Differentially expressed alternatively spliced genes in Malignant Pleural Mesothelioma identified using massively parallel transcriptome sequencing

[1]  Francisco M. De La Vega,et al.  Sequence and structural variation in a human genome uncovered by short-read, massively parallel ligation sequencing using two-base encoding. , 2009, Genome research.

[2]  Sherif Abou Elela,et al.  Identification of alternative splicing markers for breast cancer. , 2008, Cancer research.

[3]  Anne-Mette K. Hein,et al.  Alternative Splicing in Colon, Bladder, and Prostate Cancer Identified by Exon Array Analysis*S , 2008, Molecular & Cellular Proteomics.

[4]  R. Hayes,et al.  Variation in the Selenoenzyme Genes and Risk of Advanced Distal Colorectal Adenoma , 2008, Cancer Epidemiology Biomarkers & Prevention.

[5]  S. Blain.  Switching cyclin D-Cdk4 kinase activity on and off , 2008, Cell cycle.

[6]  Neil A. Miller,et al.  Transcriptome sequencing of malignant pleural mesothelioma tumors , 2008, Proceedings of the National Academy of Sciences.

[7]  Byung-Hoon Lee,et al.  Gene expression profiling in human lung fibroblast following cadmium exposure. , 2008, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.

[8]  J. Venables,et al.  Multiple alternative splicing markers for ovarian cancer. , 2008, Cancer research.

[9]  Elias S. J. Arnér,et al.  Induction of Cell Membrane Protrusions by the N-terminal Glutaredoxin Domain of a Rare Splice Variant of Human Thioredoxin Reductase 1*♦ , 2008, Journal of Biological Chemistry.

[10]  M. Loh,et al.  Differential Expression of Novel Tyrosine Kinase Substrates during Breast Cancer Development *S , 2007, Molecular & Cellular Proteomics.

[11]  Edgar Erdfelder,et al.  G*Power 3: A flexible statistical power analysis program for the social, behavioral, and biomedical sciences , 2007, Behavior research methods.

[12]  Crispin Miller,et al.  Clinical quantitation of diagnostic and predictive gene expression levels in follicular and diffuse large B-cell lymphoma by RT-PCR gene expression profiling. , 2007, Blood.

[13]  L. Hawthorn,et al.  The application of microarray technology to the analysis of the cancer genome. , 2007, Current molecular medicine.

[14]  Laura Smith,et al.  Expression microarray analysis reveals genes associated with in vitro resistance to cisplatin in a cell line model , 2007, Acta oncologica.

[15]  J. Thierry-Mieg,et al.  AceView: a comprehensive cDNA-supported gene and transcripts annotation , 2006, Genome Biology.

[16]  E. Buratti,et al.  Defective splicing, disease and therapy: searching for master checkpoints in exon definition , 2006, Nucleic acids research.

[17]  Maurice P H M Jansen,et al.  Molecular profiling of platinum resistant ovarian cancer , 2006, International journal of cancer.

[18]  Marco Di Giovanni,et al.  Similar Tumor Suppressor Gene Alteration Profiles in Asbestos-Induced Murine and Human Mesothelioma , 2005, Cell cycle.

[19]  James R. Knight,et al.  Genome sequencing in microfabricated high-density picolitre reactors , 2005, Nature.

[20]  Christopher J. Lee,et al.  Evidence that public database records for many cancer-associated genes reflect a splice form found in tumors and lack normal splice forms , 2005, Nucleic acids research.

[21]  J. Venables.  Aberrant and Alternative Splicing in Cancer , 2004, Cancer Research.

[22]  Martin Vingron,et al.  Genome wide identification and classification of alternative splicing based on EST data , 2004, Bioinform..

[23]  Tadashi Kondo,et al.  Alternative splice variant of actinin-4 in small cell lung cancer , 2004, Oncogene.

[24]  J. Castle,et al.  Genome-Wide Survey of Human Alternative Pre-mRNA Splicing with Exon Junction Microarrays , 2003, Science.

[25]  J. Knezetic,et al.  A hMLH1 genomic mutation and associated novel mRNA defects in a hereditary non-polyposis colorectal cancer family. , 2002, Mutation research.

[26]  Christopher J. Lee,et al.  Genome-wide detection of alternative splicing in expressed sequences of human genes , 2001, Nucleic Acids Res..

[27]  D. Weiss,et al.  Mutational analysis of N-ras, p53, p16INK4a, p14ARF and CDK4 genes in primary human malignant mesotheliomas. , 2001, International journal of oncology.

[28]  J. Quinn,et al.  A splice variant of the neuron-restrictive silencer factor repressor is expressed in small cell lung cancer: a potential role in derepression of neuroendocrine genes and a useful clinical marker. , 2000, Cancer research.

[29]  E. Lyon,et al.  Implications of a novel cryptic splice site in the BRCA1 gene. , 1998, American journal of medical genetics.

[30]  B. Ripley,et al.  Modern Applied Statistics with S-Plus. , 1996 .

[31]  G. Powis,et al.  Cloning and sequencing of a human thioredoxin reductase , 1995, FEBS letters.

[32]  David A. James.  Modern Applied Statistics With S-PLUS , 1994 .

[33]  G. Bruns,et al.  Structure, chromosome location, and expression of the human smooth muscle (enteric type) gamma-actin gene: evolution of six human actin genes , 1991, Molecular and cellular biology.

[34]  M. Garcia-Blanco.  Alternative splicing: therapeutic target and tool. , 2006, Progress in molecular and subcellular biology.