IL-22 suppresses IFN-γ-mediated lung inflammation in asthmatic patients.
暂无分享,去创建一个
S. Durham | K. Chung | A. Cavani | C. Schmidt‐Weber | K. Eyerich | L. Cifuentes | Pascal Venn | P. Bhavsar | S. Eyerich | M. Quaranta | D. Pennino | S. Avitabile | V. Marzaioli | R. Effner
[1] F. Nestle,et al. The multitasking organ: recent insights into skin immune function. , 2011, Immunity.
[2] J. Ring,et al. Mutual antagonism of T cells causing psoriasis and atopic eczema. , 2011, The New England journal of medicine.
[3] J. Ring,et al. IL‐22 and TNF‐α represent a key cytokine combination for epidermal integrity during infection with Candida albicans , 2011, European journal of immunology.
[4] M. Willart,et al. Dual Role of IL-22 in allergic airway inflammation and its cross-talk with IL-17A. , 2011, American journal of respiratory and critical care medicine.
[5] P. Pontisso,et al. Overexpression of SERPIN B3 promotes epithelial proliferation and lung fibrosis in mice , 2011, Laboratory Investigation.
[6] Timothy J. Break,et al. IL-22 Production Is Regulated by IL-23 During Listeria monocytogenes Infection but Is Not Required for Bacterial Clearance or Tissue Protection , 2011, PloS one.
[7] W. Born,et al. γδ T cells protect against lung fibrosis via IL-22 , 2010, The Journal of experimental medicine.
[8] A. Cavani,et al. IL-17 and IL-22: siblings, not twins. , 2010, Trends in immunology.
[9] N. Grabe,et al. Impaired skin regeneration and remodeling after cutaneous injury and chemically induced hyperplasia in taps-transgenic mice. , 2010, The Journal of investigative dermatology.
[10] D. Artis,et al. Pathological versus protective functions of IL-22 in airway inflammation are regulated by IL-17A , 2010, The Journal of experimental medicine.
[11] D. Topham,et al. Interleukin-22 (IL-22) Production by Pulmonary Natural Killer Cells and the Potential Role of IL-22 during Primary Influenza Virus Infection , 2010, Journal of Virology.
[12] S. Garcovich,et al. IL-17 Amplifies Human Contact Hypersensitivity by Licensing Hapten Nonspecific Th1 Cells to Kill Autologous Keratinocytes , 2010, The Journal of Immunology.
[13] P. Howarth,et al. Chemokine Receptor 4 Plays a Key Role in T Cell Recruitment into the Airways of Asthmatic Patients , 2010, The Journal of Immunology.
[14] H. Spits,et al. Human NKp44+IL-22+ cells and LTi-like cells constitute a stable RORC+ lineage distinct from conventional natural killer cells , 2010, The Journal of experimental medicine.
[15] H. Spits,et al. Human NKp 44 + IL-22 + cells and LTi-like cells constitute a stable RORC + lineage distinct from conventional natural killer cells , 2010 .
[16] S. Durham,et al. Th22 cells represent a distinct human T cell subset involved in epidermal immunity and remodeling. , 2009, The Journal of clinical investigation.
[17] D. Jarrossay,et al. Production of interleukin 22 but not interleukin 17 by a subset of human skin-homing memory T cells , 2009, Nature Immunology.
[18] H. Spits,et al. Identification of a human helper T cell population that has abundant production of interleukin 22 and is distinct from TH-17, TH1 and TH2 cells , 2009, Nature Immunology.
[19] M. Colonna. Interleukin-22-producing natural killer cells and lymphoid tissue inducer-like cells in mucosal immunity. , 2009, Immunity.
[20] U. Schlötzer-Schrehardt,et al. Mice Lacking the Extracellular Matrix Protein WARP Develop Normally but Have Compromised Peripheral Nerve Structure and Function* , 2009, Journal of Biological Chemistry.
[21] J. Darnell,et al. Serpin B3/B4, activated by STAT3, promote survival of squamous carcinoma cells. , 2009, Biochemical and biophysical research communications.
