Supplementation of whole-cell pertussis vaccines with lipopolysaccharide analogs: modification of vaccine-induced immune responses.

[1]  F. Mooi,et al.  Lipopolysaccharide Analogs Improve Efficacy of Acellular Pertussis Vaccine and Reduce Type I Hypersensitivity in Mice , 2007, Clinical and Vaccine Immunology.

[2]  A. Sher,et al.  Cooperation of Toll-like receptor signals in innate immune defence , 2007, Nature Reviews Immunology.

[3]  D. Schwartz,et al.  The transcriptional response to lipopolysaccharide reveals a role for interferon-gamma in lung neutrophil recruitment. , 2006, American journal of physiology. Lung cellular and molecular physiology.

[4]  J. Tommassen,et al.  Expression of the Lipopolysaccharide-Modifying Enzymes PagP and PagL Modulates the Endotoxic Activity of Bordetella pertussis , 2006, Infection and Immunity.

[5]  L. O’Neill,et al.  How Toll-like receptors signal: what we know and what we don't know. , 2006, Current opinion in immunology.

[6]  Eric T. Harvill,et al.  The Bordetella bronchiseptica Type III Secretion System Inhibits Gamma Interferon Production That Is Required for Efficient Antibody-Mediated Bacterial Clearance , 2006, Infection and Immunity.

[7]  K. Hanada,et al.  A Potent Adjuvant Monophosphoryl Lipid A Triggers Various Immune Responses, but Not Secretion of IL-1β or Activation of Caspase-11 , 2006, The Journal of Immunology.

[8]  Meenu Singh,et al.  Whooping cough: the current scene. , 2006, Chest.

[9]  Paul B. Mann,et al.  The Complex Mechanism of Antibody-Mediated Clearance of Bordetella from the Lungs Requires TLR41 , 2005, The Journal of Immunology.

[10]  F. Mooi,et al.  The Bordetella pertussis virulence factor P.69 pertactin retains its immunological properties after overproduction in Escherichia coli. , 2005, Protein expression and purification.

[11]  D. Tough,et al.  Shaping of adaptive immune responses to soluble proteins by TLR agonists: A role for IFN‐α/β , 2004, Immunology and cell biology.

[12]  E. Pålsson-McDermott,et al.  Signal transduction by the lipopolysaccharide receptor, Toll‐like receptor‐4 , 2004, Immunology.

[13]  T. Ellis,et al.  Interferon‐γ activation of polymorphonuclear neutrophil function , 2004 .

[14]  M. Kennett,et al.  Antibody‐mediated bacterial clearance from the lower respiratory tract of mice requires complement component C3 , 2004, European journal of immunology.

[15]  Paul B. Mann,et al.  Role of Antibodies in Immunity to Bordetella Infections , 2003, Infection and Immunity.

[16]  M. Lynch,et al.  IL-1β-dependent neurological effects of the whole cell pertussis vaccine: a role for IL-1-associated signalling components in vaccine reactogenicity , 2003, Journal of Neuroimmunology.

[17]  J. Tommassen,et al.  Expression of foreign LpxA acyltransferases in Neisseria meningitidis results in modified lipid A with reduced toxicity and retained adjuvant activity , 2002, Cellular microbiology.

[18]  B. Zomer,et al.  Modification of Lipid A Biosynthesis inNeisseria meningitidis lpxL Mutants: Influence on Lipopolysaccharide Structure, Toxicity, and Adjuvant Activity , 2001, Infection and Immunity.

[19]  K. Mills.  Immunity to Bordetella pertussis. , 2001, Microbes and infection.

[20]  F. Mooi,et al.  Protection and humoral immune responses against Bordetella pertussis infection in mice immunized with acellular or cellular pertussis immunogens. , 2000, Vaccine.

[21]  J. Baldridge,et al.  Monophosphoryl lipid A (MPL) formulations for the next generation of vaccines. , 1999, Methods.

[22]  M. Lynch,et al.  Proinflammatory Cytokines in the Adverse Systemic and Neurologic Effects Associated with Parenteral Injection of a Whole Cell Pertussis Vaccine , 1998, Annals of the New York Academy of Sciences.

