Central carbon metabolism in the progression of mammary carcinoma
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Adam D. Richardson | Jeffrey W. Smith | A. Osterman | A. Richardson | Chen Yang | Chen Yang | Andrei Osterman
[1] F. Miller,et al. Profiling the progression of cancer: Separation of microsomal proteins in MCF10 breast epithelial cell lines using nonporous chromatophoresis , 2003, Proteomics.
[2] J. Schouten,et al. High-resolution mapping of molecular events associated with immortalization, transformation, and progression to breast cancer in the MCF10 model , 2006, Breast Cancer Research and Treatment.
[3] Chi V Dang,et al. Cancer's molecular sweet tooth and the Warburg effect. , 2006, Cancer research.
[4] T. Ferenci,et al. Global metabolite analysis: the influence of extraction methodology on metabolome profiles of Escherichia coli. , 2003, Analytical biochemistry.
[5] F. Kuhajda,et al. Fatty acid synthase and cancer: new application of an old pathway. , 2006, Cancer research.
[6] F. Miller. Xenograft Models of Premalignant Breast Disease , 2000, Journal of Mammary Gland Biology and Neoplasia.
[7] Marta Cascante,et al. K-ras codon-specific mutations produce distinctive metabolic phenotypes in NIH3T3 mice [corrected] fibroblasts. , 2005, Cancer research.
[8] Marta Cascante,et al. K-ras Codon-Specific Mutations Produce Distinctive Metabolic Phenotypes in Human Fibroblasts , 2005 .
[9] Chen Yang,et al. Comparative Metabolomics of Breast Cancer , 2006, Pacific Symposium on Biocomputing.
[10] Jeffrey W. Smith,et al. Orlistat Is a Novel Inhibitor of Fatty Acid Synthase with Antitumor Activity , 2004, Cancer Research.
[11] D. Schoeller,et al. Measurement of fractional lipid synthesis using deuterated water (2H2O) and mass isotopomer analysis. , 1994, The American journal of physiology.
[12] E. Gabrielson,et al. Regulation of fatty acid synthase expression in breast cancer by sterol regulatory element binding protein-1c. , 2003, Experimental cell research.
[13] S. Marshall. Role of Insulin, Adipocyte Hormones, and Nutrient-Sensing Pathways in Regulating Fuel Metabolism and Energy Homeostasis: A Nutritional Perspective of Diabetes, Obesity, and Cancer , 2006, Science's STKE.
[14] L. Gille,et al. The Exogenous NADH Dehydrogenase of Heart Mitochondria Is the Key Enzyme Responsible for Selective Cardiotoxicity of Anthracyclines , 1998, Zeitschrift fur Naturforschung. C, Journal of biosciences.
[15] M. Guppy,et al. Cancer metabolism: facts, fantasy, and fiction. , 2004, Biochemical and biophysical research communications.
[16] K. Conklin. Cancer chemotherapy and antioxidants. , 2004, The Journal of nutrition.
[17] F. Berrino,et al. Erythrocyte membrane fatty acids and subsequent breast cancer: a prospective Italian study. , 2001, Journal of the National Cancer Institute.
[18] F. Miller,et al. MCF10AT: a model for the evolution of cancer from proliferative breast disease. , 1996, The American journal of pathology.
[19] J. Warrington,et al. Identification of a Mammalian Long Chain Fatty Acyl Elongase Regulated by Sterol Regulatory Element-binding Proteins* , 2001, The Journal of Biological Chemistry.
[20] T. Kurosu,et al. BCL6 overexpression prevents increase in reactive oxygen species and inhibits apoptosis induced by chemotherapeutic reagents in B-cell lymphoma cells , 2003, Oncogene.
[21] Fred R. Miller,et al. Malignant MCF10CA1 Cell Lines Derived from Premalignant Human Breast Epithelial MCF10AT Cells , 2004, Breast Cancer Research and Treatment.
[22] F. Kuhajda. Fatty-acid synthase and human cancer: new perspectives on its role in tumor biology. , 2000, Nutrition.
[23] J R Griffiths,et al. Accumulation of Krebs cycle intermediates and over-expression of HIF1alpha in tumours which result from germline FH and SDH mutations. , 2005, Human molecular genetics.
[24] Huasheng Lu,et al. Hypoxia-inducible Factor 1 Activation by Aerobic Glycolysis Implicates the Warburg Effect in Carcinogenesis* , 2002, The Journal of Biological Chemistry.
[25] J. Yodoi,et al. Cellular levels of thioredoxin associated with drug sensitivity to cisplatin, mitomycin C, doxorubicin, and etoposide. , 1995, Cancer research.
[26] Michael E Phelps,et al. Positron emission tomography scanning: current and future applications. , 2002, Annual review of medicine.
[27] P. Hammerman,et al. Cytokine stimulation of aerobic glycolysis in hematopoietic cells exceeds proliferative demand , 2004, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[28] W. Aclimandos,et al. Erythrocyte stearic to oleic acid ratio in patients with ocular melanoma , 1992, Eye.
[29] E. Heinzle,et al. Mass spectrometry for metabolic flux analysis. , 1999, Biotechnology and bioengineering.
[30] Eyal Gottlieb,et al. Succinate links TCA cycle dysfunction to oncogenesis by inhibiting HIF-alpha prolyl hydroxylase. , 2005, Cancer cell.
[31] J. Swinnen,et al. Increased lipogenesis in cancer cells: new players, novel targets , 2006, Current opinion in clinical nutrition and metabolic care.
[32] O. Warburg,et al. THE METABOLISM OF TUMORS IN THE BODY , 1927, The Journal of general physiology.
[33] M. Lippman,et al. Toxicity and effects of epidermal growth factor on glucose metabolism of MDA-468 human breast cancer cells. , 1990, The Journal of biological chemistry.
[34] R. A. Igal,et al. Stearoyl-CoA Desaturase Is Involved in the Control of Proliferation, Anchorage-independent Growth, and Survival in Human Transformed Cells* , 2005, Journal of Biological Chemistry.
[35] Marta Cascante,et al. Mass isotopomer study of the nonoxidative pathways of the pentose cycle with [1,2-13C2]glucose. , 1998, American journal of physiology. Endocrinology and metabolism.
[36] J. Klaunig,et al. The role of oxidative stress in carcinogenesis. , 2004, Annual review of pharmacology and toxicology.
[37] A. Klein-Szanto,et al. Transformation of Human Breast Epithelial Cells by c‐Ha‐ras Oncogene , 1991, Molecular carcinogenesis.
[38] Adam L. Meadows,et al. Estradiol stimulates the biosynthetic pathways of breast cancer cells: detection by metabolic flux analysis. , 2006, Metabolic engineering.
[39] David G. Watson,et al. Succinate links TCA cycle dysfunction to oncogenesis by inhibiting HIF-alpha prolyl hydroxylase. , 2005, Cancer cell.