Cockayne Syndrome Type A Protein Protects Primary Human Keratinocytes from Senescence.
暂无分享,去创建一个
E. Dellambra | P. Degan | D. Orioli | M. Teson | G. Zambruno | M. Stefanini | L. Guerra | R. Cardin | S. Cordisco | Lavinia Tinaburri
[1] E. Dellambra,et al. miR-200a Modulates the Expression of the DNA Repair Protein OGG1 Playing a Role in Aging of Primary Human Keratinocytes , 2018, Oxidative medicine and cellular longevity.
[2] M. Kubota,et al. Functional and clinical relevance of novel mutations in a large cohort of patients with Cockayne syndrome , 2018, Journal of Medical Genetics.
[3] E. Parlanti,et al. CSA and CSB play a role in the response to DNA breaks , 2018, Oncotarget.
[4] G. Nelson,et al. The senescent bystander effect is caused by ROS-activated NF-κB signalling , 2017, Mechanisms of Ageing and Development.
[5] V. Bohr,et al. Cockayne syndrome: Clinical features, model systems and pathways , 2017, Ageing Research Reviews.
[6] F. Rodier,et al. Keeping the senescence secretome under control: Molecular reins on the senescence-associated secretory phenotype , 2016, Experimental Gerontology.
[7] P. Degan,et al. Overexpression of parkin rescues the defective mitochondrial phenotype and the increased apoptosis of Cockayne Syndrome A cells , 2016, Oncotarget.
[8] S. Arron,et al. Absence of skin cancer in the DNA repair-deficient disease Cockayne Syndrome (CS): A survey study. , 2016, Journal of the American Academy of Dermatology.
[9] S. Jirawatnotai,et al. Ultraviolet Radiation-Induced Skin Aging: The Role of DNA Damage and Oxidative Stress in Epidermal Stem Cell Damage Mediated Skin Aging , 2016, Stem cells international.
[10] E. Dellambra,et al. The role of oncogenic Ras in human skin tumorigenesis depends on the clonogenic potential of the founding keratinocytes , 2016, Journal of Cell Science.
[11] E. Candi,et al. ßNp63 controls cellular redox status , 2015, Oncoscience.
[12] J. Goodship,et al. The Cockayne Syndrome Natural History (CoSyNH) study: clinical findings in 102 individuals and recommendations for care , 2015, Genetics in Medicine.
[13] M. Ricchetti,et al. Reversal of mitochondrial defects with CSB-dependent serine protease inhibitors in patient cells of the progeroid Cockayne syndrome , 2015, Proceedings of the National Academy of Sciences.
[14] M. Rinnerthaler,et al. Oxidative Stress in Aging Human Skin , 2015, Biomolecules.
[15] R. Rossignol,et al. Premature skin aging features rescued by inhibition of NADPH oxidase activity in XPC-deficient mice. , 2015, The Journal of investigative dermatology.
[16] C. Abbadie,et al. Level of macroautophagy drives senescent keratinocytes into cell death or neoplastic evasion , 2014, Cell Death and Disease.
[17] R. Brandes,et al. Nox family NADPH oxidases: Molecular mechanisms of activation. , 2014, Free radical biology & medicine.
[18] S. Ozeki,et al. Reactive oxygen species promotes cellular senescence in normal human epidermal keratinocytes through epigenetic regulation of p16(INK4a.). , 2014, Biochemical and biophysical research communications.
[19] J. Hoeijmakers,et al. Understanding nucleotide excision repair and its roles in cancer and ageing , 2014, Nature Reviews Molecular Cell Biology.
[20] J. Deursen. The role of senescent cells in ageing , 2014, Nature.
[21] S. Aho,et al. p16INK4A Influences the Aging Phenotype in the Living Skin Equivalent , 2013, The Journal of investigative dermatology.
[22] Kelly J. Morris,et al. A complex secretory program orchestrated by the inflammasome controls paracrine senescence , 2013, Nature Cell Biology.
[23] R. Lake,et al. Structure, function and regulation of CSB: A multi-talented gymnast , 2013, Mechanisms of Ageing and Development.
[24] V. Laugel. Cockayne syndrome: The expanding clinical and mutational spectrum , 2013, Mechanisms of Ageing and Development.