[23]  F. Mooi,et al.  The efficacy of a whole cell pertussis vaccine and fimbriae against Bordetella pertussis and Bordetella parapertussis infections in a respiratory mouse model. , 1998, Vaccine.

[24]  K. Mills,et al.  A Murine Model in Which Protection Correlates with Pertussis Vaccine Efficacy in Children Reveals Complementary Roles for Humoral and Cell-Mediated Immunity in Protection againstBordetella pertussis , 1998, Infection and Immunity.

[25]  K. Mills,et al.  Distinct T‐cell subtypes induced with whole cell and acellular pertussis vaccines in children , 1998, Immunology.

[26]  D. Burns,et al.  Role of gamma interferon in natural clearance of Bordetella pertussis infection , 1997, Infection and immunity.

[27]  K. Mills,et al.  Atypical Disease after Bordetella pertussis Respiratory Infection of Mice with Targeted Disruptions of Interferon-γ Receptor or Immunoglobulin μ Chain Genes , 1997, The Journal of experimental medicine.

[28]  S. Vogel,et al.  Lipopolysaccharide and monophosphoryl lipid A differentially regulate interleukin-12, gamma interferon, and interleukin-10 mRNA production in murine macrophages , 1997, Infection and immunity.

[29]  A. la Sala,et al.  Vaccine- and antigen-dependent type 1 and type 2 cytokine induction after primary vaccination of infants with whole-cell or acellular pertussis vaccines , 1997, Infection and immunity.

[30]  K. Mills,et al.  Bordetella pertussis-specific Th1/Th2 cells generated following respiratory infection or immunization with an acellular vaccine: comparison of the T cell cytokine profiles in infants and mice. , 1997, Developments in biological standardization.

[31]  G. Gustafson,et al.  Effects of tumor necrosis factor and dexamethasone on the regulation of interferon-gamma induction by monophosphoryl lipid A. , 1994, Journal of Immunotherapy with Emphasis on Tumor Immunology.

[32]  Rudbach Ja,et al.  Prophylactic use of monophosphoryl lipid A in patients at risk for sepsis. , 1994 .

[33]  K. Mills,et al.  Effective immunization against Bordetella pertussis respiratory infection in mice is dependent on induction of cell-mediated immunity , 1993, Infection and immunity.

[34]  C. Manthey,et al.  Dissociation of lipopolysaccharide (LPS)-inducible gene expression in murine macrophages pretreated with smooth LPS versus monophosphoryl lipid A , 1993, Infection and immunity.

[35]  A. Bradley,et al.  Multiple defects of immune cell function in mice with disrupted interferon-gamma genes. , 1993, Science.

[36]  S. Vogel,et al.  Rhodopseudomonas sphaeroides lipid A derivatives block in vitro induction of tumor necrosis factor and endotoxin tolerance by smooth lipopolysaccharide and monophosphoryl lipid A , 1992, Infection and immunity.

[37]  D. Golenbock,et al.  Lipid A-like molecules that antagonize the effects of endotoxins on human monocytes. , 1991, The Journal of biological chemistry.

[38]  Qureshi,et al.  Diphosphoryl lipid A obtained from the nontoxic lipopolysaccharide of Rhodopseudomonas sphaeroides is an endotoxin antagonist in mice , 1991, Infection and immunity.

[39]  H. Flad,et al.  IL-1 induction-capacity of defined lipopolysaccharide partial structures. , 1989, Journal of immunology.

[40]  B. Beutler,et al.  Diphosphoryl lipid A from Rhodopseudomonas sphaeroides ATCC 17023 blocks induction of cachectin in macrophages by lipopolysaccharide , 1989, Infection and immunity.

[41]  Volker Herzog,et al.  Establishment of a human cell line (mono mac 6) with characteristics of mature monocytes , 1988, International journal of cancer.

[42]  J. Poolman,et al.  Whole‐cell ELISA for typing Neisseria meningitidis with monoclonal antibodies , 1987 .

[43]  T. Yasuda,et al.  Structural requirements of lipid A responsible for the functions: a study with chemically synthesized lipid A and its analogues. , 1985, Journal of biochemistry.

[44]  T. Tanaka,et al.  Studies on Haemophilus pertussis. IX. On the immunochemical properties of the toxin of H. pertussis. , 1957, Japanese journal of microbiology.