The selection of tumor variants with altered expression of classical and nonclassical MHC class I molecules: implications for tumor immune escape
暂无分享,去创建一个
Federico Garrido | F. Garrido | F. Ruiz-Cabello | T. Cabrera | A. Garcia-Lora | I. Algarra | Teresa Cabrera | Ignacio Algarra | Angel García-Lora | Francisco Ruiz-Cabello | Angel M. Garcia-Lora
[1] P. Bouteiller. HLA‐G: ON THE TRACK OF IMMUNOLOGICAL FUNCTIONS , 1997 .
[2] J. Yokota,et al. Tumor progression and metastasis. , 2000, Carcinogenesis.
[3] D. Pardoll,et al. Does the immune system see tumors as foreign or self? , 2003, Annual review of immunology.
[4] R. Prehn,et al. Immunity to methylcholanthrene-induced sarcomas. , 1957, Journal of the National Cancer Institute.
[5] X. Sastre,et al. Differences in the antigens recognized by cytolytic T cells on two successive metastases of a melanoma patient are consistent with immune selection , 1995, European journal of immunology.
[6] Takuma Hayashi,et al. Development of spontaneous uterine tumors in low molecular mass polypeptide-2 knockout mice. , 2002, Cancer research.
[7] B. Drénou,et al. HLA-G protein expression is not induced during malignant transformation. , 1999, Tissue antigens.
[8] F. Garrido,et al. MHC antigens and tumor escape from immune surveillance. , 2001, Advances in cancer research.
[9] F. Garrido,et al. MHC class I‐deficient metastatic tumor variants immunoselected by T lymphocytes originate from the coordinated downregulation of APM components , 2003, International journal of cancer.
[10] R. Schreiber,et al. Demonstration of an interferon gamma-dependent tumor surveillance system in immunocompetent mice. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[11] I. Svane,et al. MCA Sarcomas Induced in scid Mice are More Immunogenic than MCA Sarcomas Induced in Congenic, Immunocompetent Mice , 1997, Scandinavian journal of immunology.
[12] R. Schreiber,et al. IFNγ and lymphocytes prevent primary tumour development and shape tumour immunogenicity , 2001, Nature.
[13] S. Bauer,et al. Recognition of stress-induced MHC molecules by intestinal epithelial gammadelta T cells. , 1998, Science.
[14] P. Bruggen,et al. T cell defined tumor antigens , 1997 .
[15] A Steinle,et al. Activation of NK cells and T cells by NKG2D, a receptor for stress-inducible MICA. , 1999, Science.
[16] F. Garrido,et al. Influence of class I H-2 gene expression on local tumor growth. Description of a model obtained from clones derived from a solid BALB/c tumor. , 1986, Experimental and clinical immunogenetics.
[17] N. Restifo,et al. Assumptions of the tumor 'escape' hypothesis. , 2002, Seminars in cancer biology.
[18] D. Templeton,et al. Deficiency of transporter for antigen presentation (TAP) in tumor cells allows evasion of immune surveillance and increases tumorigenesis. , 1999, Journal of immunology.
[19] F. Oesch,et al. Inverse relationship of melanocyte differentiation antigen expression in melanoma tissues and CD8+ cytotoxic‐T‐cell responses: Evidence for immunoselection of antigen‐loss variants in vivo , 1996, International journal of cancer.
[20] F. Garrido,et al. Immunoselection by T lymphocytes generates repeated MHC class I‐deficient metastatic tumor variants , 2001, International Journal of Cancer.
[21] J. Roder,et al. Spontaneous and induced primary oncogenesis in natural killer (nk)‐cell‐deficient beige mutant mice , 1985, International journal of cancer.
[22] F. Garrido,et al. Analysis of HLA class I expression in different metastases from two melanoma patients undergoing peptide immunotherapy. , 2001, Tissue antigens.
[23] P. Stern,et al. Natural history of HLA expression during tumour development. , 1993, Immunology today.
[24] R. Schreiber,et al. Cancer immunoediting: from immunosurveillance to tumor escape , 2002, Nature Immunology.
[25] F. Garrido,et al. Expression of HLA G in human tumors is not a frequent event , 1999, International journal of cancer.
[26] Hans Hengartner,et al. Cytotoxicity mediated by T cells and natural killer cells is greatly impaired in perforin-deficient mice , 1994, Nature.
[27] M. Llano,et al. HLA‐E‐bound peptides influence recognition by inhibitory and triggering CD94/NKG2 receptors: preferential response to an HLA‐G‐derived nonamer , 1998, European journal of immunology.
[28] O. Stutman. Chemical carcinogenesis in nude mice: comparison between nude mice from homozygous matings and heterozygous matings and effect of age and carcinogen dose. , 1979, Journal of the National Cancer Institute.
[29] P. Stern,et al. Implications for immunosurveillance of altered HLA class I phenotypes in human tumours. , 1997, Immunology today.
[30] I. Svane,et al. CD8+ T cells are crucial for the ability of congenic normal mice to reject highly immunogenic sarcomas induced in nude mice with 3‐methylcholanthrene , 2000, Clinical and experimental immunology.
[31] Lloyd J. Old,et al. and shape tumour immunogenicity , 2001 .
