Coronary surgery without cardiotomy suction and autotransfusion reduces the postoperative systemic inflammatory response.

[1]  J. Steinke,et al.  2. Cytokines and chemokines. , 2003, The Journal of allergy and clinical immunology.

[2]  K. Engström,et al.  The inflammatory response to recycled pericardial suction blood and the influence of cell-saving , 2003, Scandinavian cardiovascular journal : SCJ.

[3]  A. Larsson,et al.  Coagulation, fibrinolysis, and cell activation in patients and shed mediastinal blood during coronary artery bypass grafting with a new heparin-coated surface. , 2002, The Journal of thoracic and cardiovascular surgery.

[4]  C. Triggs,et al.  Limitation of thrombin generation, platelet activation, and inflammation by elimination of cardiotomy suction in patients undergoing coronary artery bypass grafting treated with heparin-bonded circuits. , 2002, The Journal of thoracic and cardiovascular surgery.

[5]  J. Pincemail,et al.  Levels of inflammatory markers in the blood processed by autotransfusion devices during cardiac surgery associated with cardiopulmonary bypass circuit , 2002, Perfusion.

[6]  T. Yau,et al.  Cardiopulmonary bypass induced inflammation: pathophysiology and treatment. An update. , 2002, European journal of cardio-thoracic surgery : official journal of the European Association for Cardio-thoracic Surgery.

[7]  J. Murkin.  Attenuation of neurologic injury during cardiac surgery. , 2001, The Annals of thoracic surgery.

[8]  V. Richard,et al.  Complement and its implications in cardiac ischemia/reperfusion: strategies to inhibit complement , 2001, Fundamental & clinical pharmacology.

[9]  J. Hammon,et al.  Processing scavenged blood with a cell saver reduces cerebral lipid microembolization. , 2000, The Annals of thoracic surgery.

[10]  Schmucker,et al.  Perioperative serum levels of tumour‐necrosis‐factor alpha (TNF‐α), IL‐1β, IL‐6, IL‐10 and soluble IL‐2 receptor in patients undergoing cardiac surgery with cardiopulmonary bypass without and with correction for haemodilution , 1999, Clinical and experimental immunology.

[11]  G. Tangen,et al.  Autotransfusion in coronary artery bypass grafting: disparity in laboratory tests and clinical performance. , 1999, The Journal of thoracic and cardiovascular surgery.

[12]  W. Reents,et al.  Influence of different autotransfusion devices on the quality of salvaged blood. , 1999, The Annals of thoracic surgery.

[13]  S. Zahler,et al.  Acute cardiac inflammatory responses to postischemic reperfusion during cardiopulmonary bypass. , 1999, Cardiovascular research.

[14]  D. Reboussin,et al.  Cardiotomy suction: a major source of brain lipid emboli during cardiopulmonary bypass. , 1998, The Annals of thoracic surgery.

[15]  J. Vincent,et al.  Inflammatory response to cardiopulmonary bypass: mechanisms involved and possible therapeutic strategies. , 1997, Chest.

[16]  W. van Oeveren,et al.  Retransfusion of suctioned blood during cardiopulmonary bypass impairs hemostasis. , 1995, The Annals of thoracic surgery.

[17]  S Westaby,et al.  Inflammatory response to cardiopulmonary bypass. , 1993, The Annals of thoracic surgery.

[18]  W. van Oeveren,et al.  Endotoxin release and tumor necrosis factor formation during cardiopulmonary bypass. , 1992, The Annals of thoracic surgery.

[19]  Xiang Xiaoyong,et al.  Cardiopulmonary bypass induced inflammation: pathophysiology and treatment , 2005 .

[20]  Larry Borish,et al.  Cytokines and chemokines , 2003 .

[21]  W. van Oeveren,et al.  Retransfusion of thoracic wound blood during heart surgery obscures biocompatibility of the extracorporeal circuit. , 1996, The Journal of thoracic and cardiovascular surgery.