A single-cell transcriptional gradient in human cutaneous memory T cells restricts Th17/Tc17 identity
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Jeffrey B. Cheng | E. Purdom | A. Marson | Hao Wang | R. Cho | S. Benz | S. Ramos | Hao Wang | Yanhong Shou | J. North | P. Harirchian | Jaehyuk Choi | R. Schmidt | A. Sedgewick | S. Kashem | C. Cook | Mark Taylor | Yale Liu | Esther Kim | A. Hailer | Timothy C. McCalmont | Ralf Schmidt
[1] Jeffrey B. Cheng,et al. Classification of human chronic inflammatory skin disease based on single-cell immune profiling , 2022, Science Immunology.
[2] T. Radstake,et al. ZFP36 family members regulate the pro-inflammatory features of psoriatic dermal fibroblasts. , 2021, The Journal of investigative dermatology.
[3] G. Rabinovich,et al. Galectin-1 fosters an immunosuppressive microenvironment in colorectal cancer by reprogramming CD8+ regulatory T cells , 2021, Proceedings of the National Academy of Sciences.
[4] T. Hirai,et al. 036 IL-23 maintains tissue resident memory Th17 cells in murine and psoriatic skin , 2021, Journal of Investigative Dermatology.
[5] Jihyun Yu,et al. CD82 promotes CD8+ T cell immune responses by mediating T cell polarization and immunological synapse formation , 2021, The Journal of Immunology.
[6] S. Teichmann,et al. Developmental cell programs are co-opted in inflammatory skin disease , 2021, Science.
[7] M. Mildner,et al. Persistence of mature dendritic cells, TH2A, and Tc2 cells characterize clinically resolved atopic dermatitis under IL-4Rα blockade , 2021, Science Immunology.
[8] B. Brüne,et al. Role of Tristetraprolin in the Resolution of Inflammation , 2021, Biology.
[9] S. Arron,et al. Single cell RNA-seq of psoriatic skin identifies pathogenic Tc17 subsets and reveals distinctions between CD8+ T cells in autoimmunity and cancer. , 2020, The Journal of allergy and clinical immunology.
[10] Helena L. Crowell,et al. muscat detects subpopulation-specific state transitions from multi-sample multi-condition single-cell transcriptomics data , 2020, Nature Communications.
[11] J. C. Love,et al. Second-Strand Synthesis-Based Massively Parallel scRNA-Seq Reveals Cellular States and Molecular Features of Human Inflammatory Skin Pathologies , 2020, Immunity.
[12] Jun S. Song,et al. Single-Cell Profiling Reveals Divergent, Globally Patterned Immune Responses in Murine Skin Inflammation , 2020, iScience.
[13] M. Kopf,et al. The thioredoxin‐1 inhibitor Txnip restrains effector T‐cell and germinal center B‐cell expansion , 2020, European journal of immunology.
[14] Kotaro Suzuki,et al. RNA-Binding Protein ZFP36L2 Downregulates Helios Expression and Suppresses the Function of Regulatory T Cells , 2020, Frontiers in Immunology.
[15] Hua Zhou,et al. iCellR: Combined Coverage Correction and Principal Component Alignment for Batch Alignment in Single-Cell Sequencing Analysis , 2020, bioRxiv.
[16] R. Flavell,et al. mRNA destabilization by BTG1 and BTG2 maintains T cell quiescence , 2020, Science.
[17] Peng Qiu,et al. Embracing the dropouts in single-cell RNA-seq analysis , 2020, Nature Communications.
[18] S. Warming,et al. CD96 functions as a co‐stimulatory receptor to enhance CD8+ T cell activation and effector responses , 2020, European journal of immunology.
[19] Kieran R. Campbell,et al. Dissociation of solid tumor tissues with cold active protease for single-cell RNA-seq minimizes conserved collagenase-associated stress responses , 2019, Genome Biology.
[20] Kouji Matsushima,et al. CXCR6 regulates localization of tissue-resident memory CD8 T cells to the airways , 2019, The Journal of experimental medicine.
[21] Kamil Slowikowski,et al. Fast, sensitive, and accurate integration of single cell data with Harmony , 2019, Nature Methods.
[22] C. Larminie,et al. Single-cell transcriptomics identifies an effectorness gradient shaping the response of CD4+ T cells to cytokines , 2019, Nature Communications.
[23] Nicholas A. Rossi,et al. Inference of CRISPR Edits from Sanger Trace Data , 2019, bioRxiv.
[24] Monika S. Kowalczyk,et al. Effector TH17 Cells Give Rise to Long-Lived TRM Cells that Are Essential for an Immediate Response against Bacterial Infection , 2019, Cell.
[25] M. Willemsen,et al. Skin‐resident memory T cells as a potential new therapeutic target in vitiligo and melanoma , 2019, Pigment cell & melanoma research.
[26] T. Aune,et al. Biological Effects of IL-26 on T Cell-Mediated Skin Inflammation, Including Psoriasis. , 2019, The Journal of investigative dermatology.
[27] Yvan Saeys,et al. A comparison of single-cell trajectory inference methods , 2019, Nature Biotechnology.
[28] U. V. von Andrian,et al. CCL22 controls immunity by promoting regulatory T cell communication with dendritic cells in lymph nodes , 2019, The Journal of experimental medicine.
