SMRT corepressor interacts with PLZF and with the PML-retinoic acid receptor α (RARα) and PLZF-RARα oncoproteins associated with acute promyelocytic leukemia
暂无分享,去创建一个
A. Dejean | G. David | M. Privalsky | Chi-wai Wong | Chi Wai Wong | Anne Dejean | Martin L. Privalsky | Gregory David | Suk-Hyun Hong | Suk Hyun Hong
[1] J. Dixon,et al. Eukaryotic proteins expressed in Escherichia coli: an improved thrombin cleavage and purification procedure of fusion proteins with glutathione S-transferase. , 1991, Analytical biochemistry.
[2] P. Martiat,et al. Poor response to all-trans retinoic acid therapy in a t(11;17) PLZF/RAR alpha patient. , 1994, Leukemia.
[3] P. Chambon,et al. PMLRAR homodimers: distinct DNA binding properties and heteromeric interactions with RXR. , 1993, The EMBO journal.
[4] A. Dejean,et al. A retrovirus carrying the promyelocyte‐retinoic acid receptor PML‐RARalpha fusion gene transforms haematopoietic progenitors in vitro and induces acute leukaemias. , 1996, The EMBO journal.
[5] R. Treisman,et al. The POZ domain: a conserved protein-protein interaction motif. , 1994, Genes & development.
[6] Miguel Beato,et al. Steroid hormone receptors: Many Actors in search of a plot , 1995, Cell.
[7] Thorsten Heinzel,et al. Ligand-independent repression by the thyroid hormone receptor mediated by a nuclear receptor co-repressor , 1995, Nature.
[8] K. Umesono,et al. Chromosomal translocation t(15;17) in human acute promyelocytic leukemia fuses RARα with a novel putative transcription factor, PML , 1991, Cell.
[9] P. Pelicci,et al. Acute promyelocytic leukemia: from genetics to treatment. , 1994 .
[10] K. Umesono,et al. SMRT isoforms mediate repression and anti-repression of nuclear receptor heterodimers. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[11] Christine Chomienne,et al. The t(15;17) translocation of acute promyelocytic leukaemia fuses the retinoic acid receptor α gene to a novel transcribed locus , 1990, Nature.
[12] D. Leprince,et al. The LAZ3/BCL6 oncogene encodes a sequence-specific transcriptional inhibitor: a novel function for the BTB/POZ domain as an autonomous repressing domain. , 1995, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.
[13] A. Wolffe. Sinful repression , 1997, nature.
[14] B. O’Malley,et al. Molecular mechanisms of action of steroid/thyroid receptor superfamily members. , 1994, Annual review of biochemistry.
[15] P. Chambon,et al. Structure, localization and transcriptional properties of two classes of retinoic acid receptor alpha fusion proteins in acute promyelocytic leukemia (APL): structural similarities with a new family of oncoproteins. , 1992, The EMBO journal.
[16] A. Dejean,et al. The t(15;17) translocation in acute promyelocytic leukemia. , 1994, Pathologie-biologie.
[17] Christine Chomienne,et al. The PML-RARα fusion mRNA generated by the t(15;17) translocation in acute promyelocytic leukemia encodes a functionally altered RAR , 1991, Cell.
[18] A. Mahfoudi,et al. Multimeric complexes of the PML-retinoic acid receptor alpha fusion protein in acute promyelocytic leukemia cells and interference with retinoid and peroxisome-proliferator signaling pathways. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[19] T. Ley,et al. Altered myeloid development and acute leukemia in transgenic mice expressing PML-RAR alpha under control of cathepsin G regulatory sequences. , 1997, Blood.
[20] D. Sheer,et al. Molecular analysis of acute promyelocytic leukemia breakpoint cluster region on chromosome 17. , 1990, Science.
[21] A. Zelent,et al. PLZF-RAR alpha fusion proteins generated from the variant t(11;17)(q23;q21) translocation in acute promyelocytic leukemia inhibit ligand-dependent transactivation of wild-type retinoic acid receptors. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[22] R. Evans,et al. The RXR heterodimers and orphan receptors , 1995, Cell.
[23] Philippe Kastner,et al. Nonsteroid nuclear receptors: What Are genetic studies telling us about their role in real life? , 1995, Cell.
[24] D. Moore,et al. Two receptor interacting domains in the nuclear hormone receptor corepressor RIP13/N-CoR. , 1996, Molecular endocrinology.
[25] J. Licht,et al. Reduced and altered DNA-binding and transcriptional properties of the PLZF-retinoic acid receptor-alpha chimera generated in t(11;17)-associated acute promyelocytic leukemia. , 1996, Oncogene.
[26] Myles Brown,et al. Polarity-specific activities of retinoic acid receptors determined by a co-repressor , 1995, Nature.
[27] M. Pazin,et al. What's Up and Down with Histone Deacetylation and Transcription? , 1997, Cell.
[28] M. Privalsky,et al. Identification of TRACs (T3 receptor-associating cofactors), a family of cofactors that associate with, and modulate the activity of, nuclear hormone receptors. , 1996, Molecular endocrinology.
[29] L. Staudt,et al. Transcriptional repression by the proto-oncogene BCL-6. , 1996, Oncogene.
[30] K. Umesono,et al. The nuclear receptor superfamily: The second decade , 1995, Cell.
[31] J. Licht,et al. Amino-terminal protein-protein interaction motif (POZ-domain) is responsible for activities of the promyelocytic leukemia zinc finger-retinoic acid receptor-alpha fusion protein. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[32] A. Zelent,et al. Fusion between a novel Krüppel‐like zinc finger gene and the retinoic acid receptor‐alpha locus due to a variant t(11;17) translocation associated with acute promyelocytic leukaemia. , 1993, The EMBO journal.
[33] I. Weissman,et al. A PMLRARα transgene initiates murine acute promyelocytic leukemia , 1997 .
[34] F. Jeanmougin,et al. A possible involvement of TIF1 alpha and TIF1 beta in the epigenetic control of transcription by nuclear receptors. , 1996, The EMBO journal.
[35] C. Glass,et al. A nuclear hormone receptor corepressor mediates transcriptional silencing by receptors with distinct repression domains , 1996, Molecular and cellular biology.
[36] J. Licht,et al. Expression of the zinc-finger gene PLZF at rhombomere boundaries in the vertebrate hindbrain. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[37] V. Chatterjee,et al. Thyroid hormone resistance syndrome manifests as an aberrant interaction between mutant T3 receptors and transcriptional corepressors. , 1997, Molecular endocrinology.
[38] R S Chaganti,et al. BCL-6, a POZ/zinc-finger protein, is a sequence-specific transcriptional repressor. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[39] K. Horwitz,et al. Nuclear receptor coactivators and corepressors. , 1996, Molecular endocrinology.
[40] S. Corey,et al. The t(5;17) variant of acute promyelocytic leukemia expresses a nucleophosmin-retinoic acid receptor fusion. , 1996, Blood.
[41] P. Pandolfi,et al. Translocation breakpoint of acute promyelocytic leukemia lies within the retinoic acid receptor alpha locus. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[42] R. Warrell,et al. Acute promyelocytic leukemia. , 1993, The New England journal of medicine.