Calprotectin in myeloid and epithelial cells of laminae from horses with black walnut extract-induced laminitis.
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[1] G. Nuovo,et al. In vivo and in vitro evidence of the involvement of CXCL1, a keratinocyte-derived chemokine, in equine laminitis. , 2009, Journal of veterinary internal medicine.
[2] M. Polyak,et al. Detection of calprotectin and its correlation to the accumulation of neutrophils within equine large colon during ischaemia and reperfusion. , 2008, Equine veterinary journal.
[3] M. Sakaguchi,et al. S100A8/A9, a key mediator for positive feedback growth stimulation of normal human keratinocytes , 2008, Journal of cellular biochemistry.
[4] W. Chazin,et al. S100A8/9 induces cell death via a novel, RAGE-independent pathway that involves selective release of Smac/DIABLO and Omi/HtrA2. , 2008, Biochimica et biophysica acta.
[5] W. Nacken,et al. Myeloid-related proteins S100A8/S100A9 regulate joint inflammation and cartilage destruction during antigen-induced arthritis , 2007, Annals of the rheumatic diseases.
[6] Rustin M. Moore,et al. Local and remote lesions in horses subjected to small colon distension and decompression. , 2008, Canadian journal of veterinary research = Revue canadienne de recherche veterinaire.
[7] S. Black,et al. Early laminar events involving endothelial activation in horses with black walnut- induced laminitis. , 2007, American journal of veterinary research.
[8] W. Nacken,et al. Mrp8 and Mrp14 are endogenous activators of Toll-like receptor 4, promoting lethal, endotoxin-induced shock , 2007, Nature Medicine.
[9] A. V. van Eps,et al. Equine laminitis: ultrastructural lesions detected 24-30 hours after induction with oligofructose. , 2007, Equine veterinary journal.
[10] M. Hersberger,et al. Myeloid-related protein 8/14 complex is released by monocytes and granulocytes at the site of coronary occlusion: a novel, early, and sensitive marker of acute coronary syndromes. , 2007, European heart journal.
[11] K. Schulze-Osthoff,et al. MRP 8 / MRP 14 impairs endothelial integrity and induces a caspase-dependent and-independent cell death program , 2007 .
[12] M. Bianchi. DAMPs, PAMPs and alarmins: all we need to know about danger , 2007, Journal of leukocyte biology.
[13] A. V. van Eps,et al. Lamellar pro-inflammatory cytokine expression patterns in laminitis at the developmental stage and at the onset of lameness: innate vs. adaptive immune response. , 2007, Equine veterinary journal.
[14] C. Heizmann,et al. Pathologies involving the S100 proteins and RAGE. , 2007, Sub-cellular biochemistry.
[15] KA Brown,et al. Neutrophils in development of multiple organ failure in sepsis , 2006, The Lancet.
[16] D. Foell,et al. Elevated serum levels of calcium‐binding S100 proteins A8 and A9 reflect disease activity and abnormal differentiation of keratinocytes in psoriasis , 2006, The British journal of dermatology.
[17] S. Black,et al. Leukocyte emigration in the early stages of laminitis. , 2006, Veterinary immunology and immunopathology.
[18] C. Pollitt,et al. Equine laminitis: loss of hemidesmosomes in hoof secondary epidermal lamellae correlates to dose in an oligofructose induction model: an ultrastructural study. , 2010, Equine veterinary journal.
[19] Rustin M. Moore,et al. Assessment of apoptosis in epidermal lamellar cells in clinically normal horses and those with laminitis. , 2004, American journal of veterinary research.
[20] S. Yui,et al. Calprotectin (S100A8/S100A9), an inflammatory protein complex from neutrophils with a broad apoptosis-inducing activity. , 2003, Biological & pharmaceutical bulletin.
[21] Ann-Marie Broome,et al. S100 Protein Subcellular Localization During Epidermal Differentiation and Psoriasis , 2003, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.
[22] P. Rouleau,et al. Proinflammatory Activities of S100: Proteins S100A8, S100A9, and S100A8/A9 Induce Neutrophil Chemotaxis and Adhesion 1 , 2003, The Journal of Immunology.
[23] P. Rouleau,et al. Proinflammatory Activities of S 100 : Proteins S 100 A 8 , S 100 A 9 , and S 100 A 8 / A 9 Induce Neutrophil Chemotaxis and Adhesion 1 , 2003 .
[24] D. Goble,et al. Laminar microvascular flow, measured by means of laser Doppler flowmetry, during the prodromal stages of black walnut-induced laminitis in horses. , 2000, American journal of veterinary research.
[25] David A. Hume,et al. S100A8: emerging functions and regulation , 1999, Journal of leukocyte biology.
[26] S. Eades,et al. Digital Starling forces and hemodynamics during early laminitis induced by an aqueous extract of black walnut (Juglans nigra) in horses. , 1995, American journal of veterinary research.
[27] R. Moritz,et al. Identification of a chemotactic domain of the pro-inflammatory S100 protein CP-10. , 1993, Journal of immunology.
[28] S. Fleming,et al. Morphological evidence for calcium‐dependent association of calgranulin with the epidermal cytoskeleton in inflammatory dermatoses , 1991, The British journal of dermatology.
[29] S. Fleming,et al. Calgranulin expression in inflammatory dermatoses , 1989, The Journal of pathology.