993, Neuron.
[54] M. Bear,et al. Common forms of synaptic plasticity in the hippocampus and neocortex in vitro. , 1993, Science.
[55] G. Collingridge,et al. Induction of LTP in the hippocampus needs synaptic activation of glutamate metabotropic receptors , 1993, Nature.
[56] P. Calabresi,et al. Long‐term Potentiation in the Striatum is Unmasked by Removing the Voltage‐dependent Magnesium Block of NMDA Receptor Channels , 1992, The European journal of neuroscience.
[57] G. Collingridge,et al. Long-term potentiation of NMDA receptor-mediated synaptic transmission in the hippocampus , 1991, Nature.