LDL-cholesterol concentrations: a genome-wide association study
暂无分享,去创建一个
D. Strachan | W. McArdle | M. Inouye | S. Grundy | P. Deloukas | M. Sandhu | R. Mahley | D. Hadley | R. Luben | N. Wareham | V. Mooser | P. Barter | R. McPherson | I. Barroso | T. Johnson | P. Vollenweider | G. Waeber | R. Loos | K. Khaw | J. Zhao | S. Bingham | E. Wheeler | D. Waterworth | Konstantinos Papadakis | K. Song | Xin Yuan | M. Sims | Y. Kesäniemi | Sofie Ashford | S. Debenham | Toby Johnson | Kijoung Song | R. Loos
[1] F. Paccaud,et al. Prevalence and characteristics of vitamin or dietary supplement users in Lausanne, Switzerland: the CoLaus study , 2009, European Journal of Clinical Nutrition.
[2] Jonathan C. Cohen,et al. Genetic and phenotypic architecture of metabolic syndrome-associated components in dyslipidemic and normolipidemic subjects: the GEMS Study. , 2008, Atherosclerosis.
[3] L. Tiret,et al. Genetics of Cardiovascular Diseases: From Single Mutations to the Whole Genome , 2007, Circulation.
[4] Bernard Keavney,et al. Association of apolipoprotein E genotypes with lipid levels and coronary risk. , 2007, JAMA.
[5] C. Gieger,et al. Genomewide association analysis of coronary artery disease. , 2007, The New England journal of medicine.
[6] P. Deloukas,et al. A genome-wide association study for celiac disease identifies risk variants in the region harboring IL2 and IL21 , 2007, Nature Genetics.
[7] Inês Barroso,et al. TCF7L2 Polymorphisms Modulate Proinsulin Levels and β-Cell Function in a British Europid Population , 2007, Diabetes.
[8] P. Donnelly,et al. A new multipoint method for genome-wide association studies by imputation of genotypes , 2007, Nature Genetics.
[9] P. Donnelly,et al. Replicating genotype–phenotype associations , 2007, Nature.
[10] Simon C. Potter,et al. Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls , 2007, Nature.
[11] Jonathan C. Cohen,et al. Molecular biology of PCSK9: its role in LDL metabolism. , 2007, Trends in biochemical sciences.
[12] W. Howard. Efficacy and safety of cholesterol-lowering treatment: prospective meta-analysis of data from 90 056 participants in 14 randomised trials of statins , 2007 .
[13] S. Humphries,et al. Genetic causes of familial hypercholesterolaemia in patients in the UK: relation to plasma lipid levels and coronary heart disease risk , 2006, Journal of Medical Genetics.
[14] E. Topol,et al. Genetic susceptibility to myocardial infarction and coronary artery disease. , 2006, Human molecular genetics.
[15] Jonathan C. Cohen,et al. A spectrum of PCSK9 alleles contributes to plasma levels of low-density lipoprotein cholesterol. , 2006, American journal of human genetics.
[16] A. Hingorani,et al. Nature's randomised trials , 2005, The Lancet.
[17] L. Kruglyak. Power tools for human genetics , 2005, Nature Genetics.
[18] R. Collins,et al. Efficacy and safety of cholesterol-lowering treatment: prospective meta-analysis of data from 90 056 participants in 14 randomised trials of statins , 2005, The Lancet.
[19] M. Daly,et al. Genome-wide association studies for common diseases and complex traits , 2005, Nature Reviews Genetics.
[20] Mark Daly,et al. Haploview: analysis and visualization of LD and haplotype maps , 2005, Bioinform..
[21] Jonathan D. Cohen,et al. Monogenic hypercholesterolemia: new insights in pathogenesis and treatment. , 2003, The Journal of clinical investigation.
[22] S. Gabriel,et al. The Structure of Haplotype Blocks in the Human Genome , 2002, Science.
[23] K. Roeder,et al. Genomic Control for Association Studies , 1999, Biometrics.
[24] N. Day,et al. EPIC-Norfolk: study design and characteristics of the cohort. European Prospective Investigation of Cancer. , 1999, British journal of cancer.
[25] G. Mcclearn,et al. Genetic and environmental influences on serum lipid levels in twins. , 1993, The New England journal of medicine.
[26] R. Mahley,et al. Apolipoprotein E: cholesterol transport protein with expanding role in cell biology. , 1988, Science.
[27] R. Levy,et al. Estimation of the concentration of low-density lipoprotein cholesterol in plasma, without use of the preparative ultracentrifuge. , 1972, Clinical chemistry.