Apaf1 Is Required for Mitochondrial Pathways of Apoptosis and Brain Development
暂无分享,去创建一个
T. Mak | A. Hakem | R. Hakem | J. Penninger | Y. Kong | A. Elia | Tak W Mak | H. Yoshida | Razqallah Hakem | Young-Yun Kong | Anne Hakem | Ritsuko Yoshida | Josef M Penninger | Hiroki Yoshida | Andrew J Elia | R. Yoshida | Ritsuko Yoshida | H. Yoshida
[1] J. Walker,et al. Distantly related sequences in the alpha‐ and beta‐subunits of ATP synthase, myosin, kinases and other ATP‐requiring enzymes and a common nucleotide binding fold. , 1982, The EMBO journal.
[2] H. Horvitz,et al. C. elegans cell survival gene ced-9 encodes a functional homolog of the mammalian proto-oncogene bcl-2 , 1994, Cell.
[3] Matthias Mann,et al. FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling Complex , 1996, Cell.
[4] José Luis de la Pompa,et al. Differential Requirement for Caspase 9 in Apoptotic Pathways In Vivo , 1998, Cell.
[5] S. Nagata,et al. Apoptosis by Death Factor , 1997, Cell.
[6] John Calvin Reed,et al. Cloning and functional analysis of BAG-1: A novel Bcl-2-binding protein with anti-cell death activity , 1995, Cell.
[7] Francesco Cecconi,et al. Apaf1 (CED-4 Homolog) Regulates Programmed Cell Death in Mammalian Development , 1998, Cell.
[8] G. Núñez,et al. Interaction and Regulation of the Caenorhabditis elegans Death Protease CED-3 by CED-4 and CED-9* , 1997, The Journal of Biological Chemistry.
[9] D. Green,et al. The Release of Cytochrome c from Mitochondria: A Primary Site for Bcl-2 Regulation of Apoptosis , 1997, Science.
[10] Keisuke Kuida,et al. Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice , 1996, Nature.
[11] Keisuke Kuida,et al. Reduced Apoptosis and Cytochrome c–Mediated Caspase Activation in Mice Lacking Caspase 9 , 1998, Cell.
[12] S. Srinivasula,et al. Cytochrome c and dATP-Dependent Formation of Apaf-1/Caspase-9 Complex Initiates an Apoptotic Protease Cascade , 1997, Cell.
[13] Xiaodong Wang,et al. Bid, a Bcl2 Interacting Protein, Mediates Cytochrome c Release from Mitochondria in Response to Activation of Cell Surface Death Receptors , 1998, Cell.
[14] J. Sprent,et al. T-cell apoptosis detected in situ during positive and negative selection in the thymus , 1994, Nature.
[15] S. Srinivasula,et al. Molecular ordering of the Fas-apoptotic pathway: the Fas/APO-1 protease Mch5 is a CrmA-inhibitable protease that activates multiple Ced-3/ICE-like cysteine proteases. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[16] M. Peter,et al. Two CD95 (APO‐1/Fas) signaling pathways , 1998, The EMBO journal.
[17] Junying Yuan,et al. Activation of Caspase-2 in Apoptosis* , 1997, The Journal of Biological Chemistry.
[18] R. Oppenheim,et al. Control of embryonic motoneuron survival in vivo by ciliary neurotrophic factor. , 1991, Science.
[19] R. Oppenheim. Cell death during development of the nervous system. , 1991, Annual review of neuroscience.
[20] N. Thornberry. Caspases: key mediators of apoptosis. , 1998, Chemistry & biology.
[21] S. Korsmeyer,et al. Fas-induced Activation of the Cell Death-related Protease CPP32 Is Inhibited by Bcl-2 and by ICE Family Protease Inhibitors* , 1996, The Journal of Biological Chemistry.
[22] G. Núñez,et al. Interaction and Regulation of Subcellular Localization of CED-4 by CED-9 , 1997, Science.
[23] Matthias Mann,et al. FLICE is activated by association with the CD95 death‐inducing signaling complex (DISC) , 1997, The EMBO journal.
[24] Dean P. Jones,et al. Prevention of Apoptosis by Bcl-2: Release of Cytochrome c from Mitochondria Blocked , 1997, Science.
[25] Yuanming Hu,et al. Bcl-XL interacts with Apaf-1 and inhibits Apaf-1-dependent caspase-9 activation. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[26] E. Alnemri,et al. CPP32, a novel human apoptotic protein with homology to Caenorhabditis elegans cell death protein Ced-3 and mammalian interleukin-1 beta-converting enzyme. , 1994, The Journal of biological chemistry.
[27] H. Horvitz,et al. Genetic control of programmed cell death in the nematode C. elegans , 1986, Cell.
[28] Junying Yuan,et al. Cleavage of BID by Caspase 8 Mediates the Mitochondrial Damage in the Fas Pathway of Apoptosis , 1998, Cell.
