论文信息 - Acute areflexic paralysis. Association with hyperalimentation and hypophosphatemia.

Acute areflexic paralysis. Association with hyperalimentation and hypophosphatemia.

Acute areflexic paralysis associated with diffuse sensory loss, cranial nerve paisies, and respiratory insufficiency occurred in two patients who developed hypophosphatemia during hyperalimentation. Prompt recovery followed replacement of serum phosphorus in both cases. An electromyogram performed on one patient revealed only decreased insertional activity. A muscle biopsy specimen from the same patient showed minor, nonspecific neurogenic changes.

R. Farmer | A. Cooperman | A. Furlan | M. W. Hanson

[1] J. Cr. Letter: The echocardiogram in asymmetrical septal hypertrophy. , 1974 .

[2] S E Silvis,et al. Paresthesias, weakness, seizures, and hypophosphatemia in patients receiving hyperalimentation. , 1972, Gastroenterology.

[3] S. Silvis,et al. Fatal hyperalimentation syndrome. Animal studies. , 1971, The Journal of laboratory and clinical medicine.

[4] M. Delivoria-Papadopoulos,et al. Alterations of red-cell glycolytic intermediates and oxygen transport as a consequence of hypophosphatemia in patients receiving intravenous hyperalimentation. , 1971, The New England journal of medicine.

[5] M. Lichtman,et al. Reduced red cell glycolysis, 2, 3-diphosphoglycerate and adenosine triphosphate concentration, and increased hemoglobin-oxygen affinity caused by hypophosphatemia. , 1971, Annals of internal medicine.

[6] F. Bartter,et al. Evidence for a phosphorus-depletion syndrome in man. , 1968, The New England journal of medicine.

[7] C. Aubel,et al. The effects of low-phosphorus rations on growing pigs. , 1936 .