Proteomic investigations reveal a role for RNA processing factor THRAP3 in the DNA damage response.
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Chunaram Choudhary | Jesper V Olsen | Natalia Lukashchuk | Matthias Mann | Sebastian A. Wagner | M. Mann | J. Olsen | S. Jackson | Chunaram Choudhary | B. Weinert | Petra Beli | Sebastian A Wagner | Petra Beli | Brian T Weinert | Linda Baskcomb | Stephen P Jackson | L. Baskcomb | N. Lukashchuk | S. Jackson
[1] René Bernards,et al. Loss of the cylindromatosis tumour suppressor inhibits apoptosis by activating NF-κB , 2003, Nature.
[2] A. Ashworth,et al. CYLD is a deubiquitinating enzyme that negatively regulates NF-κB activation by TNFR family members , 2003, Nature.
[3] D. H. Larsen,et al. Site-specific Phosphorylation Dynamics of the Nuclear Proteome during the DNA Damage Response* , 2010, Molecular & Cellular Proteomics.
[4] A. Whetton,et al. An ataxia-telangiectasia-mutated (ATM) kinase mediated response to DNA damage down-regulates the mRNA-binding potential of THOC5. , 2011, RNA.
[5] R. Wollman,et al. A genome-wide siRNA screen reveals diverse cellular processes and pathways that mediate genome stability. , 2009, Molecular cell.
[6] K. Tanaka,et al. Purification and cloning of a nucleotide excision repair complex involving the xeroderma pigmentosum group C protein and a human homologue of yeast RAD23. , 1994, The EMBO journal.
[7] A. Tefferi. Chronic myeloid disorders: Classification and treatment overview. , 2001, Seminars in hematology.
[8] R. Tibbetts,et al. Molecular Linkage Between the Kinase ATM and NF-κB Signaling in Response to Genotoxic Stimuli , 2006, Science.
[9] W. Tarn,et al. TRAP150 activates pre-mRNA splicing and promotes nuclear mRNA degradation , 2010, Nucleic acids research.
[10] E. Shaulian,et al. Fumarase: A Mitochondrial Metabolic Enzyme and a Cytosolic/Nuclear Component of the DNA Damage Response , 2010, PLoS biology.
[11] J. Bartek,et al. The DNA-damage response in human biology and disease , 2009, Nature.
[12] R. Aebersold,et al. Phosphoproteome resource for systems biology research. , 2011, Methods in molecular biology.
[13] Michael B Yaffe,et al. p53-deficient cells rely on ATM- and ATR-mediated checkpoint signaling through the p38MAPK/MK2 pathway for survival after DNA damage. , 2007, Cancer cell.
[14] S. Miyamoto,et al. PIASy mediates NEMO sumoylation and NF-κB activation in response to genotoxic stress , 2006, Nature Cell Biology.
[15] S. Jackson,et al. Regulation of p53 in response to DNA damage , 1999, Oncogene.
[16] A. Krainer,et al. Jcb: Article Introduction , 2022 .
[17] A. Krainer,et al. Alternative splicing regulation by interaction of phosphatase PP2Cγ with nucleic acid–binding protein YB-1 , 2007, Nature Structural &Molecular Biology.
[18] M. Mann,et al. Iodoacetamide-induced artifact mimics ubiquitination in mass spectrometry , 2008, Nature Methods.
[19] S. Jackson,et al. Dynamics of DNA damage response proteins at DNA breaks: a focus on protein modifications. , 2011, Genes & development.
[20] M. Stratton,et al. Somatic SF3B1 mutation in myelodysplasia with ring sideroblasts. , 2011, The New England journal of medicine.
[21] M. Mann,et al. MaxQuant enables high peptide identification rates, individualized p.p.b.-range mass accuracies and proteome-wide protein quantification , 2008, Nature Biotechnology.
[22] B. A. Ballif,et al. ATM and ATR Substrate Analysis Reveals Extensive Protein Networks Responsive to DNA Damage , 2007, Science.
[23] C. Bracken,et al. Regulation of cyclin D1 RNA stability by SNIP1. , 2008, Cancer research.
[24] G. Courtois,et al. The tumour suppressor CYLD negatively regulates NF-κB signalling by deubiquitination , 2003, Nature.
[25] Jiri Bartek,et al. ATM- and cell cycle-dependent regulation of ATR in response to DNA double-strand breaks , 2006, Nature Cell Biology.
[26] J. Nitiss. Targeting DNA topoisomerase II in cancer chemotherapy , 2009, Nature Reviews Cancer.
[27] B. Neumann,et al. 53BP1 nuclear bodies form around DNA lesions generated by mitotic transmission of chromosomes under replication stress , 2011, Nature Cell Biology.
[28] Shao-Cong Sun,et al. Regulation of the Deubiquitinating Enzyme CYLD by IκB Kinase Gamma-Dependent Phosphorylation , 2005, Molecular and Cellular Biology.
