Nociception increases during opioid infusion in opioid receptor triple knock-out mice

[1]  K. Rice,et al.  Opioid receptor-mediated hyperalgesia and antinociceptive tolerance induced by sustained opiate delivery , 2006, Neuroscience Letters.

[2]  B. Kest,et al.  Morphine hyperalgesia in mice is unrelated to opioid activity, analgesia, or tolerance: Evidence for multiple diverse hyperalgesic systems , 2006, Brain Research.

[3]  J. Pintar,et al.  Autoradiography in opioid triple knockout mice reveals opioid and opioid receptor like binding of naloxone benzoylhydrazone , 2005, Neuropharmacology.

[4]  T. Vanderah,et al.  Cholecystokinin in the Rostral Ventromedial Medulla Mediates Opioid-Induced Hyperalgesia and Antinociceptive Tolerance , 2005, The Journal of Neuroscience.

[5]  V. Hruby,et al.  Antinociceptive and nociceptive actions of opioids. , 2004, Journal of neurobiology.

[6]  J. Mogil,et al.  Modulation of morphine analgesia by site-specific N-methyl-d-aspartate receptor antagonists: dependence on sex, site of antagonism, morphine dose, and time , 2004, Pain.

[7]  F. Colpaert,et al.  Opioid hyperalgesia and tolerance versus 5-HT1A receptor-mediated inverse tolerance. , 2003, Trends in pharmacological sciences.

[8]  Maree T. Smith,et al.  Morphine-3-Glucuronide’s Neuro-Excitatory Effects Are Mediated via Indirect Activation of N-Methyl-d-Aspartic Acid Receptors: Mechanistic Studies in Embryonic Cultured Hippocampal Neurones , 2003, Anesthesia and analgesia.

[9]  S. C. Armstrong,et al.  Pharmacokinetic drug interactions of morphine, codeine, and their derivatives: theory and clinical reality, Part II. , 2003, Psychosomatics.

[10]  C. Rivat,et al.  Opioid-induced hyperalgesia: abnormal or normal pain? , 2003, Neuroreport.

[11]  J. Mogil,et al.  Genetic variation in morphine analgesic tolerance A survey of 11 inbred mouse strains , 2002, Pharmacology Biochemistry and Behavior.

[12]  J. Pintar,et al.  Autoradiography of opioid and ORL1 ligands in opioid receptor triple knockout mice , 2002, The European journal of neuroscience.

[13]  J. Mao,et al.  Chronic Morphine Induces Downregulation of Spinal Glutamate Transporters: Implications in Morphine Tolerance and Abnormal Pain Sensitivity , 2022 .

[14]  K. Shen,et al.  Cholera toxin-B subunit blocks excitatory opioid receptor-mediated hyperalgesic effects in mice, thereby unmasking potent opioid analgesia and attenuating opioid tolerance/dependence , 2001, Brain Research.

[15]  P. Popik,et al.  Uncompetitive NMDA receptor antagonists potentiate morphine antinociception recorded from the tail but not from the hind paw in rats. , 2001, European journal of pharmacology.

[16]  T. Vanderah,et al.  Tonic Descending Facilitation from the Rostral Ventromedial Medulla Mediates Opioid-Induced Abnormal Pain and Antinociceptive Tolerance , 2001, The Journal of Neuroscience.

[17]  E. Zhang,et al.  Dynorphin Promotes Abnormal Pain and Spinal Opioid Antinociceptive Tolerance , 2000, The Journal of Neuroscience.

[18]  A. Dahan,et al.  Gender Differences in Opioid-mediated Analgesia: Animal and Human Studies , 2000, Anesthesiology.

[19]  K. Shen,et al.  Antagonists of excitatory opioid receptor functions enhance morphine's analgesic potency and attenuate opioid tolerance/dependence liability , 2000, PAIN®.

[20]  S. Hansen,et al.  Is development of hyperalgesia, allodynia and myoclonus related to morphine metabolism during long‐term administration?: Six case histories , 1998, Acta anaesthesiologica Scandinavica.

[21]  U. Lendahl Transgenic analysis of central nervous system development and regeneration , 1997, Acta anaesthesiologica Scandinavica. Supplementum.

[22]  A. Basbaum,et al.  NMDA-receptor regulation of substance P release from primary afferent nociceptors , 1997, Nature.

[23]  D. Price,et al.  Mechanisms of hyperalgesian and morphine tolerance: a current view of their possible interactions , 1995, Pain.

[24]  Y. Jan,et al.  Evidence for presynaptic N-methyl-D-aspartate autoreceptors in the spinal cord dorsal horn. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[25]  P. Dodd,et al.  Pharmacology of morphine and morphine-3-glucuronide at opioid, excitatory amino acid, GABA and glycine binding sites. , 1994, Pharmacology & toxicology.

[26]  D. Price,et al.  Thermal hyperalgesia in association with the development of morphine tolerance in rats: roles of excitatory amino acid receptors and protein kinase C , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[27]  A. Caraceni,et al.  Hyperalgesia and myoclonus with intrathecal infusion of high-dose morphine , 1991, Pain.

[28]  N. Hanioka,et al.  Species difference of site-selective glucuronidation of morphine. , 1991, Journal of pharmacobio-dynamics.

[29]  N. Rawal,et al.  Clinical experience of long–term treatment with epidural and intrathecal opioids – a nationwide survey , 1988 .

[30]  T. Yaksh,et al.  High Doses of Spinal Morphine Produce a Nonopiate Receptormediated Hyperesthesia: Clinical and Theoretic Implications , 1986, Anesthesiology.

[31]  M. Kavaliers,et al.  Daily rhythms of analgesia in mice: effects of age and photoperiod , 1983, Brain Research.

[32]  C. Woolf Intrathecal high dose morphine produces hyperalgesia in the rat , 1981, Brain Research.

[33]  C. Pinsky,et al.  Morphine derivatives with diminished opiate receptor potency show enhanced central excitatory activity , 1979, Brain Research.

[34]  Fred E. D'Amour,et al.  A METHOD FOR DETERMINING LOSS OF PAIN SENSATION , 1941 .

[35]  D. Carr,et al.  Morphine-3-glucuronide: silent regulator of morphine actions. , 1994, Life sciences.

[36]  S. Bartlett,et al.  The excitatory effects of morphine-3-glucuronide are attenuated by LY274614, a competitive NMDA receptor antagonist, and by midazolam, an agonist at the benzodiazepine site on the GABAA receptor complex. , 1994, Life sciences.

[37]  A. Somogyi,et al.  Mu receptor binding of some commonly used opioids and their metabolites. , 1991, Life sciences.

[38]  L. Hammar,et al.  Morphine metabolism in mouse brain. , 1986, NIDA research monograph.