-terminal kinase activity in human skeletal muscle 2 Eccentric exercise markedly increases c-Jun NH

[1]  R. Kolesnick,et al.  Stress signals for apoptosis: ceramide and c-Jun kinase , 1998, Oncogene.

[2]  R. Fielding,et al.  Exercise stimulates c-Jun NH2 kinase activity and c-Jun transcriptional activity in human skeletal muscle. , 1998, Biochemical and biophysical research communications.

[3]  R. Hajjar,et al.  Role of the stress-activated protein kinases in endothelin-induced cardiomyocyte hypertrophy. , 1998, The Journal of clinical investigation.

[4]  J. Henriksson,et al.  Divergent effects of exercise on metabolic and mitogenic signaling pathways in human skeletal muscle , 1998, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[5]  O. Ljungqvist,et al.  Extracellular-regulated protein kinase cascades are activated in response to injury in human skeletal muscle. , 1998, American journal of physiology. Cell physiology.

[6]  D. Templeton,et al.  Induction of Cyclooxygenase-2 by the Activated MEKK1 → SEK1/MKK4 → p38 Mitogen-activated Protein Kinase Pathway* , 1998, The Journal of Biological Chemistry.

[7]  B. Pedersen,et al.  Evidence that interleukin‐6 is produced in human skeletal muscle during prolonged running , 1998, The Journal of physiology.

[8]  K. Yokoyama,et al.  Stretch Activates Jun N-terminal Kinase/Stress-activated Protein Kinase in Vascular Smooth Muscle Cells through Mechanisms Involving Autocrine ATP Stimulation of Purinoceptors* , 1998, The Journal of Biological Chemistry.

[9]  Jiahuai Han,et al.  Cardiac Hypertrophy Induced by Mitogen-activated Protein Kinase Kinase 7, a Specific Activator for c-Jun NH2-terminal Kinase in Ventricular Muscle Cells* , 1998, The Journal of Biological Chemistry.

[10]  S. Logan,et al.  Antagonism of glucocorticoid receptor transcriptional activation by the c-Jun N-terminal kinase. , 1998, Proceedings of the National Academy of Sciences of the United States of America.

[11]  J. Westermarck,et al.  Enhancement of Fibroblast Collagenase (Matrix Metalloproteinase-1)Gene Expression by Ceramide Is Mediated by Extracellular Signal-regulated and Stress-activated Protein Kinase Pathways* , 1998, The Journal of Biological Chemistry.

[12]  M. Jordana,et al.  IL-6 is an antiinflammatory cytokine required for controlling local or systemic acute inflammatory responses. , 1998, The Journal of clinical investigation.

[13]  E Ruoslahti,et al.  Extracellular signal-regulated kinase and c-Jun NH2-terminal kinase activation by mechanical stretch is integrin-dependent and matrix-specific in rat cardiac fibroblasts. , 1998, The Journal of clinical investigation.

[14]  E. Nishida,et al.  A novel SAPK/JNK kinase, MKK7, stimulated by TNFα and cellular stresses , 1997, The EMBO journal.

[15]  S. Dufresne,et al.  Contractile Activity Stimulates the c-Jun NH2-terminal Kinase Pathway in Rat Skeletal Muscle* , 1997, The Journal of Biological Chemistry.

[16]  B. Pedersen,et al.  Exercise‐induced increase in serum interleukin‐6 in humans is related to muscle damage. , 1997, The Journal of physiology.

[17]  M. Karin,et al.  The Ras-JNK pathway is involved in shear-induced gene expression , 1996, Molecular and cellular biology.

[18]  D. Moller,et al.  Effects of exercise and insulin on mitogen-activated protein kinase signaling pathways in rat skeletal muscle. , 1996, The American journal of physiology.

[19]  S. Pyne,et al.  Sphingomyelin-derived lipids differentially regulate the extracellular signal-regulated kinase 2 (ERK-2) and c-Jun N-terminal kinase (JNK) signal cascades in airway smooth muscle. , 1996, European journal of biochemistry.

[20]  M. Karin.  The Regulation of AP-1 Activity by Mitogen-activated Protein Kinases (*) , 1995, The Journal of Biological Chemistry.

[21]  M. Karin,et al.  Identification of a dual specificity kinase that activates the Jun kinases and p38-Mpk2. , 1995, Science.

[22]  Jiahuai Han,et al.  Pro-inflammatory Cytokines and Environmental Stress Cause p38 Mitogen-activated Protein Kinase Activation by Dual Phosphorylation on Tyrosine and Threonine (*) , 1995, The Journal of Biological Chemistry.

[23]  B. Dérijard,et al.  Transcription factor ATF2 regulation by the JNK signal transduction pathway , 1995, Science.

[24]  Michel Morange,et al.  A novel kinase cascade triggered by stress and heat shock that stimulates MAPKAP kinase-2 and phosphorylation of the small heat shock proteins , 1994, Cell.

[25]  R. Davis,et al.  An osmosensing signal transduction pathway in mammalian cells. , 1994, Science.

[26]  J. Woodgett,et al.  The stress-activated protein kinase subfamily of c-Jun kinases , 1994, Nature.

[27]  S. Akira,et al.  Interleukin-6 and its receptor: a paradigm for cytokines. , 1992, Science.

[28]  James R. Woodgett,et al.  Phosphorylation of c-jun mediated by MAP kinases , 1991, Nature.

[29]  R. Fielding,et al.  Acute phase response in exercise: interaction of age and vitamin E on neutrophils and muscle enzyme release. , 1990, The American journal of physiology.

[30]  B H Jones,et al.  Metabolic changes following eccentric exercise in trained and untrained men. , 1986, Journal of applied physiology.

[31]  K. R. Mills,et al.  Ultrastructural changes after concentric and eccentric contractions of human muscle , 1983, Journal of the Neurological Sciences.

[32]  M. Sjöström,et al.  Adaptive Response in Human Skeletal Muscle Subjected to Prolonged Eccentric Training , 1983, International journal of sports medicine.

[33]  D. Jones,et al.  Large delayed plasma creatine kinase changes after stepping exercise , 1983, Muscle & nerve.

[34]  M. M. Bradford.  A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. , 1976, Analytical biochemistry.

[35]  C. Widmann,et al.  Mitogen-activated protein kinase: conservation of a three-kinase module from yeast to human. , 1999, Physiological reviews.

[36]  E. Asmussen,et al.  Observations on experimental muscular soreness. , 1956, Acta rheumatologica Scandinavica.