Dimethyl Fumarate Mitigates Tauopathy in Aβ-Induced Neuroblastoma SH-SY5Y Cells
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[1] Yu Tian Wang,et al. MKP-1 reduces Aβ generation and alleviates cognitive impairments in Alzheimer's disease models. , 2019, Signal Transduction and Targeted Therapy.
[2] P. Vangheluwe,et al. Dimethyl fumarate alters intracellular Ca2+ handling in immune cells by redox-mediated pleiotropic effects. , 2019, Free radical biology & medicine.
[3] Hassan K. Obied,et al. Olive Biophenols Reduces Alzheimer’s Pathology in SH-SY5Y Cells and APPswe Mice , 2018, International journal of molecular sciences.
[4] Karolina Pierzynowska,et al. Age-dependent effects of dimethyl fumarate on cognitive and neuropathological features in the streptozotocin-induced rat model of Alzheimer’s disease , 2018, Brain Research.
[5] S. Kügler,et al. Pharmacological targeting of GSK-3 and NRF2 provides neuroprotection in a preclinical model of tauopathy , 2017, Redox biology.
[6] P. Palumaa,et al. In situ fibrillizing amyloid-beta 1-42 induces neurite degeneration and apoptosis of differentiated SH-SY5Y cells , 2017, PloS one.
[7] S. Cuzzocrea,et al. Multiple mechanisms of dimethyl fumarate in amyloid β‐induced neurotoxicity in human neuronal cells , 2017, Journal of cellular and molecular medicine.
[8] Pedro Rosa-Neto,et al. Synergistic interaction between amyloid and tau predicts the progression to dementia , 2017, Alzheimer's & Dementia.
[9] A. Oliveira‐Brett,et al. Alzheimer's disease amyloid beta peptides in vitro electrochemical oxidation. , 2017, Bioelectrochemistry.
[10] T. Wisniewski,et al. Alzheimer’s disease: experimental models and reality , 2017, Acta Neuropathologica.
[11] H. Hayashi,et al. Involvement of GSK-3β Phosphorylation Through PI3-K/Akt in Cerebral Ischemia-Induced Neurogenesis in Rats , 2016, Molecular Neurobiology.
[12] N. Inestrosa,et al. How the Wnt signaling pathway protects from neurodegeneration: the mitochondrial scenario , 2015, Front. Cell. Neurosci..
[13] L. Shriver,et al. Dimethyl fumarate modulates antioxidant and lipid metabolism in oligodendrocytes , 2015, Redox biology.
[14] A. Hake,et al. Alzheimer's disease clinical trials: past failures and future opportunities , 2015 .
[15] D. Arnold,et al. Time course of clinical and neuroradiological effects of delayed-release dimethyl fumarate in multiple sclerosis , 2015, European journal of neurology.
[16] G. Bloom. Amyloid-β and tau: the trigger and bullet in Alzheimer disease pathogenesis. , 2014, JAMA neurology.
[17] A. Mietelska-Porowska,et al. Tau Protein Modifications and Interactions: Their Role in Function and Dysfunction , 2014, International journal of molecular sciences.
[18] L. Serpell,et al. The relationship between amyloid structure and cytotoxicity , 2014, Prion.
[19] F. Kees. Dimethyl fumarate: a Janus-faced substance? , 2013, Expert opinion on pharmacotherapy.
[20] N. Inestrosa,et al. Canonical Wnt signaling protects hippocampal neurons from Aβ oligomers: role of non-canonical Wnt-5a/Ca2+ in mitochondrial dynamics , 2013, Front. Cell. Neurosci..
[21] I. Parker,et al. Cytotoxicity of Intracellular Aβ42 Amyloid Oligomers Involves Ca2+ Release from the Endoplasmic Reticulum by Stimulated Production of Inositol Trisphosphate , 2013, The Journal of Neuroscience.
[22] Qiang Wu,et al. Effect of c-Jun N-terminal kinase (JNK)/p38 mitogen-activated protein kinase (p38 MAPK) in morphine-induced tau protein hyperphosphorylation , 2013, Behavioural Brain Research.
[23] C. Oliveira,et al. Amyloid-beta peptide 1-42 causes microtubule deregulation through N-methyl-D-aspartate receptors in mature hippocampal cultures. , 2012, Current Alzheimer research.
