Suppression of insulin‐like growth factor signalling pathway and collagen expression in keloid‐derived fibroblasts by quercetin: its therapeutic potential use in the treatment and/or prevention of keloids
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H. Huynh | E. Tran | T T Phan | S T Lee | P See | E Tran | T T T Nguyen | S Y Chan | H Huynh | P. See | S. Chan | T. Phan | T.T.T. Nguyen | S.T. Lee | Sui Yung Chan | T. T. Phan | Patrick See | Sui Yung Chan | Seng-Teik Lee | Hung Huynh | Seng-Teik Lee
[1] H. Namba,et al. Keloid fibroblasts resist ceramide-induced apoptosis by overexpression of insulin-like growth factor I receptor. , 2000, The Journal of investigative dermatology.
[2] M. Marcelli,et al. Overexpression of BCL-XL Underlies the Molecular Basis for Resistance to Staurosporine-induced Apoptosis in PC-3 Cells , 2001 .
[3] R. Dobson,et al. British Association of Dermatologists , 2014 .
[4] S. Lehar,et al. Expression of the insulin-like growth factor I receptor C terminus as a myristylated protein leads to induction of apoptosis in tumor cells. , 1998, Cancer research.
[5] R. Anton. Flavonoids and traditional medicine. , 1988, Progress in clinical and biological research.
[6] D. Yee,et al. Insulin‐like growth factor (IGF)‐I rescues breast cancer cells from chemotherapy‐induced cell death – proliferative and anti‐apoptotic effects , 1999, Breast Cancer Research and Treatment.
[7] C. Larsson,et al. Tumor suppressor activity of the gene encoding mammary-derived growth inhibitor. , 1995, Cancer research.
[8] M. Fukushima,et al. Differential inhibitory effects of various flavonoids on the activities of reverse transcriptase and cellular DNA and RNA polymerases. , 1990, European journal of biochemistry.
[9] D. Leroith. Insulin-like growth factor I receptor signaling--overlapping or redundant pathways? , 2000, Endocrinology.
[10] X. Li,et al. Overexpression of BCL-X(L) underlies the molecular basis for resistance to staurosporine-induced apoptosis in PC-3 cells. , 2001, Cancer research.
[11] J. Blenis,et al. Signal transduction via the MAP kinases: proceed at your own RSK. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[12] M. Gishizky,et al. Identification of structural characteristics that contribute to a difference in antiapoptotic function between human insulin and insulin-like growth factor 1 receptors. , 1998, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.
[13] D. Ladin,et al. Excessive scarring as a consequence of healing , 1995, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.
[14] A. Sommer,et al. Modulation of IGF-I Therapy by IGFBP-3: Potential Utility in Wound Healing , 1997 .
[15] C. Kischer,et al. Structure of the collagen nodule from hypertrophic scars and keloids. , 1981, Scanning electron microscopy.
[16] J. P. Watson,et al. Downregulation of apoptosis-related genes in keloid tissues. , 1999, The Journal of surgical research.
[17] R. Wb,et al. Keloids and hypertrophic scars: a comprehensive review. , 1989, Plastic and reconstructive surgery.
[18] D. Clemmons. Insulin-like growth factor binding proteins and their role in controlling IGF actions. , 1997, Cytokine & growth factor reviews.
[19] T. Tuan,et al. The molecular basis of keloid and hypertrophic scar formation. , 1998, Molecular medicine today.
[20] C. Vlahos,et al. The inhibition of phosphatidylinositol 3-kinase by quercetin and analogs. , 1992, Biochemical and biophysical research communications.
[21] D Kromhout,et al. Intake of potentially anticarcinogenic flavonoids and their determinants in adults in The Netherlands. , 1993, Nutrition and cancer.
[22] A. Srivastava,et al. Inhibition of phosphorylase kinase, and tyrosine protein kinase activities by quercetin. , 1985, Biochemical and biophysical research communications.
[23] C. Kischer,et al. Collagen and mucopolysaccharides in the hypertrophic scar. , 1974, Connective tissue research.
[24] T. Sugihara,et al. Keloid-derived fibroblasts are refractory to Fas-mediated apoptosis and neutralization of autocrine transforming growth factor-beta1 can abrogate this resistance. , 2000, The American journal of pathology.
[25] W C Grabb,et al. Keloids and hypertrophic scars. , 1967, University of Michigan Medical Center journal.
