Signalling by senescent melanocytes hyperactivates hair growth

[1]  A. R. Cameron,et al.  Muscle injury induces a transient senescence‐like state that is required for myofiber growth during muscle regeneration , 2022, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[2]  L. Lau,et al.  Senescent hepatic stellate cells promote liver regeneration through IL-6 and ligands of CXCR2 , 2022, JCI insight.

[3]  Zhengquan Yu,et al.  Hedgehog signaling reprograms hair follicle niche fibroblasts to a hyper-activated state. , 2022, Developmental cell.

[4]  Clifford A. Meyer,et al.  Topical therapy for regression and melanoma prevention of congenital giant nevi , 2022, Cell.

[5]  N. LeBrasseur,et al.  Senolytic Drugs: Reducing Senescent Cell Viability to Extend Health Span. , 2020, Annual review of pharmacology and toxicology.

[6]  M. Fujimiya,et al.  Exercise enhances skeletal muscle regeneration by promoting senescence in fibro-adipogenic progenitors , 2020, Nature Communications.

[7]  M. Collado,et al.  Cell senescence contributes to tissue regeneration in zebrafish , 2019, Aging cell.

[8]  Toshiyuki Yamamoto,et al.  Alopecia totalis sparing congenital melanocytic nevus: Renbök phenomenon , 2019, Dermatologica Sinica.

[9]  Soyoung Lee,et al.  The dynamic nature of senescence in cancer , 2019, Nature Cell Biology.

[10]  Maria Kasper,et al.  Single-Cell Transcriptomics of Traced Epidermal and Hair Follicle Stem Cells Reveals Rapid Adaptations during Wound Healing. , 2018, Cell reports.

[11]  Qing Nie,et al.  A multi-scale model for hair follicles reveals heterogeneous domains driving rapid spatiotemporal hair growth patterning , 2017, eLife.

[12]  F. Nestle,et al.  Regulatory T Cells in Skin Facilitate Epithelial Stem Cell Differentiation , 2017, Cell.

[13]  M. Blasco,et al.  Tissue damage and senescence provide critical signals for cellular reprogramming in vivo , 2016, Science.

[14]  Maria Kasper,et al.  Single-Cell Transcriptomics Reveals that Differentiation and Spatial Signatures Shape Epidermal and Hair Follicle Heterogeneity , 2016, Cell systems.

[15]  F. Notta,et al.  Senescent Carcinoma-Associated Fibroblasts Upregulate IL8 to Enhance Prometastatic Phenotypes , 2016, Molecular Cancer Research.

[16]  R. Yi,et al.  Signaling Networks among Stem Cell Precursors, Transit-Amplifying Progenitors, and their Niche in Developing Hair Follicles. , 2016, Cell reports.

[17]  Sameer Gupta,et al.  Genetics of melanocytic nevi , 2015, Pigment cell & melanoma research.

[18]  John J. Cole,et al.  Mitotic Stress Is an Integral Part of the Oncogene-Induced Senescence Program that Promotes Multinucleation and Cell Cycle Arrest , 2015, Cell reports.

[19]  Lei Wang,et al.  Organ-Level Quorum Sensing Directs Regeneration in Hair Stem Cell Populations , 2015, Cell.

[20]  J. Hoeijmakers,et al.  An essential role for senescent cells in optimal wound healing through secretion of PDGF-AA. , 2014, Developmental cell.

[21]  D. Scadden Nice Neighborhood: Emerging Concepts of the Stem Cell Niche , 2014, Cell.

[22]  J. Huse,et al.  Osteopontin-CD44 signaling in the glioma perivascular niche enhances cancer stem cell phenotypes and promotes aggressive tumor growth. , 2014, Cell stem cell.

[23]  A. Rodríguez-Baeza,et al.  Programmed Cell Senescence during Mammalian Embryonic Development , 2013, Cell.

[24]  K. Blyth,et al.  Wnt signaling potentiates nevogenesis , 2013, Proceedings of the National Academy of Sciences.

[25]  J. Campisi Aging, cellular senescence, and cancer. , 2013, Annual review of physiology.

[26]  F. Real,et al.  Postzygotic HRAS and KRAS mutations cause nevus sebaceous and Schimmelpenning syndrome , 2012, Nature Genetics.

[27]  D. Foran,et al.  Identification of Function for CD44 Intracytoplasmic Domain (CD44-ICD) , 2012, The Journal of Biological Chemistry.

[28]  E. Fuchs,et al.  A family business: stem cell progeny join the niche to regulate homeostasis , 2012, Nature Reviews Molecular Cell Biology.

[29]  N. LeBrasseur,et al.  Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders , 2011, Nature.

[30]  V. Horsley,et al.  Adipocyte Lineage Cells Contribute to the Skin Stem Cell Niche to Drive Hair Cycling , 2011, Cell.

[31]  P. Maini,et al.  Self-Organizing and Stochastic Behaviors During the Regeneration of Hair Stem Cells , 2011, Science.

[32]  S. Stewart,et al.  Senescent stromal-derived osteopontin promotes preneoplastic cell growth. , 2009, Cancer research.

[33]  Judith Campisi,et al.  Senescence-Associated Secretory Phenotypes Reveal Cell-Nonautonomous Functions of Oncogenic RAS and the p53 Tumor Suppressor , 2008, PLoS biology.

[34]  T. Shaw,et al.  Molecular mechanisms linking wound inflammation and fibrosis: knockdown of osteopontin leads to rapid repair and reduced scarring , 2008, The Journal of experimental medicine.

[35]  Ruth E. Baker,et al.  Cyclic dermal BMP signalling regulates stem cell activation during hair regeneration , 2008, Nature.

[36]  A. Dongre,et al.  Acquired smooth muscle hamartoma. , 2006, Indian journal of dermatology, venereology and leprology.

[37]  J. Shay,et al.  BRAFE600-associated senescence-like cell cycle arrest of human naevi , 2005, Nature.

[38]  R Paus,et al.  A comprehensive guide for the accurate classification of murine hair follicles in distinct hair cycle stages. , 2001, The Journal of investigative dermatology.

[39]  T. Sun,et al.  Label-retaining cells reside in the bulge area of pilosebaceous unit: Implications for follicular stem cells, hair cycle, and skin carcinogenesis , 1990, Cell.