[24] Yuhong Yang,et al. Dimethyl Fumarate Inhibits Dendritic Cell Maturation via Nuclear Factor κB (NF-κB) and Extracellular Signal-regulated Kinase 1 and 2 (ERK1/2) and Mitogen Stress-activated Kinase 1 (MSK1) Signaling* , 2012, The Journal of Biological Chemistry.
[25] J. Garrido,et al. Molecular Neuroscience Review Article Modulation of Gsk-3 as a Therapeutic Strategy on Tau Pathologies Role and Regulation of Gsk-3 Activity , 2022 .
[26] Shaomin Li,et al. Soluble Aβ Oligomers Inhibit Long-Term Potentiation through a Mechanism Involving Excessive Activation of Extrasynaptic NR2B-Containing NMDA Receptors , 2011, The Journal of Neuroscience.
[27] A. Miranker,et al. Protein-induced photophysical changes to the amyloid indicator dye thioflavin T , 2010, Proceedings of the National Academy of Sciences.
[28] E. F. da Cruz e Silva,et al. Aβ promotes Alzheimer’s disease‐like cytoskeleton abnormalities with consequences to APP processing in neurons , 2010, Journal of neurochemistry.
[29] R. Youle,et al. The role of mitochondria in apoptosis*. , 2009, Annual review of genetics.
[30] Jianqing Ding,et al. Protective effects of erythropoietin on tau phosphorylation induced by β‐amyloid , 2008, Journal of neuroscience research.
[31] L. Serpell,et al. Amyloid fibrils , 2008, Prion.
[32] Ying Jin,et al. Effects of sodium ferulate on amyloid-beta-induced MKK3/MKK6-p38 MAPK-Hsp27 signal pathway and apoptosis in rat hippocampus , 2006, Acta Pharmacologica Sinica.
[33] J. Lucas,et al. Full Reversal of Alzheimer's Disease-Like Phenotype in a Mouse Model with Conditional Overexpression of Glycogen Synthase Kinase-3 , 2006, The Journal of Neuroscience.
[34] Thuy-vi Nguyen,et al. β-Amyloid-Induced Neuronal Apoptosis Involves c-Jun N-Terminal Kinase-Dependent Downregulation of Bcl-w , 2005, The Journal of Neuroscience.
[35] Y. Surh,et al. Bcl-2 protects against Abeta(25-35)-induced oxidative PC12 cell death by potentiation of antioxidant capacity. , 2004, Biochemical and biophysical research communications.
[36] L. K. Baker,et al. Oligomeric and Fibrillar Species of Amyloid-β Peptides Differentially Affect Neuronal Viability* , 2002, The Journal of Biological Chemistry.
[37] H. Joshi,et al. Differential utilization of beta-tubulin isotypes in differentiating neurites , 1989, The Journal of cell biology.
[38] R. Cole,et al. Effect of ATP on the kinetics of microtubule assembly. , 1982, The Journal of biological chemistry.
[39] Brendon M. Baker,et al. Therapeutic Targeting of TAZ and YAP by Dimethyl Fumarate in Systemic Sclerosis Fibrosis. , 2018, The Journal of investigative dermatology.
[40] Fei Liu,et al. Tau and neurodegenerative disease: the story so far , 2016, Nature Reviews Neurology.
[41] Justin C. Y. Wu,et al. Isorhynchophylline treatment improves the amyloid-β-induced cognitive impairment in rats via inhibition of neuronal apoptosis and tau protein hyperphosphorylation. , 2014, Journal of Alzheimer's disease : JAD.
[42] Qiang Du,et al. Cross-Regulation Between Wnt and NF-κB Signaling Pathways. , 2010, Forum on immunopathological diseases and therapeutics.
[43] Gregory J Brewer,et al. Amyloid-β as a modulator of synaptic plasticity. , 2010, Journal of Alzheimer's disease : JAD.
[44] John M. Walker,et al. The Protein Protocols Handbook , 1996, Humana Press.
[45] J M Walker,et al. The bicinchoninic acid (BCA) assay for protein quantitation. , 1994, Methods in molecular biology.