Entry of glucose- and glutamine-derived carbons into the citric acid cycle supports early steps of HIV-1 infection in CD4 T cells
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T. Hope | I. Clerc | M. Sitbon | N. Taylor | V. Dardalhon | S. Tardito | Cédric Mongellaz | Leal Oburoglu | Z. Vahlas | Daouda Abba Moussa | Zoi Vahlas
[1] M. Sitbon,et al. Cellular Metabolism Is a Major Determinant of HIV-1 Reservoir Seeding in CD4+ T Cells and Offers an Opportunity to Tackle Infection. , 2019, Cell metabolism.
[2] H. Huthoff,et al. Upregulation of Glucose Uptake and Hexokinase Activity of Primary Human CD4+ T Cells in Response to Infection with HIV-1 , 2018, Viruses.
[3] N. Taylor,et al. Metabolic orchestration of T lineage differentiation and function , 2017, FEBS letters.
[4] S. Lewin,et al. Metabolically active CD4+ T cells expressing Glut1 and OX40 preferentially harbor HIV during in vitro infection , 2017, FEBS letters.
[5] H. Huthoff,et al. Evidence for Altered Glutamine Metabolism in Human Immunodeficiency Virus Type 1 Infected Primary Human CD4+ T Cells , 2017, AIDS research and human retroviruses.
[6] Michael D. Buck,et al. Metabolic Instruction of Immunity , 2017, Cell.
[7] J. Asara,et al. Inhibiting Oxidative Phosphorylation In Vivo Restrains Th17 Effector Responses and Ameliorates Murine Colitis , 2017, The Journal of Immunology.
[8] T. Curiel,et al. Suppressive IL-17A+Foxp3+ and ex-Th17 IL-17AnegFoxp3+ Treg cells are a source of tumour-associated Treg cells , 2017, Nature Communications.
[9] T. Sparwasser,et al. Metabolic pathways in T cell activation and lineage differentiation. , 2016, Seminars in immunology.
[10] Michal Sheffer,et al. Mitochondrial Biogenesis and Proteome Remodeling Promote One-Carbon Metabolism for T Cell Activation. , 2016, Cell metabolism.
[11] Marco Craveiro,et al. Cell surface Glut1 levels distinguish human CD4 and CD8 T lymphocyte subsets with distinct effector functions , 2016, Scientific Reports.
[12] L. Van Haute,et al. Human Cytomegalovirus Infection Upregulates the Mitochondrial Transcription and Translation Machineries , 2016, mBio.
[13] H. Wang,et al. Replication of the Shrimp Virus WSSV Depends on Glutamate-Driven Anaplerosis , 2016, PloS one.
[14] T. Graeber,et al. MYC-induced reprogramming of glutamine catabolism supports optimal virus replication , 2015, Nature Communications.
[15] David K. Finlay,et al. Immunometabolism: Cellular Metabolism Turns Immune Regulator* , 2015, The Journal of Biological Chemistry.
[16] M. Weekes,et al. Cell Surface Proteomic Map of HIV Infection Reveals Antagonism of Amino Acid Metabolism by Vpu and Nef , 2015, Cell host & microbe.
[17] Linda V. Sinclair,et al. The cytotoxic T cell proteome and its shaping by mammalian Target of Rapamycin , 2015, Nature Immunology.
[18] Philippe A. Robert,et al. Glutamine-dependent α-ketoglutarate production regulates the balance between T helper 1 cell and regulatory T cell generation , 2015, Science Signaling.
[19] D. Ulaeto,et al. Semliki Forest virus and Sindbis virus, but not vaccinia virus, require glycolysis for optimal replication. , 2015, The Journal of general virology.
[20] M. Weekes,et al. Antagonism of aminoacid transport in primary CD4 T cells by HIV-1 Vpu , 2015, The Lancet.
[21] A. Lane,et al. Regulation of mammalian nucleotide metabolism and biosynthesis , 2015, Nucleic acids research.
[22] J. Locasale,et al. Metabolic programming and PDHK1 control CD4+ T cell subsets and inflammation. , 2015, The Journal of clinical investigation.
[23] Roman Camarda,et al. Dengue Virus Induces and Requires Glycolysis for Optimal Replication , 2014, Journal of Virology.
[24] H. Huthoff,et al. HIV-1 pathogenicity and virion production are dependent on the metabolic phenotype of activated CD4+ T cells , 2014, Retrovirology.