[22] R. Djukanović,et al. Virus infection, asthma, and chronic obstructive pulmonary disease. , 2008, The New England journal of medicine.
[23] Jeffrey D. Morton,et al. Persistent activation of an innate immune response translates respiratory viral infection into chronic lung disease , 2008, Nature Medicine.
[24] M. Fei,et al. IL-22 mediates mucosal host defense against Gram-negative bacterial pneumonia , 2008, Nature Medicine.
[25] W. Jeong,et al. Innate immunity and alcoholic liver fibrosis , 2008, Journal of gastroenterology and hepatology.
[26] P. Barnes,et al. The Role of IκB Kinase 2, but Not Activation of NF-κB, in the Release of CXCR3 Ligands from IFN-γ-Stimulated Human Bronchial Epithelial Cells , 2007, The Journal of Immunology.
[27] A. Murphy,et al. Interleukin-22 but not interleukin-17 provides protection to hepatocytes during acute liver inflammation. , 2007, Immunity.
[28] S. Johnston,et al. IFN-γ–induced protein 10 is a novel biomarker of rhinovirus-induced asthma exacerbations , 2007, Journal of Allergy and Clinical Immunology.
[29] P. Barnes,et al. The role of IkappaB kinase 2, but not activation of NF-kappaB, in the release of CXCR3 ligands from IFN-gamma-stimulated human bronchial epithelial cells. , 2007, Journal of immunology.
[30] L. Fouser,et al. Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides , 2006, The Journal of experimental medicine.
[31] S. Wenzel. Asthma: defining of the persistent adult phenotypes , 2006, The Lancet.
[32] K. Asadullah,et al. IL‐22 regulates the expression of genes responsible for antimicrobial defense, cellular differentiation, and mobility in keratinocytes: a potential role in psoriasis , 2006, European journal of immunology.
[33] P. Howarth,et al. Understanding the pathophysiology of severe asthma to generate new therapeutic opportunities. , 2006, The Journal of allergy and clinical immunology.
[34] J. Shelhamer,et al. Influence of IFN-γ on gene expression in normal human bronchial epithelial cells: modulation of IFN-γ effects by dexamethasone , 2005 .
[35] M. Kacena,et al. Loss of the transcription factor p45 NF-E2 results in a developmental arrest of megakaryocyte differentiation and the onset of a high bone mass phenotype. , 2005, Bone.
[36] J. Shelhamer,et al. Influence of IFN-gamma on gene expression in normal human bronchial epithelial cells: modulation of IFN-gamma effects by dexamethasone. , 2005, Physiological genomics.
[37] K. Asadullah,et al. IL-22 increases the innate immunity of tissues. , 2004, Immunity.
[38] E. Gelfand,et al. Effector CD8+ T cells mediate inflammation and airway hyper-responsiveness , 2004, Nature Medicine.
[39] A. Kay,et al. The role of T lymphocytes in the pathogenesis of asthma. , 2003, The Journal of allergy and clinical immunology.
[40] Miller,et al. of Tissues , 2003 .
[41] J. Bateman,et al. WARP is a new member of the von Willebrand factor A‐domain superfamily of extracellular matrix proteins , 2002, FEBS letters.
[42] C. Albanesi,et al. Disparate Cytotoxic Activity of Nickel-Specific CD8+ and CD4+ T Cell Subsets Against Keratinocytes1 , 2000, The Journal of Immunology.
[43] S. Spector,et al. Proceedings of the ATS workshop on refractory asthma: current understanding, recommendations, and unanswered questions. American Thoracic Society. , 2000, American journal of respiratory and critical care medicine.
[44] E. Gelfand,et al. CD8 T cells are essential in the development of respiratory syncytial virus-induced lung eosinophilia and airway hyperresponsiveness. , 1999, Journal of immunology.
[45] T. Miyauchi,et al. [MASS phenotype]. , 1996, Ryoikibetsu shokogun shirizu.
[46] R. Herberman. Natural killer cells. , 1989, Progress in clinical and biological research.