[25] J. Egly,et al. Cockayne syndrome group B (CSB) protein: At the crossroads of transcriptional networks , 2013, Mechanisms of Ageing and Development.
[26] E. Huang,et al. Conceptual developments in the causes of Cockayne syndrome , 2013, Mechanisms of Ageing and Development.
[27] V. Bohr,et al. Mitochondrial deficiency in Cockayne syndrome , 2013, Mechanisms of Ageing and Development.
[28] Bruno Vaz,et al. From laboratory tests to functional characterisation of Cockayne syndrome , 2013, Mechanisms of Ageing and Development.
[29] E. Dogliotti,et al. The role of CSA and CSB protein in the oxidative stress response , 2013, Mechanisms of Ageing and Development.
[30] E. Dellambra,et al. Induction of senescence pathways in Kindler syndrome primary keratinocytes , 2013, The British journal of dermatology.
[31] M. Dizdaroglu. Oxidatively induced DNA damage: mechanisms, repair and disease. , 2012, Cancer letters.
[32] P. Pandolfi,et al. Akt Phosphorylates the Transcriptional Repressor Bmi1 to Block Its Effects on the Tumor-Suppressing Ink4a-Arf Locus , 2012, Science Signaling.
[33] Y. Barrandon,et al. Capturing epidermal stemness for regenerative medicine. , 2012, Seminars in cell & developmental biology.
[34] B. Van Houten,et al. An altered redox balance mediates the hypersensitivity of Cockayne syndrome primary fibroblasts to oxidative stress , 2012, Aging cell.
[35] A. Salminen,et al. Emerging role of NF-κB signaling in the induction of senescence-associated secretory phenotype (SASP). , 2012, Cellular signalling.
[36] J. Gil,et al. Senescence: a new weapon for cancer therapy. , 2012, Trends in cell biology.
[37] M. Pasparakis. Role of NF‐κB in epithelial biology , 2012, Immunological reviews.
[38] V. Shi,et al. IL-4 regulates chemokine CCL26 in keratinocytes through the Jak1, 2/Stat6 signal transduction pathway: Implication for atopic dermatitis. , 2012, Molecular immunology.
[39] N. LeBrasseur,et al. Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders , 2011, Nature.
[40] Xiaowo Wang,et al. Control of the senescence-associated secretory phenotype by NF-κB promotes senescence and enhances chemosensitivity. , 2011, Genes & development.
[41] K. Bille,et al. Senescent cells develop a PARP-1 and nuclear factor-{kappa}B-associated secretome (PNAS). , 2011, Genes & development.
[42] P. Robbins,et al. NF-κB in Aging and Disease. , 2011, Aging and disease.
[43] T. Jouary,et al. XPC silencing in normal human keratinocytes triggers metabolic alterations through NOX-1 activation-mediated reactive oxygen species. , 2011, Biochimica et biophysica acta.
[44] Thomas A. Mustoe, MD, FACS,et al. MMP- and TIMP-secretion by human cutaneous keratinocytes and fibroblasts--impact of coculture and hydration. , 2011, Journal of plastic, reconstructive & aesthetic surgery : JPRAS.
[45] D. Bernard,et al. MnSOD Upregulation Induces Autophagic Programmed Cell Death in Senescent Keratinocytes , 2010, PloS one.
[46] M. Simionescu,et al. Transcriptional regulation of NADPH oxidase isoforms, Nox1 and Nox4, by nuclear factor-kappaB in human aortic smooth muscle cells. , 2010, Biochemical and biophysical research communications.
[47] Ruo-Pan Huang,et al. A biotin label-based antibody array for high-content profiling of protein expression. , 2010, Cancer genomics & proteomics.
[48] E. Dellambra,et al. Bmi-1 reduction plays a key role in physiological and premature aging of primary human keratinocytes. , 2010, The Journal of investigative dermatology.
[49] J. Campisi,et al. The senescence-associated secretory phenotype: the dark side of tumor suppression. , 2010, Annual review of pathology.
[50] P. Vogt,et al. Akt‐mediated regulation of NFκB and the essentialness of NFκB for the oncogenicity of PI3K and Akt , 2009, International journal of cancer.
[51] P. Hanawalt,et al. A UV-sensitive syndrome patient with a specific CSA mutation reveals separable roles for CSA in response to UV and oxidative DNA damage , 2009, Proceedings of the National Academy of Sciences.