[25] Nataliya Gorinski,et al. De novo fatty acid synthesis controls the fate between regulatory T and T helper 17 cells , 2014, Nature Medicine.
[26] Marco Craveiro,et al. Glucose and glutamine metabolism regulate human hematopoietic stem cell lineage specification. , 2014, Cell stem cell.
[27] J. Rathmell,et al. The glucose transporter Glut1 is selectively essential for CD4 T cell activation and effector function. , 2014, Cell metabolism.
[28] R. Deberardinis,et al. Oxidation of alpha-ketoglutarate is required for reductive carboxylation in cancer cells with mitochondrial defects. , 2014, Cell reports.
[29] Jae-Hoon Chang,et al. Inflammatory T cell responses rely on amino acid transporter ASCT2 facilitation of glutamine uptake and mTORC1 kinase activation. , 2014, Immunity.
[30] Mark S. Sundrud,et al. Small-molecule RORγt antagonists inhibit T helper 17 cell transcriptional network by divergent mechanisms. , 2014, Immunity.
[31] Roman Camarda,et al. Vaccinia Virus Requires Glutamine but Not Glucose for Efficient Replication , 2014, Journal of Virology.
[32] S. Lewin,et al. Increased glucose metabolic activity is associated with CD4+ T-cell activation and depletion during chronic HIV infection , 2014, AIDS.
[33] Jennifer B Dennison,et al. Quinoline 3-sulfonamides inhibit lactate dehydrogenase A and reverse aerobic glycolysis in cancer cells , 2013, Cancer & metabolism.
[34] B. Faubert,et al. CD8 memory T cells have a bioenergetic advantage that underlies their rapid recall ability , 2013, Proceedings of the National Academy of Sciences.
[35] Thomas M. Wasylenko,et al. Reductive glutamine metabolism is a function of the α-ketoglutarate to citrate ratio in cells , 2013, Nature Communications.
[36] B. Faubert,et al. Posttranscriptional Control of T Cell Effector Function by Aerobic Glycolysis , 2013, Cell.
[37] Marco Craveiro,et al. Metabolic pathways as regulators of HIV infection , 2013, Current opinion in HIV and AIDS.
[38] J. Zack,et al. HIV restriction in quiescent CD4+ T cells , 2013, Retrovirology.
[39] Linda V. Sinclair,et al. Control of amino-acid transport by antigen receptors coordinates the metabolic reprogramming essential for T cell differentiation , 2013, Nature Immunology.
[40] J. Licht,et al. Mitochondria are required for antigen-specific T cell activation through reactive oxygen species signaling. , 2013, Immunity.
[41] R. König,et al. SAMHD1 restricts HIV-1 infection in resting CD4+ T cells , 2012, Nature Medicine.
[42] Y. Crow,et al. SAMHD1 restricts HIV-1 reverse transcription in quiescent CD4+ T-cells , 2012, Retrovirology.
[43] E. Gottlieb,et al. Glutaminolysis activates Rag-mTORC1 signaling. , 2012, Molecular cell.
[44] Baek Kim,et al. SAMHD1 restricts the replication of human immunodeficiency virus type 1 by depleting the intracellular pool of deoxynucleoside triphosphates , 2012, Nature Immunology.
[45] Marco Craveiro,et al. Glut1-mediated glucose transport regulates HIV infection , 2012, Proceedings of the National Academy of Sciences.
[46] D. Green,et al. The transcription factor Myc controls metabolic reprogramming upon T lymphocyte activation. , 2011, Immunity.
[47] D. Green,et al. HIF1α–dependent glycolytic pathway orchestrates a metabolic checkpoint for the differentiation of TH17 and Treg cells , 2011, The Journal of experimental medicine.
[48] J. Alwine,et al. Viral effects on metabolism: changes in glucose and glutamine utilization during human cytomegalovirus infection. , 2011, Trends in microbiology.
[49] B. Sobhian,et al. SAMHD1 is the dendritic- and myeloid-cell-specific HIV-1 restriction factor counteracted by Vpx , 2011, Nature.
[50] M. Washburn,et al. Vpx relieves inhibition of HIV-1 infection of macrophages mediated by the SAMHD1 protein , 2011, Nature.
[51] J. Routy,et al. Memory CCR6+CD4+ T Cells Are Preferential Targets for Productive HIV Type 1 Infection Regardless of Their Expression of Integrin β7 , 2011, The Journal of Immunology.