[29] L. Liu,et al. Role of topoisomerase II in mediating epipodophyllotoxin-induced DNA cleavage. , 1984, Cancer research.
[30] Stephen P. Jackson,et al. hnRNP K: An HDM2 Target and Transcriptional Coactivator of p53 in Response to DNA Damage , 2005, Cell.
[31] J. Hoeijmakers. DNA damage, aging, and cancer. , 2009, The New England journal of medicine.
[32] David J. Chen,et al. ATM-Dependent and -Independent Dynamics of the Nuclear Phosphoproteome After DNA Damage , 2010, Science Signaling.
[33] M. Mann,et al. Decoding signalling networks by mass spectrometry-based proteomics , 2010, Nature Reviews Molecular Cell Biology.
[34] N. Curtin,et al. Identification and Characterization of a Novel and Specific Inhibitor of the Ataxia-Telangiectasia Mutated Kinase ATM , 2004, Cancer Research.
[35] M. Mann,et al. Lysine Acetylation Targets Protein Complexes and Co-Regulates Major Cellular Functions , 2009, Science.
[36] Claus Scheidereit,et al. A nuclear poly(ADP-ribose)-dependent signalosome confers DNA damage-induced IkappaB kinase activation. , 2009, Molecular cell.
[37] K. Iwai,et al. LUBAC regulates NF‐κB activation upon genotoxic stress by promoting linear ubiquitination of NEMO , 2011, The EMBO journal.
[38] Zhijian J. Chen,et al. ATM- and NEMO-dependent ELKS ubiquitination coordinates TAK1-mediated IKK activation in response to genotoxic stress. , 2010, Molecular cell.
[39] C. Bradshaw,et al. Replication stress induces 53BP1-containing OPT domains in G1 cells , 2011, The Journal of cell biology.
[40] Karl Mechtler,et al. BAC TransgeneOmics: a high-throughput method for exploration of protein function in mammals , 2008, Nature Methods.
[41] S. Elledge,et al. The DNA damage response: making it safe to play with knives. , 2010, Molecular cell.
[42] T. Jorgensen. Enhancing radiosensitivity: Targeting the DNA repair pathways , 2009, Cancer biology & therapy.
[43] Frequent Pathway Mutations of Splicing Machinery in Myelodysplasia , 2011 .
[44] R. Greenberg,et al. ATM-Dependent Chromatin Changes Silence Transcription In cis to DNA Double-Strand Breaks , 2010, Cell.
[45] P. Roepstorff,et al. Highly Selective Enrichment of Phosphorylated Peptides from Peptide Mixtures Using Titanium Dioxide Microcolumns* , 2005, Molecular & Cellular Proteomics.
[46] 차인호,et al. Genetic alterations in oral squamous cell carcinoma progression detected by combining array-based comparative genomic hybridization and multiplex ligation-dependent probe amplification , 2011 .
[47] J. Bartek,et al. Distinct spatiotemporal dynamics of mammalian checkpoint regulators induced by DNA damage , 2003, Nature Cell Biology.
[48] M. Mann,et al. Stable Isotope Labeling by Amino Acids in Cell Culture, SILAC, as a Simple and Accurate Approach to Expression Proteomics* , 2002, Molecular & Cellular Proteomics.
[49] N. Curtin,et al. Radiosensitization and DNA repair inhibition by the combined use of novel inhibitors of DNA-dependent protein kinase and poly(ADP-ribose) polymerase-1. , 2003, Cancer research.
[50] D. Durocher,et al. A siRNA-Based Screen for Genes Involved in Chromosome End Protection , 2011, PloS one.
[51] Damian Szklarczyk,et al. The STRING database in 2011: functional interaction networks of proteins, globally integrated and scored , 2010, Nucleic Acids Res..
[52] W. Hahn,et al. Phosphorylation of the tumor suppressor CYLD by the breast cancer oncogene IKKepsilon promotes cell transformation. , 2009, Molecular cell.
[53] J. Manley,et al. Inactivation of the SR Protein Splicing Factor ASF/SF2 Results in Genomic Instability , 2005, Cell.
[54] M. Barton,et al. UV-induced inhibition of transcription involves repression of transcription initiation and phosphorylation of RNA polymerase II. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[55] A. Krainer,et al. The type 2C Ser/Thr phosphatase PP2Cgamma is a pre-mRNA splicing factor. , 1999, Genes & development.
[56] Yoshiki Murakami,et al. Hepatitis B virus-related insertional mutagenesis occurs frequently in human liver cancers and recurrently targets human telomerase gene , 2003, Oncogene.
[57] M. Mann,et al. Global, In Vivo, and Site-Specific Phosphorylation Dynamics in Signaling Networks , 2006, Cell.
[58] T. Helleday,et al. Pathways of mammalian replication fork restart , 2010, Nature Reviews Molecular Cell Biology.