[32] T. Boon,et al. Tumor antigens recognized by T lymphocytes , 1997, International journal of clinical & laboratory research.
[33] A. McMichael,et al. Surface expression of HLA-E, an inhibitor of natural killer cells, enhanced by human cytomegalovirus gpUL40. , 2000, Science.
[34] H. Ljunggren,et al. Chemically induced sarcomas from nude mice are more immunogenic than similar sarcomas from congenic normal mice , 1996, European journal of immunology.
[35] M. Wislez,et al. Pulmonary Malignancies in the Immunocompromised Patient , 1999, Respiration.
[36] J. Dausset,et al. HLA-G expression in melanoma: a way for tumor cells to escape from immunosurveillance. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[37] E. Seregni,et al. Immunosuppressive Factors: Role in Cancer Development and Progression , 1998, The International journal of biological markers.
[38] A. Hayday,et al. Regulation of Cutaneous Malignancy by γδ T Cells , 2001, Science.
[39] A. McMichael,et al. The human major histocompatibility complex class Ib molecule HLA‐E binds signal sequence‐derived peptides with primary anchor residues at positions 2 and 9 , 1997, European journal of immunology.
[40] Federico Garrido,et al. MHC class I antigens, immune surveillance, and tumor immune escape , 2003, Journal of cellular physiology.
[41] I. Ahmed,et al. Angiosarcoma in a chronically immunosuppressed renal transplant recipient: report of a case and review of the literature. , 2002, The American Journal of dermatopathology.
[42] F. Burnet. The concept of immunological surveillance. , 1970, Progress in experimental tumor research.
[43] G. Klein,et al. A weakly tumorigenic phenotype with high mhc class‐i expression is associated with high metastatic potential after surgical removal of the primary murine fibrosarcoma , 1990, International journal of cancer.
[44] R. Zinkernagel,et al. Perforin dependence of natural killer cell‐mediated tumor control in vivo , 1995, European journal of immunology.
[45] P. Stern. Immunity to human papillomavirus-associated cervical neoplasia. , 1996, Advances in cancer research.
[46] M. Pittelkow,et al. Skin cancer in liver transplant recipients , 2000, Liver transplantation : official publication of the American Association for the Study of Liver Diseases and the International Liver Transplantation Society.
[47] M. Smyth,et al. NK cells and NKT cells collaborate in host protection from methylcholanthrene-induced fibrosarcoma. , 2001, International immunology.
[48] Wancai Yang,et al. γδ T Cells Provide an Early Source of Interferon γ in Tumor Immunity , 2003, The Journal of experimental medicine.
[49] R. Biassoni,et al. Receptors for HLA class-I molecules in human natural killer cells. , 1996, Annual review of immunology.
[50] K. McClain. Immunodeficiency states and related malignancies. , 1997, Cancer treatment and research.
[51] P. Robbins,et al. A listing of human tumor antigens recognized by T cells , 2001, Cancer Immunology, Immunotherapy.
[52] I. Algarra,et al. Heterogeneity of MHC‐class‐I antigens in clones of methylcholanthrene‐induced tumors. Implications for local growth and metastasis , 1991, International journal of cancer. Supplement = Journal international du cancer. Supplement.
[53] B. Seliger,et al. The role of HLA-G for protection of human renal cell-carcinoma cells from immune-mediated lysis: implications for immunotherapies. , 2003, Seminars in Cancer Biology.
[54] R. Schreiber,et al. Demonstration of an interferon γ-dependent tumor surveillance system in immunocompetent mice , 1998 .
[55] J. Trapani,et al. Perforin-Mediated Cytotoxicity Is Critical for Surveillance of Spontaneous Lymphoma , 2000, The Journal of experimental medicine.
[56] G. Ferrara,et al. Melanomas and melanoma cell lines do not express HLA-G, and the expression cannot be induced by gammaIFN treatment. , 2000, Tissue antigens.
[57] D. Geraghty,et al. Analysis of HLA-E expression in human tumors , 2003, Immunogenetics.
[58] S. Riddell,et al. Costimulation of CD8αβ T cells by NKG2D via engagement by MIC induced on virus-infected cells , 2001, Nature Immunology.
[59] M. Smyth,et al. Perforin and interferon-gamma activities independently control tumor initiation, growth, and metastasis. , 2001, Blood.
[60] C. Ulrich,et al. Association of human papillomavirus infections with cutaneous tumors in immunosuppressed patients , 2003, Transplant international : official journal of the European Society for Organ Transplantation.
[61] A. Hayday,et al. The Distinct Contributions of Murine T Cell Receptor (TCR)γδ+ and TCRαβ+ T Cells to Different Stages of Chemically Induced Skin Cancer , 2003, The Journal of experimental medicine.
[62] B. Seliger,et al. HLA class I antigen abnormalities and immune escape by malignant cells. , 2002, Seminars in cancer biology.
[63] A. Hayday,et al. Regulation of cutaneous malignancy by gammadelta T cells. , 2001, Science.
[64] A. McMichael,et al. The epitopes of influenza nucleoprotein recognized by cytotoxic T lymphocytes can be defined with short synthetic peptides , 1986, Cell.