Inhibitors of permeability transition interfere with the disruption of the mitochondrial transmembrane potential during apoptosis
暂无分享,去创建一个
G. Kroemer | M. Castedo | P. Marchetti | N. Zamzami | S. Susin | T. Hirsch | B. Masse | Tamara Hirsch
[1] G. Kroemer,et al. Mitochondrial perturbations define lymphocytes undergoing apoptotic depletion in vivo , 1995, European journal of immunology.
[2] Guido Kroemer,et al. The biochemistry of programmed cell death , 1995, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[3] C. Franceschi,et al. Protective effect of N-acetylcysteine in tumor necrosis factor-alpha-induced apoptosis in U937 cells: the role of mitochondria. , 1995, Experimental cell research.
[4] G. Kroemer,et al. Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death , 1995, The Journal of experimental medicine.
[5] M. Zoratti,et al. The mitochondrial permeability transition. , 1995, Biochimica et biophysica acta.
[6] P. Petit,et al. Alterations in mitochondrial structure and function are early events of dexamethasone-induced thymocyte apoptosis , 1995, The Journal of cell biology.
[7] M. Crompton,et al. Evidence for the involvement of a membrane-associated cyclosporin-A-binding protein in the Ca(2+)-activated inner membrane pore of heart mitochondria. , 1995, European journal of biochemistry.
[8] G. Kroemer,et al. Reduction in mitochondrial potential constitutes an early irreversible step of programmed lymphocyte death in vivo , 1995, The Journal of experimental medicine.
[9] J. Farber,et al. Ca2+ depletion prevents anoxic death of hepatocytes by inhibiting mitochondrial permeability transition. , 1995, The American journal of physiology.
[10] J. Hoek,et al. Protoporphyrin IX, an endogenous ligand of the peripheral benzodiazepine receptor, potentiates induction of the mitochondrial permeability transition and the killing of cultured hepatocytes by rotenone. , 1994, The Journal of biological chemistry.
[11] L. Schwartz,et al. Identification of Genes Induced during Apoptosis in T Lymphocytes , 1994, Immunological reviews.
[12] A. J. Gandolfi,et al. Novel mechanisms in chemically induced hepatotoxicity 1 , 1994, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[13] D. Newmeyer,et al. Cell-free apoptosis in Xenopus egg extracts: Inhibition by Bcl-2 and requirement for an organelle fraction enriched in mitochondria , 1994, Cell.
[14] P. Bernardi,et al. Recent progress on regulation of the mitochondrial permeability transition pore; a cyclosporin-sensitive pore in the inner mitochondrial membrane , 1994, Journal of bioenergetics and biomembranes.
[15] K. Schulze-Osthoff,et al. Cell nucleus and DNA fragmentation are not required for apoptosis , 1994, The Journal of cell biology.
[16] P. Bernardi,et al. Regulation of the permeability transition pore, a voltage-dependent mitochondrial channel inhibited by cyclosporin A. , 1994, Biochimica et biophysica acta.
[17] Y. Shinohara,et al. Importance of loops of mitochondrial ADP/ATP carrier for its transport activity deduced from reactivities of its cysteine residues with the sulfhydryl reagent eosin-5-maleimide. , 1994, Biochemistry.
[18] M. Raff,et al. Programmed cell death and Bcl‐2 protection in the absence of a nucleus. , 1994, The EMBO journal.
[19] B. Hoffmann,et al. The reconstituted ADP/ATP carrier activity has an absolute requirement for cardiolipin as shown in cysteine mutants. , 1994, The Journal of biological chemistry.
[20] T. Deckwerth,et al. Temporal analysis of events associated with programmed cell death (apoptosis) of sympathetic neurons deprived of nerve growth factor , 1993, The Journal of cell biology.
[21] J. Farber,et al. Cyclosporin and carnitine prevent the anoxic death of cultured hepatocytes by inhibiting the mitochondrial permeability transition. , 1993, The Journal of biological chemistry.
[22] M. Schreier,et al. Molecular Mechanisms of Immunosuppression by Cyclosporins , 1993, Annals of the New York Academy of Sciences.
[23] G. Kroemer,et al. Glucocorticoid-mediated control of the activation and clonal deletion of peripheral T cells in vivo , 1993, The Journal of experimental medicine.
[24] J. Klein,et al. Flow cytometric analysis of altered mononuclear cell transmembrane potential induced by cyclosporin. , 1993, Cytometry.
[25] S. Schreiber. Immunophilin-sensitive protein phosphatase action in cell signaling pathways , 1992, Cell.
[26] R. Brasseur,et al. Topography of the membrane-bound ADP/ATP carrier assessed by enzymatic proteolysis. , 1992, Biochemistry.
[27] I Nicoletti,et al. A rapid and simple method for measuring thymocyte apoptosis by propidium iodide staining and flow cytometry. , 1991, Journal of immunological methods.
[28] M. Zoratti,et al. The giant channel of the inner mitochondrial membrane is inhibited by cyclosporin A. , 1991, The Journal of biological chemistry.
[29] Y. Kawabe,et al. Programmed cell death and extrathymic reduction of Vβ8+CD4+ T cells in mice tolerant to Staphylococcus aureus enterotoxin B , 1991, Nature.
[30] M. Crompton,et al. The presence of two classes of high-affinity cyclosporin A binding sites in mitochondria. Evidence that the minor component is involved in the opening of an inner-membrane Ca(2+)-dependent pore. , 1990, European journal of biochemistry.
[31] A. Halestrap,et al. Inhibition of Ca2(+)-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adenine nucleotide translocase. , 1990, The Biochemical journal.
[32] A. Halestrap,et al. Partial inhibition by cyclosporin A of the swelling of liver mitochondria in vivo and in vitro induced by sub-micromolar [Ca2+], but not by butyrate. Evidence for two distinct swelling mechanisms. , 1990, The Biochemical journal.
[33] Gregor Rothe,et al. Flow Cytometric Analysis of Respiratory Burst Activity in Phagocytes With Hydroethidine and 2′,7′‐Dichlorofluorescin , 1990, Journal of leukocyte biology.
[34] M. Dempsey,et al. Cyclosporin A is a potent inhibitor of the inner membrane permeability transition in liver mitochondria. , 1989, The Journal of biological chemistry.
[35] M. Crompton,et al. Inhibition by cyclosporin A of a Ca2+-dependent pore in heart mitochondria activated by inorganic phosphate and oxidative stress. , 1988, The Biochemical journal.
[36] A. Toninello,et al. On the mechanism by which Mg2+ and adenine nucleotides restore membrane potential in rat liver mitochondria deenergized by Ca2+ and phosphate. , 1983, Biochemical and biophysical research communications.
[37] H. Cox,et al. Elucidation of the chemical structure of bongkrekic acid—I : Isolation, purification and properties of bongkrekic acid☆ , 1970 .
[38] G. Schatz,et al. The biogenesis of mitochondria. , 1970, The Biochemical journal.