[52] J. Rathmell,et al. Cutting Edge: Distinct Glycolytic and Lipid Oxidative Metabolic Programs Are Essential for Effector and Regulatory CD4+ T Cell Subsets , 2011, The Journal of Immunology.
[53] D. Sabatini,et al. Regulation of the mTOR complex 1 pathway by nutrients, growth factors, and stress. , 2010, Molecular cell.
[54] F. Miot,et al. The Nonphagocytic NADPH Oxidase Duox1 Mediates a Positive Feedback Loop During T Cell Receptor Signaling , 2010, Science Signaling.
[55] K. Frauwirth,et al. Glutamine Uptake and Metabolism Are Coordinately Regulated by ERK/MAPK during T Lymphocyte Activation , 2010, The Journal of Immunology.
[56] J. Routy,et al. Peripheral Blood CCR4+CCR6+ and CXCR3+CCR6+ CD4+ T Cells Are Highly Permissive to HIV-1 Infection , 2009, The Journal of Immunology.
[57] David M. Sabatini,et al. An Emerging Role of mTOR in Lipid Biosynthesis , 2009, Current Biology.
[58] J. Alwine,et al. Glutamine Metabolism Is Essential for Human Cytomegalovirus Infection , 2009, Journal of Virology.
[59] D. Sabatini,et al. mTOR signaling at a glance , 2009, Journal of Cell Science.
[60] Mark S. Sundrud,et al. Halofuginone Inhibits TH17 Cell Differentiation by Activating the Amino Acid Starvation Response , 2009, Science.
[61] Jeffrey P. MacKeigan,et al. Bidirectional Transport of Amino Acids Regulates mTOR and Autophagy , 2009, Cell.
[62] R. Finger,et al. ASCT2 silencing regulates mammalian target-of-rapamycin growth and survival signaling in human hepatoma cells. , 2007, American journal of physiology. Cell physiology.
[63] Marco Craveiro,et al. Isolated receptor binding domains of HTLV-1 and HTLV-2 envelopes bind Glut-1 on activated CD4+ and CD8+ T cells , 2007, Retrovirology.
[64] M. Sitbon,et al. Glucose transporter 1 expression identifies a population of cycling CD4+ CD8+ human thymocytes with high CXCR4-induced chemotaxis. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[65] M. Sitbon,et al. HTLV-1 and -2 envelope SU subdomains and critical determinants in receptor binding , 2004, Retrovirology.
[66] M. Sitbon,et al. The Ubiquitous Glucose Transporter GLUT-1 Is a Receptor for HTLV , 2003, Cell.
[67] C. Thompson,et al. Activated Akt promotes increased resting T cell size, CD28‐independent T cell growth, and development of autoimmunity and lymphoma , 2003, European journal of immunology.
[68] N. Taylor,et al. IL-7 surface-engineered lentiviral vectors promote survival and efficient gene transfer in resting primary T lymphocytes. , 2003, Blood.
[69] M. Sitbon,et al. The HTLV receptor is an early T-cell activation marker whose expression requires de novo protein synthesis. , 2003, Blood.
[70] W. Greene,et al. A sensitive and specific enzyme-based assay detecting HIV-1 virion fusion in primary T lymphocytes , 2002, Nature Biotechnology.
[71] C. Thompson,et al. The CD28 signaling pathway regulates glucose metabolism. , 2002, Immunity.
[72] L. Oberley,et al. Discrete Generation of Superoxide and Hydrogen Peroxide by T Cell Receptor Stimulation , 2002, The Journal of experimental medicine.
[73] Y. Korin,et al. Nonproductive Human Immunodeficiency Virus Type 1 Infection in Nucleoside-Treated G0 Lymphocytes , 1999, Journal of Virology.
[74] M. Stevenson,et al. Establishment of a Functional Human Immunodeficiency Virus Type 1 (HIV-1) Reverse Transcription Complex Involves the Cytoskeleton , 1998, The Journal of experimental medicine.
[75] Thomas J Hope,et al. Detection and Tracking of Dual-Labeled HIV Particles Using Wide-Field Live Cell Imaging to Follow Viral Core Integrity. , 2016, Methods in molecular biology.
[76] V. Prasad,et al. Analysis of 2-LTR circle junctions of viral DNA in infected cells. , 2009, Methods in molecular biology.
[77] R. Curi,et al. Glutamine, gene expression, and cell function. , 2007, Frontiers in bioscience : a journal and virtual library.