Protective effects of FUT-175 on acute massive hepatic necrosis induced in mice following endotoxin injection and immunization with liver proteins.

[1]  A. Tanner,et al.  The influence of endotoxin in vitro on hepatic macrophage lysosomal enzyme release in different rat models of hepatic injury. , 2008, Liver.

[2]  T. Uede,et al.  Flow cytometric and functional analysis of mononuclear cells infiltrating the liver in experimental autoimmune hepatitis , 1990, Clinical and experimental immunology.

[3]  Y. Shiratori,et al.  Role of endotoxin‐responsive macrophages in hepatic injury , 1990, Hepatology.

[4]  J. Nolan.  Intestinal endotoxins as mediators of hepatic injury—An idea whose time has come again , 1989, Hepatology.

[5]  J. Wallace,et al.  Endotoxin‐induced ascites formation in the rat: Partial mediation by platelet‐activating factor , 1989, Hepatology.

[6]  D. Thiele.  Tumor necrosis factor, the acute phase response and the pathogenesis of alcoholic liver disease , 1989, Hepatology.

[7]  M. Arthur,et al.  Effect of endotoxin on release of reactive oxygen intermediates by rat hepatic macrophages. , 1988, Gastroenterology.

[8]  T. Mori,et al.  Adoptive transfer of experimental autoimmune hepatitis in mice--cellular interaction between donor and recipient mice. , 1988, Clinical and Experimental Immunology.

[9]  R. Soejima,et al.  Gabexate as a therapy for disseminated intravascular coagulation. , 1988, Archives of internal medicine.

[10]  T. Mori,et al.  Inhibitory effects of prostaglandin E1 on T-cell mediated cytotoxicity against isolated mouse liver cells. , 1988, Gastroenterology.

[11]  S. Seki,et al.  The protective effects of prostaglandin E1 in an experimental massive hepatic cell necrosis model , 1987, Hepatology.

[12]  C. Ryan,et al.  Chymotrypsin-specific protease inhibitors decrease H2O2 formation by activated human polymorphonuclear leukocytes. , 1987, Carcinogenesis.

[13]  C. Galanos,et al.  Lethal toxicity of lipopolysaccharide and tumor necrosis factor in normal and D-galactosamine-treated mice , 1987, The Journal of experimental medicine.

[14]  M. Arthur,et al.  Oxygen-derived free radicals promote hepatic injury in the rat. , 1985, Gastroenterology.

[15]  T. Mori,et al.  Study of cellular immunity in experimental autoimmune hepatitis in mice: transfer of spleen cells sensitized with liver proteins. , 1985, Clinical and experimental immunology.

[16]  H. Yoshida,et al.  Cell‐mediated cytotoxicity of sensitized spleen cells against target liver cells—in vivo and in vitro study with a mouse model of experimental autoimmune hepatitis , 1985, Hepatology.

[17]  S. Ikehara,et al.  Effect of FUT-175, a new synthetic protease inhibitor, on the development of lupus nephritis in (NZB x NZW) F1 mice. , 1985, Immunology.

[18]  T. Nakamura,et al.  Increased survival of rat hepatocytes in serum-free medium by inhibition of a trypsin-like protease associated with their plasma membranes. , 1984, Experimental cell research.

[19]  L. Minning,et al.  The requirement for proteinase activity for human lymphocyte-mediated natural cytotoxicity (NK): evidence that the proteinase is serine dependent and has aromatic amino acid specificity of cleavage. , 1984, Journal of immunology.

[20]  T. Mori,et al.  Study of cellular immunity in experimental autoimmune hepatitis in mice. , 1984, Clinical and experimental immunology.

[21]  A. Kirn,et al.  Probable role of endogenous endotoxins in hepatocytolysis during murine hepatitis caused by frog virus 3. , 1984, The Journal of infectious diseases.

[22]  S. Fujii,et al.  Inhibition of various immunological reactions in vivo by a new synthetic complement inhibitor. , 1982, International archives of allergy and applied immunology.

[23]  S. Fujii,et al.  New synthetic inhibitors of C1r, C1 esterase, thrombin, plasmin, kallikrein and trypsin. , 1981, Biochimica et biophysica acta.

[24]  N. Aoki,et al.  Acute hepatic cell necrosis experimentally produced by viral agents in rabbits. , 1981, The American journal of pathology.

[25]  G. Holdstock,et al.  Proteolytic enzymes released by liver macrophages may promote hepatic injury in a rat model of hepatic damage. , 1981, Gastroenterology.

[26]  A. Allison,et al.  Identification of human mononuclear leucocyte populations by esterase staining. , 1977, Clinical and experimental immunology.

[27]  P. Seglen.  Preparation of rat liver cells. I. Effect of Ca 2+ on enzymatic dispersion of isolated, perfused liver. , 1972, Experimental cell research.

[28]  H. E. Noyes,et al.  Experimental Shigella Infections. III. Sensitivity of Normal, Starved and Carbon Tetrachloride Treated Guinea Pigs to Endotoxin , 1960, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

[29]  H. Demuth.  Recent developments in inhibiting cysteine and serine proteases. , 1990, Journal of enzyme inhibition.

[30]  J. Fierer,et al.  D-Galactosamine hepatotoxicity is associated with endotoxin sensitivity and mediated by lymphoreticular cells in mice. , 1985, Gastroenterology.

[31]  Nolan Jp,et al.  Endotoxin, sinusoidal cells, and liver injury. , 1982 .

[32]  P. Seglen.  Preparation of rat liver cells. II. Effects of ions and chelators on tissue dispersion. , 1973, Experimental cell research.

[33]  Nolan Jp,et al.  Endotoxin and the liver. II. Effect of tolerance on carbon tetrachloride-induced injury. , 1973 .

[34]  A. Karmen,et al.  Transaminase activity in human blood. , 1955, The Journal of clinical investigation.