[29] Andrew J. Hill,et al. The single cell transcriptional landscape of mammalian organogenesis , 2019, Nature.
[30] A. Butte,et al. Reference-based analysis of lung single-cell sequencing reveals a transitional profibrotic macrophage , 2018, Nature Immunology.
[31] Paul J. Hoffman,et al. Comprehensive Integration of Single-Cell Data , 2018, Cell.
[32] Vincent A. Traag,et al. From Louvain to Leiden: guaranteeing well-connected communities , 2018, Scientific Reports.
[33] Charles J. Vaske,et al. Transcriptional Programming of Normal and Inflamed Human Epidermis at Single-Cell Resolution , 2018, Cell reports.
[34] S. Yao,et al. The molecular basis of JAK/STAT inhibition by SOCS1 , 2018, Nature Communications.
[35] T. Natsume,et al. ZFP36L2 is a cell cycle-regulated CCCH protein necessary for DNA lesion-induced S-phase arrest , 2018, Biology Open.
[36] Christopher Y. Park,et al. ZFP36 RNA-binding proteins restrain T cell activation and anti-viral immunity , 2018, bioRxiv.
[37] Lior Pachter,et al. Gene-level differential analysis at transcript-level resolution , 2017, Genome Biology.
[38] Hannah A. Pliner,et al. Reversed graph embedding resolves complex single-cell trajectories , 2017, Nature Methods.
[39] P. Blackshear,et al. Tristetraprolin expression by keratinocytes controls local and systemic inflammation. , 2017, JCI insight.
[40] S. Endres,et al. CCL22 impedes T cell activation capacities of dendritic cells by reducing membrane expression of MHC molecules and CD80 , 2017, The Journal of Immunology.
[41] Gennady Korotkevich,et al. Fast gene set enrichment analysis , 2016, bioRxiv.
[42] Lior Pachter,et al. Differential analysis of RNA-seq incorporating quantification uncertainty , 2016, Nature Methods.
[43] Manuel D. Díaz-Muñoz,et al. RNA-binding proteins ZFP36L1 and ZFP36L2 promote cell quiescence , 2016, Science.
[44] Lior Pachter,et al. Near-optimal probabilistic RNA-seq quantification , 2016, Nature Biotechnology.
[45] Q. Lu,et al. The Inflammatory Response in Psoriasis: a Comprehensive Review , 2016, Clinical Reviews in Allergy & Immunology.
[46] R. Clark. Resident memory T cells in human health and disease , 2015, Science Translational Medicine.
[47] M. Ståhle,et al. Epidermal Th22 and Tc17 Cells Form a Localized Disease Memory in Clinically Healed Psoriasis , 2014, The Journal of Immunology.
[48] Cole Trapnell,et al. Pseudo-temporal ordering of individual cells reveals dynamics and regulators of cell fate decisions , 2014, Nature Biotechnology.
[49] M. Rottenberg,et al. SOCS3, a Major Regulator of Infection and Inflammation , 2014, Front. Immunol..
[50] B. Lee,et al. Tristetraprolin down‐regulates IL‐17 through mRNA destabilization , 2012, FEBS letters.
[51] Pilar Martín,et al. CD69 Association with Jak3/Stat5 Proteins Regulates Th17 Cell Differentiation , 2010, Molecular and Cellular Biology.
[52] N. Restifo,et al. Suppressors of cytokine signaling (SOCS) in T cell differentiation, maturation, and function. , 2009, Trends in immunology.
[53] Davis J. McCarthy,et al. edgeR: a Bioconductor package for differential expression analysis of digital gene expression data , 2009, Bioinform..
[54] P. Blackshear,et al. Tristetraprolin Mediates Interferon-γ mRNA Decay* , 2009, Journal of Biological Chemistry.
[55] Lisa C. Zaba,et al. Psoriasis vulgaris lesions contain discrete populations of Th1 and Th17 T cells. , 2008, The Journal of investigative dermatology.
[56] J. Mesirov,et al. From the Cover: Gene set enrichment analysis: A knowledge-based approach for interpreting genome-wide expression profiles , 2005 .
[57] J. Connolly,et al. The Role of mRNA Turnover in the Regulation of Tristetraprolin Expression: Evidence for an Extracellular Signal-Regulated Kinase-Specific, AU-Rich Element-Dependent, Autoregulatory Pathway1 , 2004, The Journal of Immunology.
[58] N. C. Silver,et al. A Monte Carlo Evaluation of Tests for Comparing Dependent Correlations , 2003, The Journal of general psychology.
[59] D. Witherden,et al. CD81 and CD28 Costimulate T Cells Through Distinct Pathways1 , 2000, The Journal of Immunology.
[60] A. Shaw,et al. Coordinate Regulation of T Cell Activation by CD2 and CD281 , 2000, The Journal of Immunology.
[61] P. Blackshear,et al. Evidence that tristetraprolin is a physiological regulator of granulocyte-macrophage colony-stimulating factor messenger RNA deadenylation and stability. , 2000, Blood.
[62] H. Hamada,et al. Overexpression of CD82 on human T cells enhances LFA‐1 / ICAM‐1‐mediated cell‐cell adhesion: functional association between CD82 and LFA‐1 in T cell activation , 1999, European journal of immunology.