Supernatants of intestinal luminal contents from mice fed high‐fat diet impair intestinal motility by injuring enteric neurons and smooth muscle cells
暂无分享,去创建一个
T. Metz | O. Balemba | T. Hofmann | H. Heyman | T. Stark | Yvonne Nyavor | Christopher Miller | G. May | Jessica L. Nicholson | Catherine R Brands | Sydney Kuther | Allysha Yasuda | Kortni K. Cox | Forrest Potter | Joshua Cady
[1] L. Forney,et al. High‐fat diet–induced alterations to gut microbiota and gut‐derived lipoteichoic acid contributes to the development of enteric neuropathy , 2020, Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society.
[2] Sugato Banerjee,et al. Gut microbiota in neurodegenerative disorders , 2019, Journal of Neuroimmunology.
[3] L. Forney,et al. Intestinal nerve cell injury occurs prior to insulin resistance in female mice ingesting a high-fat diet , 2019, Cell and Tissue Research.
[4] R. B. Oliveira,et al. Intestinal luminal content from high‐fat‐fed prediabetic mice changes epithelial barrier function in vitro , 2019, Life sciences.
[5] M. Rao,et al. Enteric nervous system manifestations of neurodegenerative disease , 2018, Brain Research.
[6] C. Knauf,et al. Galanin enhances systemic glucose metabolism through enteric Nitric Oxide Synthase-expressed neurons , 2018, Molecular metabolism.
[7] Brian D. Gulbransen,et al. The antioxidant glutathione protects against enteric neuron death in situ, but its depletion is protective during colitis. , 2018, American journal of physiology. Gastrointestinal and liver physiology.
[8] R. Schey,et al. Diabetes and the Small Intestine , 2017, Current Treatment Options in Gastroenterology.
[9] B. Berger,et al. Luminal contents from the gut of colicky infants induce visceral hypersensitivity in mice , 2017, Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society.
[10] O. Balemba,et al. Diet‐induced dysmotility and neuropathy in the gut precedes endotoxaemia and metabolic syndrome: the chicken and the egg revisited , 2017, The Journal of physiology.
[11] M. Cicala,et al. Supernatants of irritable bowel syndrome mucosal biopsies impair human colonic smooth muscle contractility , 2017, Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society.
[12] Dean P. Jones,et al. Western diet induces colonic nitrergic myenteric neuropathy and dysmotility in mice via saturated fatty acid‐ and lipopolysaccharide‐induced TLR4 signalling , 2016, The Journal of physiology.
[13] D. Fried,et al. High-fat diet-induced obesity alters nitric oxide-mediated neuromuscular transmission and smooth muscle excitability in the mouse distal colon. , 2016, American journal of physiology. Gastrointestinal and liver physiology.
[14] S. Srinivasan,et al. Intestinal Dysbiosis Contributes to the Delayed Gastrointestinal Transit in High-Fat Diet Fed Mice , 2016, Cellular and molecular gastroenterology and hepatology.
[15] Brian D. Gulbransen,et al. Enteric Glia Mediate Neuron Death in Colitis Through Purinergic Pathways That Require Connexin-43 and Nitric Oxide , 2015, Cellular and molecular gastroenterology and hepatology.
[16] C. Knauf,et al. Apelin targets gut contraction to control glucose metabolism via the brain , 2015, Gut.
[17] J. Wasilewska,et al. Gastrointestinal symptoms and autism spectrum disorder: links and risks – a possible new overlap syndrome , 2015, Pediatric health, medicine and therapeutics.
[18] P. Kempler,et al. Diabetes-Related Dysfunction of the Small Intestine and the Colon: Focus on Motility , 2015, Current Diabetes Reports.
[19] G. Farrugia. Histologic changes in diabetic gastroparesis. , 2015, Gastroenterology clinics of North America.
[20] S. Srinivasan,et al. Diabetic gastrointestinal motility disorders and the role of enteric nervous system: Current status and future directions , 2014, Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society.
[21] Ashutosh Kumar,et al. Neuroinflammation and Oxidative Stress in Diabetic Neuropathy: Futuristic Strategies Based on These Targets , 2014, International journal of endocrinology.
[22] P. François,et al. Microbiome of prebiotic-treated mice reveals novel targets involved in host response during obesity , 2014, The ISME Journal.
[23] Amber D. Miller,et al. Role of innate immunity and altered intestinal motility in LPS- and MnCl2-induced intestinal intussusception in mice. , 2014, American journal of physiology. Gastrointestinal and liver physiology.
[24] J. Shaw,et al. Global estimates of diabetes prevalence for 2013 and projections for 2035. , 2014, Diabetes Research and Clinical Practice.
