Effects of avermectin B1a and picrotoxin on striatal release of dopamine with reference to replacement of extracellular chloride with nitrate
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[1] A. Guidotti,et al. GABAergic synapses supramolecular organization and biochemical regulation , 1983, Neuropharmacology.
[2] C. Inagaki,et al. Inhibitory effects of brotizolam, a new thienodiazepine, on limbic forebrain and neostriatal dopaminergic systems in vivo and in vitro , 1983, Neuropharmacology.
[3] E. Daniel,et al. Chloride removal selectively inhibits phasic contractions of guinea pig ileal longitudinal smooth muscle. , 1982, Canadian journal of physiology and pharmacology.
[4] K. Starke,et al. γ‐Aminobutyric Acid Can Both Inhibit and Facilitate Dopamine Release in the Caudate Nucleus of the Rabbit , 1982, Journal of neurochemistry.
[5] S. Pong,et al. Avermectin B1a Modulation of γ‐Aminobutyric Acid Receptors in Rat Brain Membranes , 1982 .
[6] B. Cox,et al. GABA enhancement of [3H]dopamine release from slices of rat striatum: dependence on slice size. , 1981, European journal of pharmacology.
[7] L. Fritz,et al. Studies on the Mechanism of Action of Avermectin B1a: Stimulation of Release of γ‐Aminobutyric Acid from Brain Synaptosomes , 1980, Journal of neurochemistry.
[8] J. Glowinski,et al. Symmetric bilateral changes in dopamine release from the caudate nuclei of the cat induced by unilateral nigral application of glycine and gaba-related compounds , 1979, Brain Research.
[9] C. Pycock,et al. Effects of omega-amino acids on tritiated dopamine release from rat striatum: evidence for a possible glycinergic mechanism. , 1979, Biochemical pharmacology.
[10] J. C. Stoof,et al. GABA modulates the release of dopamine and acetylcholine from rat caudate nucleus slices. , 1979, European journal of pharmacology.
[11] A. Gorio,et al. Avermectin B1a irreversibly blocks postsynaptic potentials at the lobster neuromuscular junction by reducing muscle membrane resistance. , 1979, Proceedings of the National Academy of Sciences of the United States of America.
[12] M. Starr. GABA-mediated potentiation of amine release from nigrostriatal dopamine neurones in vitro. , 1979, European journal of pharmacology.
[13] G. Bartholini. Interaction of striatal dopaminergic, cholinergic and GABA-ergic neurons: relation to extrapyramidal function , 1979 .
[14] M. Starr. GABA potentiates potassium-stimulated 3H-dopamine release from slices of rat substantia nigra and corpus striatum. , 1978, European journal of pharmacology.
[15] H. Stadler,et al. Evidence for an intrastriatal GABA-ergic influence on dopamine neurones of the cat , 1977, Neuropharmacology.
[16] K. Okamoto,et al. Action of amino acids and convulsants on cerebellar spontaneous action potentials in vitro: Effects of deprivation of Cl−, K+ or Na+ , 1976, Brain Research.
[17] N. Sperelakis,et al. Verapamil blockade of slow Na+ and Ca++ responses in myocardial cells. , 1974, The Journal of pharmacology and experimental therapeutics.
[18] S. Hagiwara,et al. Mechanism of anion permeation through the muscle fibre membrane of an elasmobranch fish, Taeniura lymma , 1974, The Journal of physiology.
[19] M. Ito,et al. Anion permeability of the synaptic and non‐synaptic motoneurone membrane , 1961, The Journal of physiology.