[26] L. Larocca,et al. Inhibitory effect of quercetin on OVCA 433 cells and presence of type II oestrogen binding sites in primary ovarian tumours and cultured cells. , 1990, British Journal of Cancer.
[27] R. Treisman,et al. The SRF accessory protein Elk-1 contains a growth factor-regulated transcriptional activation domain , 1993, Cell.
[28] M Kon,et al. On the nature of hypertrophic scars and keloids: a review. , 1999, Plastic and reconstructive surgery.
[29] Y. Yeh,et al. Quercetin: synergistic action with carboxyamidotriazole in human breast carcinoma cells. , 1995, Life sciences.
[30] I. K. Cohen,et al. Keloids and Hypertrophic Scars: A Comprehensive Review , 1991 .
[31] H. Grunicke,et al. Enhancement of the antiproliferative effect of cis‐diamminedichloroplatinum(II) and nitrogen mustard by inhibitors of protein kinase C , 1988, International journal of cancer.
[32] C. Marshall,et al. Specificity of receptor tyrosine kinase signaling: Transient versus sustained extracellular signal-regulated kinase activation , 1995, Cell.
[33] G. Saed,et al. Analysis of p53 gene mutations in keloids using polymerase chain reaction-based single-strand conformational polymorphism and DNA sequencing. , 1998, Archives of dermatology.
[34] D. D. Datubo-Brown. Keloids: a review of the literature. , 1990, British journal of plastic surgery.
[35] Paul Martin,et al. Wound Healing--Aiming for Perfect Skin Regeneration , 1997, Science.
[36] E. Racker,et al. Quercetin, an artificial regulator of the high aerobic glycolysis of tumor cells. , 1974, Journal of the National Cancer Institute.
[37] C. Kischer,et al. Perivascular myofibroblasts and microvascular occlusion in hypertrophic scars and keloids. , 1982, Human pathology.
[38] J. Kühnau. The flavonoids. A class of semi-essential food components: their role in human nutrition. , 1976, World review of nutrition and dietetics.
[39] D. Leroith. Editorial: Insulin-Like Growth Factor I Receptor Signaling-Overlapping or Redundant Pathways? , 2000, Endocrinology.
[40] G. Saed,et al. p53 and apoptosis alterations in keloids and keloid fibroblasts , 1998, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.
[41] I. McCutcheon,et al. The effects of insulin-like growth factors on tumorigenesis and neoplastic growth. , 2000, Endocrine reviews.
[42] Roger L. Williams,et al. Structural determinants of phosphoinositide 3-kinase inhibition by wortmannin, LY294002, quercetin, myricetin, and staurosporine. , 2000, Molecular cell.
[43] G. Christofori,et al. Impairment of survival factor function potentiates chemotherapy-induced apoptosis in tumor cells. , 1998, Cancer research.
[44] A. Uchida,et al. Induction of apoptosis by quercetin: involvement of heat shock protein. , 1994, Cancer research.
[45] H. Namba,et al. Overexpression of insulin-like growth factor-1 (IGF-I) receptor and the invasiveness of cultured keloid fibroblasts. , 1999, The American journal of pathology.
[46] H. Werner,et al. The insulin-like growth factor-I receptor signaling pathways are important for tumorigenesis and inhibition of apoptosis. , 1997, Critical reviews in oncogenesis.
[47] D L Larson,et al. Historical notes on the use of pressure in the treatment of hypertrophic scars or keloids. , 1993, Burns : journal of the International Society for Burn Injuries.
[48] E. Froesch,et al. Recombinant human insulin-like growth factor I stimulates growth and has distinct effects on organ size in hypophysectomized rats. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[49] T. Nikaido,et al. Quercetin arrests human leukemic T-cells in late G1 phase of the cell cycle. , 1992, Cancer research.
[50] H. Huynh,et al. Insulin-like growth factor I gene expression in the uterus is stimulated by tamoxifen and inhibited by the pure antiestrogen ICI 182780. , 1993, Cancer research.
[51] Hung Huynh,et al. Fibroblasts cocultured with keloid keratinocytes: normal fibroblasts secrete collagen in a keloidlike manner. , 2002, American journal of physiology. Cell physiology.
[52] R. Treisman. Ternary complex factors: growth factor regulated transcriptional activators. , 1994, Current opinion in genetics & development.