[25] Navpreet Kaur,et al. Diabetic peripheral neuropathy: current perspective and future directions. , 2014, Pharmacological research.
[26] V. Mohan,et al. Increased circulatory levels of lipopolysaccharide (LPS) and zonulin signify novel biomarkers of proinflammation in patients with type 2 diabetes , 2014, Molecular and Cellular Biochemistry.
[27] B. Olde,et al. Enteric Neuropathy Can Be Induced by High Fat Diet In Vivo and Palmitic Acid Exposure In Vitro , 2013, PloS one.
[28] M. Islam. Animal Models of Diabetic Neuropathy: Progress Since 1960s , 2013, Journal of diabetes research.
[29] O. Balemba,et al. High-fat diet ingestion correlates with neuropathy in the duodenum myenteric plexus of obese mice with symptoms of type 2 diabetes , 2013, Cell and Tissue Research.
[30] Joseph M Pappachan,et al. Gastrointestinal complications of diabetes mellitus. , 2013, World journal of diabetes.
[31] J. Doré,et al. Gut microbiota and gastrointestinal health: current concepts and future directions , 2013, Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society.
[32] J. Galligan,et al. Impaired propulsive motility in the distal but not proximal colon of BK channel β1‐subunit knockout mice , 2012, Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society.
[33] C. Pothoulakis,et al. Elevated lipopolysaccharide in the colon evokes intestinal inflammation, aggravated in immune modulator-impaired mice. , 2012, American journal of physiology. Gastrointestinal and liver physiology.
[34] E. Benarroch,et al. Pathological correlates of gastrointestinal dysfunction in Parkinson's disease , 2012, Neurobiology of Disease.
[35] N. Bódi,et al. Diabetes-related alterations in the enteric nervous system and its microenvironment. , 2012, World journal of diabetes.
[36] S. Srinivasan,et al. Colonic motor dysfunction in human diabetes is associated with enteric neuronal loss and increased oxidative stress , 2011, Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society.
[37] Ferdinando Giacco,et al. Oxidative stress and diabetic complications. , 2010, Circulation research.
[38] C. Jobin,et al. High-Fat Diet: Bacteria Interactions Promote Intestinal Inflammation Which Precedes and Correlates with Obesity and Insulin Resistance in Mouse , 2010, PloS one.
[39] J. Furness,et al. Immunohistochemical analysis of neuron types in the mouse small intestine , 2008, Cell and Tissue Research.
[40] R. Bibiloni,et al. Changes in Gut Microbiota Control Metabolic Endotoxemia-Induced Inflammation in High-Fat Diet–Induced Obesity and Diabetes in Mice , 2008, Diabetes.
[41] J. Ferrières,et al. Metabolic Endotoxemia Initiates Obesity and Insulin Resistance , 2007, Diabetes.
[42] C. Knauf,et al. Comment on: Cani et al. (2007) Metabolic Endotoxemia Initiates Obesity and Insulin Resistance: Diabetes , 2007 .
[43] O. Melander,et al. Oesophageal dysmotility, delayed gastric emptying and autonomic neuropathy correlate to disturbed glucose homeostasis , 2006, Diabetologia.
[44] S. Srinivasan,et al. GDNF rescues hyperglycemia-induced diabetic enteric neuropathy through activation of the PI3K/Akt pathway. , 2006, The Journal of clinical investigation.
[45] Simon Smyth,et al. Diabetes and obesity: the twin epidemics , 2006, Nature Medicine.
[46] Daniel J Klionsky,et al. Sera from patients with type 2 Diabetes and Neuropathy Induce Autophagy and Colocalization with Mitochondria in SY5Y cells , 2005, Autophagy.
[47] T. Hofmann,et al. Isolation, structure determination, synthesis, and sensory activity of N-phenylpropenoyl-L-amino acids from cocoa (Theobroma cacao). , 2005, Journal of agricultural and food chemistry.
[48] S. Simpson. Of Mice . . . , 2004, Science.
[49] E. Feldman,et al. Oxidative stress in the pathogenesis of diabetic neuropathy. , 2004, Endocrine reviews.
[50] I. Fridovich,et al. Critical evaluation of the use of hydroethidine as a measure of superoxide anion radical. , 1998, Free radical biology & medicine.
[51] K. Hall,et al. Serum from patients with type 2 diabetes with neuropathy induces complement-independent, calcium-dependent apoptosis in cultured neuronal cells. , 1998, The Journal of clinical investigation.
[52] H. Abrahamsson. Gastrointestinal motility disorders in patients with diabetes mellitus , 1995, Journal of internal medicine.
[53] L. Schiller,et al. Disorders of gastrointestinal motility associated with diabetes mellitus. , 1983, Annals of internal medicine.