The Relative Distribution of Membranous and Cytoplasmic Met Is a Prognostic Indicator in Stage I and II Colon Cancer
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Fiona Ginty | Sudeshna Adak | Robert Filkins | Ali Can | Melinda Larsen | Michael C Montalto | Zhengyu Pang | Harvey Cline | A. Can | H. Cline | S. Adak | M. Montalto | M. Gerdes | R. Filkins | M. Larsen | Z. Pang | F. Ginty | Qing Li | Michael Gerdes | Qing Li
[1] C. Ko,et al. Colon cancer survival rates with the new American Joint Committee on Cancer sixth edition staging. , 2005, Journal of the National Cancer Institute.
[2] M. Weiser,et al. Molecular co-expression of the c-Met oncogene and hepatocyte growth factor in primary colon cancer predicts tumor stage and clinical outcome. , 2007, Cancer letters.
[3] G. Condorelli,et al. Insulin-like Growth Factor-I Receptor Internalization Regulates Signaling via the Shc/Mitogen-activated Protein Kinase Pathway, but Not the Insulin Receptor Substrate-1 Pathway* , 1998, The Journal of Biological Chemistry.
[4] R. Simon,et al. A simulation study of cross-validation for selecting an optimal cutpoint in univariate survival analysis. , 1996, Statistics in medicine.
[5] Roderick Turner,et al. c-MET expression level in primary colon cancer: a predictor of tumor invasion and lymph node metastases. , 2003, Clinical cancer research : an official journal of the American Association for Cancer Research.
[6] S. Schmid,et al. Control of EGF Receptor Signaling by Clathrin-Mediated Endocytosis , 1996, Science.
[7] W. Hohenberger,et al. High‐risk groups of patients with Stage II colon carcinoma , 2001, Cancer.
[8] I. Tsarfaty,et al. Algorithms for quantitation of protein expression variation in normal versus tumor tissue as a prognostic factor in cancer: Met oncogene expression, and breast cancer as a model. , 2000, Cytometry.
[9] D. Rimm,et al. Met, the hepatocyte growth factor receptor, localizes to the nucleus in cells at low density. , 2006, Cancer research.
[10] H. Kanetake,et al. Presence of Phosphorylated Hepatocyte Growth Factor Receptor/c-Met Is Associated with Tumor Progression and Survival in Patients with Conventional Renal Cell Carcinoma , 2006, Clinical Cancer Research.
[11] Madhu Mazumdar,et al. Methods for categorizing a prognostic variable in a multivariable setting , 2003, Statistics in medicine.
[12] F. Schmitz,et al. Functional Expression of HGF and Its Receptor in Human Colorectal Cancer , 2000, Digestion.
[13] Torsten Hothorn,et al. On the Exact Distribution of Maximally Selected Rank Statistics , 2002, Comput. Stat. Data Anal..
[14] J. Pessin,et al. Inhibition of Clathrin-Mediated Endocytosis Selectively Attenuates Specific Insulin Receptor Signal Transduction Pathways , 1998, Molecular and Cellular Biology.
[15] E. Sabo,et al. Epidermal Growth Factor Receptor, c-MET, β-Catenin, and p53 Expression as Prognostic Indicators in Stage II Colon Cancer , 2004, Clinical Cancer Research.
[16] D. Rimm,et al. Automated subcellular localization and quantification of protein expression in tissue microarrays , 2002, Nature Medicine.
[17] T. Sakaki,et al. Clinical features and management of brain arteriovenous malformations in elderly patients , 2004, Acta Neurochirurgica.
[18] M. Schumacher,et al. Evaluating the effect of optimized cutoff values in the assessment of prognostic factors , 1996 .
[19] T. Karrison,et al. Use of Irwin's restricted mean as an index for comparing survival in different treatment groups--interpretation and power considerations. , 1997, Controlled clinical trials.
[20] G. V. Vande Woude,et al. Neutralizing monoclonal antibodies to hepatocyte growth factor/scatter factor (HGF/SF) display antitumor activity in animal models , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[21] Baljit Singh,et al. Tissue microarray analysis of hepatocyte growth factor/Met pathway components reveals a role for Met, matriptase, and hepatocyte growth factor activator inhibitor 1 in the progression of node-negative breast cancer. , 2003, Cancer research.
[22] I. Yang,et al. Molecular staging for survival prediction of colorectal cancer patients. , 2005, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[23] Jilin Sun,et al. Fully human monoclonal antibodies to hepatocyte growth factor with therapeutic potential against hepatocyte growth factor/c-Met-dependent human tumors. , 2006, Cancer research.
[24] D. Rimm,et al. Classification of Breast Cancer Using Genetic Algorithms and Tissue Microarrays , 2006, Clinical Cancer Research.
[25] G. V. Vande Woude,et al. Endosomal dynamics of Met determine signaling output. , 2003, Molecular biology of the cell.
[26] P. Várnai,et al. c-Met Must Translocate to the Nucleus to Initiate Calcium Signals*♦ , 2008, Journal of Biological Chemistry.
[27] G. Woude,et al. HGF/SF-Met signaling in tumor progression , 2005, Cell Research.
[28] J. Laterra,et al. Targeting the c-Met Pathway Potentiates Glioblastoma Responses to γ-Radiation , 2005, Clinical Cancer Research.
[29] J. Laterra,et al. Systemic anti-hepatocyte growth factor monoclonal antibody therapy induces the regression of intracranial glioma xenografts. , 2006, Clinical cancer research : an official journal of the American Association for Cancer Research.
[30] A. Sobrero. Should adjuvant chemotherapy become standard treatment for patients with stage II colon cancer? For the proposal. , 2006, The Lancet. Oncology.
[31] M. Campbell,et al. Met Receptor Signaling: A Key Effector in Esophageal Adenocarcinoma , 2006, Clinical Cancer Research.
[32] B. Nordlinger,et al. Overexpression and amplification of the met/HGF receptor gene during the progression of colorectal cancer. , 1995, Clinical cancer research : an official journal of the American Association for Cancer Research.
[33] E. Rimm,et al. Met expression is associated with poor outcome in patients with axillary lymph node negative breast carcinoma , 1999, Cancer.
[34] C. Köhne. Should adjuvant chemotherapy become standard treatment for patients with stage II colon cancer? Against the proposal. , 2006, The Lancet. Oncology.
[35] John O'Quigley,et al. An application of changepoint methods in studying the effect of age on survival in breast cancer , 1999 .
[36] B. Teh,et al. Met protein expression level correlates with survival in patients with late-stage nasopharyngeal carcinoma. , 2002, Cancer research.
[37] D. Sargent,et al. Current Issues in Adjuvant Treatment of Stage II Colon Cancer , 2006, Annals of Surgical Oncology.
[38] C. Howe,et al. NGF Signaling from Clathrin-Coated Vesicles Evidence that Signaling Endosomes Serve as a Platform for the Ras-MAPK Pathway , 2001, Neuron.
[39] Mark von Zastrow,et al. Signal transduction and endocytosis: close encounters of many kinds , 2002, Nature Reviews Molecular Cell Biology.
[40] P. Parker,et al. PKC controls HGF‐dependent c‐Met traffic, signalling and cell migration , 2004, The EMBO journal.
[41] D. Rimm,et al. Tissue microarray‐based studies of patients with lymph node negative breast carcinoma show that met expression is associated with worse outcome but is not correlated with epidermal growth factor family receptors , 2003, Cancer.