[29] A. Joyner,et al. A mouse model of Greig cephalo–polysyndactyly syndrome: the extra–toesJ mutation contains an intragenic deletion of the Gli3 gene , 1993, Nature Genetics.
[30] R. Schreiber,et al. Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell death , 1990, Nature.
[31] N. Copeland,et al. Induction of apoptosis by the mouse Nedd2 gene, which encodes a protein similar to the product of the Caenorhabditis elegans cell death gene ced-3 and the mammalian IL-1 beta-converting enzyme. , 1994, Genes & development.
[32] S. Lowe,et al. Essential contribution of caspase 3/CPP32 to apoptosis and its associated nuclear changes. , 1998, Genes & development.
[33] P. Bucher,et al. The CARD domain: a new apoptotic signalling motif. , 1997, Trends in biochemical sciences.
[34] M. Egerton,et al. The generation and fate of thymocytes. , 1990, Seminars in immunology.
[35] H. Horvitz,et al. Mutations that affect neural cell lineages and cell fates during the development of the nematode Caenorhabditis elegans. , 1983, Cold Spring Harbor symposia on quantitative biology.
[36] A. Chinnaiyan,et al. Interaction of CED-4 with CED-3 and CED-9: A Molecular Framework for Cell Death , 1997, Science.
[37] M. Raff,et al. Role of Ced-3/ICE-family proteases in staurosporine-induced programmed cell death , 1996, The Journal of cell biology.
[38] Xiaodong Wang,et al. Apaf-1, a Human Protein Homologous to C. elegans CED-4, Participates in Cytochrome c–Dependent Activation of Caspase-3 , 1997, Cell.
[39] V. Caviness,et al. Numbers, time and neocortical neuronogenesis: a general developmental and evolutionary model , 1995, Trends in Neurosciences.
[40] G. Kroemer,et al. Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death , 1995, The Journal of experimental medicine.
[41] Y. Tsujimoto,et al. Bcl-2 blocks loss of mitochondrial membrane potential while ICE inhibitors act at a different step during inhibition of death induced by respiratory chain inhibitors. , 1996, Oncogene.
[42] S. Korsmeyer. Bcl-2: a repressor of lymphocyte death. , 1992, Immunology today.
[43] P. Golstein. Controlling Cell Death , 1997, Science.
[44] G. Crabtree,et al. The transcription factor NF-ATc1 regulates lymphocyte proliferation and Th2 cytokine production. , 1998, Immunity.
[45] M. Raff,et al. Programmed Cell Death in Animal Development , 1997, Cell.
[46] D. Green,et al. Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD‐specific caspase activation and independently of mitochondrial transmembrane depolarization , 1998, The EMBO journal.
[47] G. Kroemer,et al. The Central Executioner of Apoptosis: Multiple Connections between Protease Activation and Mitochondria in Fas/APO-1/CD95- and Ceramide-induced Apoptosis , 1997, The Journal of experimental medicine.
[48] A. Blaschke,et al. Widespread programmed cell death in proliferative and postmitotic regions of the fetal cerebral cortex. , 1996, Development.
[49] Guido Kroemer,et al. The biochemistry of programmed cell death , 1995, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[50] V. Dixit,et al. Caspase-9, Bcl-XL, and Apaf-1 Form a Ternary Complex* , 1998, The Journal of Biological Chemistry.
[51] J. Dichgans,et al. Developmental and genetic regulation of programmed neuronal death. , 1997, Journal of neural transmission. Supplementum.
[52] M. Spector,et al. Interaction between the C. elegans cell-death regulators CED-9 and CED-4 , 1997, Nature.
[53] P. Petit,et al. Alterations in mitochondrial structure and function are early events of dexamethasone-induced thymocyte apoptosis , 1995, The Journal of cell biology.
[54] N. Thornberry,et al. Caspases: killer proteases. , 1997, Trends in biochemical sciences.
[55] John Calvin Reed. Bcl-2 and the regulation of programmed cell death , 1994, The Journal of cell biology.
[56] H. Thoenen,et al. Ciliary neurotrophic factor. , 1994, Journal of neurobiology.
[57] C. Thompson,et al. Bcl-x(L) can inhibit apoptosis in cells that have undergone Fas-induced protease activation. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[58] S. Korsmeyer,et al. bcl-2 inhibits multiple forms of apoptosis but not negative selection in thymocytes , 1991, Cell.
[59] Yves-Alain Barde,et al. Trophic factors and neuronal survival , 1989, Neuron.
[60] V. Fadok,et al. Exposure of phosphatidylserine on the surface of apoptotic lymphocytes triggers specific recognition and removal by macrophages. , 1992, Journal of immunology.
[61] V. Caviness,et al. The cell cycle of the pseudostratified ventricular epithelium of the embryonic murine cerebral wall , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[62] H. Horvitz,et al. Developing Caenorhabditis elegans neurons may contain both cell-death protective and killer activities. , 1996, Genes & development.
[63] Shai Shaham,et al. The C. elegans cell death gene ced-3 encodes a protein similar to mammalian interleukin-1β-converting enzyme , 1993, Cell.