[52] L. Niedernhofer. Tissue-specific accelerated aging in nucleotide excision repair deficiency , 2008, Mechanisms of Ageing and Development.
[53] Jonathan Melamed,et al. Chemokine Signaling via the CXCR2 Receptor Reinforces Senescence , 2008, Cell.
[54] S. Ishikawa,et al. p63 - Key molecule in the early phase of epithelial abnormality in idiopathic pulmonary fibrosis. , 2007, Experimental and molecular pathology.
[55] J. Campisi,et al. Cellular senescence: when bad things happen to good cells , 2007, Nature Reviews Molecular Cell Biology.
[56] I Iavarone,et al. The role of CSA in the response to oxidative DNA damage in human cells , 2007, Oncogene.
[57] E. Dogliotti,et al. Cell type and DNA damage specific response of human skin cells to environmental agents. , 2007, Mutation research.
[58] E. Dellambra,et al. Inactivation of p16INK4a (inhibitor of cyclin‐dependent kinase 4A) immortalizes primary human keratinocytes by maintaining cells in the stem cell compartment , 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[59] A. Weiner,et al. Cockayne syndrome group B protein (CSB) plays a general role in chromatin maintenance and remodeling. , 2006, Proceedings of the National Academy of Sciences of the United States of America.
[60] S. Lowe,et al. p63 deficiency activates a program of cellular senescence and leads to accelerated aging. , 2005, Genes & development.
[61] J. Kohyama,et al. Oxidative nucleotide damage and superoxide dismutase expression in the brains of xeroderma pigmentosum group A and Cockayne syndrome , 2005, Brain and Development.
[62] E. Dogliotti,et al. Differential role of transcription-coupled repair in UVB-induced response of human fibroblasts and keratinocytes. , 2005, Cancer research.
[63] D. Bernard,et al. Involvement of Rel/Nuclear Factor-κB Transcription Factors in Keratinocyte Senescence , 2004, Cancer Research.
[64] S. Lowe,et al. Rb-Mediated Heterochromatin Formation and Silencing of E2F Target Genes during Cellular Senescence , 2003, Cell.
[65] D. Bernard,et al. cRel induces mitochondrial alterations in correlation with proliferation arrest. , 2001, Free radical biology & medicine.
[66] A. M. Pedrini,et al. Different dynamics in nuclear entry of subunits of the repair/transcription factor TFIIH. , 2001, Nucleic acids research.
[67] E. Dellambra,et al. Downregulation of 14-3-3σ Prevents Clonal Evolution and Leads to Immortalization of Primary Human Keratinocytes , 2000, The Journal of cell biology.
[68] Y. Zhai,et al. Two distinct mechanisms for inhibition of cell growth in human prostate carcinoma cells with antioxidant enzyme imbalance. , 1999, Free radical biology & medicine.
[69] A. Lehmann,et al. Genetic analysis of twenty-two patients with Cockayne syndrome , 1996, Human Genetics.
[70] I. Kola,et al. Elevation in the ratio of Cu/Zn-superoxide dismutase to glutathione peroxidase activity induces features of cellular senescence and this effect is mediated by hydrogen peroxide. , 1996, Human molecular genetics.
[71] Y. Barrandon,et al. Three clonal types of keratinocyte with different capacities for multiplication. , 1987, Proceedings of the National Academy of Sciences of the United States of America.
[72] Y. Barrandon,et al. Cell size as a determinant of the clone-forming ability of human keratinocytes. , 1985, Proceedings of the National Academy of Sciences of the United States of America.
[73] A. Lehmann,et al. Failure of RNA synthesis to recover after UV irradiation: an early defect in cells from individuals with Cockayne's syndrome and xeroderma pigmentosum. , 1982, Cancer research.
[74] H. Green,et al. Seria cultivation of strains of human epidemal keratinocytes: the formation keratinizin colonies from single cell is , 1975, Cell.
[75] J. Campisi,et al. Cell senescence: role in aging and age-related diseases. , 2014, Interdisciplinary topics in gerontology.
[76] Michael J Morgan,et al. Crosstalk of reactive oxygen species and NF-κB signaling , 2011, Cell Research.