Cyclosporin A increases resting mitochondrial membrane potential in SY5Y cells and reverses the depressed mitochondrial membrane potential of Alzheimer's disease cybrids.
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R. Swerdlow | J. Parks | D. Cassarino | J. Bennett | W. Parker | J. Bennett | R. Swerdlow
[1] C. Filley,et al. Cytochrome oxidase deficiency in Alzheimer's disease , 1990, Neurology.
[2] G. Kroemer,et al. Redox regulation of apoptosis: Impact of thiol oxidation status on mitochondrial function , 1997, European journal of immunology.
[3] M. Zoratti,et al. The mitochondrial permeability transition. , 1995, Biochimica et biophysica acta.
[4] J. Pettegrew,et al. Alzheimer's Disease: Membrane‐Associated Metabolic Changes a , 1994, Annals of the New York Academy of Sciences.
[5] M. Rugolo,et al. Monitoring of the mitochondrial and plasma membrane potentials in human fibroblasts by tetraphenylphosphonium ion distribution , 1987, Journal of bioenergetics and biomembranes.
[6] M. Beal. Mitochondria, free radicals, and neurodegeneration , 1996, Current Opinion in Neurobiology.
[7] G. Cortopassi,et al. mtDNA mutations confer cellular sensitivity to oxidant stress that is partially rescued by calcium depletion and cyclosporin A. , 1997, Biochemical and biophysical research communications.
[8] S. Orrenius,et al. The role of calcium in the regulation of apoptosis. , 1996, Journal of leukocyte biology.
[9] Tamara Hirsch,et al. Mitochondrial Implication in Accidental and Programmed Cell Death: Apoptosis and Necrosis , 1997, Journal of bioenergetics and biomembranes.
[10] P. Bernardi. The permeability transition pore. Control points of a cyclosporin A-sensitive mitochondrial channel involved in cell death. , 1996, Biochimica et biophysica acta.
[11] J. Farber,et al. Cyclosporin and carnitine prevent the anoxic death of cultured hepatocytes by inhibiting the mitochondrial permeability transition. , 1993, The Journal of biological chemistry.
[12] A. Tzagoloff,et al. [45] Cytochrome oxidase from beef heart mitochondria , 1967 .
[13] D. J. Reed,et al. Influence of metabolic inhibitors on mitochondrial permeability transition and glutathione status. , 1995, Biochimica et biophysica acta.
[14] Patrizia Mecocci,et al. Oxidative damage to mitochondrial DNA is increased in Alzheimer's disease , 1994, Annals of neurology.
[15] C. Piantadosi,et al. Release of cytochrome c from liver mitochondria during permeability transition. , 1997, Biochemical and biophysical research communications.
[16] Xiaodong Wang,et al. Induction of Apoptotic Program in Cell-Free Extracts: Requirement for dATP and Cytochrome c , 1996, Cell.
[17] H. Reichmann,et al. Electron transport chain defects in Alzheimer's disease. , 1994, Neurology.
[18] T. Bird,et al. Genetic factors in Alzheimer's disease: A review of recent advances , 1996, Annals of neurology.
[19] M. Crompton,et al. Inhibition by cyclosporin A of a Ca2+-dependent pore in heart mitochondria activated by inorganic phosphate and oxidative stress. , 1988, The Biochemical journal.
[20] Jason P. Sheehan,et al. Calcium Homeostasis and Reactive Oxygen Species Production in Cells Transformed by Mitochondria from Individuals with Sporadic Alzheimer’s Disease , 1997, The Journal of Neuroscience.
[21] R. Davis,et al. Cybrids in Alzheimer's disease: A cellular model of the disease? , 1997, Neurology.
[22] W R Markesbery,et al. Oxidative stress hypothesis in Alzheimer's disease. , 1997, Free radical biology & medicine.
[23] P. Mecocci,et al. Mitochondrial membrane fluidity and oxidative damage to mitochondrial DNA in aged and AD human brain. , 1997, Molecular and chemical neuropathology.
[24] R. Swerdlow,et al. Origin and functional consequences of the complex I defect in Parkinson's disease , 1996, Annals of neurology.