AT1-receptor autoantibodies and uteroplacental RAS in pregnancy and pre-eclampsia

[1]  D. Diz,et al.  Activation of Local Chorionic Villi Angiotensin II Levels But Not Angiotensin (1-7) in Preeclampsia , 2008, Hypertension.

[2]  S. Ramin,et al.  Potential Roles of Angiotensin Receptor-Activating Autoantibody in the Pathophysiology of Preeclampsia , 2007, Hypertension.

[3]  M. Wolf,et al.  Agonistic Angiotensin II Type 1 Receptor Autoantibodies in Postpartum Women With a History of Preeclampsia , 2007, Hypertension.

[4]  G. Wallukat,et al.  Dysregulation of the Circulating and Tissue-Based Renin–Angiotensin System in Preeclampsia , 2007, Hypertension.

[5]  Asif Ahmed,et al.  Angiotensin II Induces Soluble fms-Like Tyrosine Kinase-1 Release via Calcineurin Signaling Pathway in Pregnancy , 2007, Circulation research.

[6]  A. Germain,et al.  Urinary vasodilator and vasoconstrictor angiotensins during menstrual cycle, pregnancy, and lactation , 2001, Endocrine.

[7]  G. Wallukat,et al.  Agonistic autoantibodies to the AT1 receptor in rat models of preeclampsia: induced by chronic reductions in uterine perfusion pressure (RUPP) and low dose TNFalpha infusion , 2006 .

[8]  M. Lindheimer,et al.  Explaining and predicting preeclampsia. , 2006, The New England journal of medicine.

[9]  G. Wallukat,et al.  Relation between circulating angiotensin II type 1 receptor agonistic autoantibodies and soluble fms-like tyrosine kinase 1 in the pathogenesis of preeclampsia. , 2006, The Journal of clinical endocrinology and metabolism.

[10]  T. Libermann,et al.  Soluble endoglin contributes to the pathogenesis of preeclampsia , 2006, Nature Medicine.

[11]  D. Shah.  The role of RAS in the pathogenesis of preeclampsia , 2006, Current hypertension reports.

[12]  James M. Roberts,et al.  Preeclampsia: recent insights. , 2005, Hypertension.

[13]  G. Wallukat,et al.  Angiotensin II Type 1 Receptor Agonistic Antibodies Reflect Fundamental Alterations in the Uteroplacental Vasculature , 2005, Hypertension.

[14]  Megumi Tanaka,et al.  Clinical evaluation of 3rd generation assay for thyrotropin receptor antibodies: the M22-biotin-based ELISA initiated by Smith. , 2005, Endocrine journal.

[15]  I. Sargent,et al.  Latest Advances in Understanding Preeclampsia , 2005, Science.

[16]  J. Wetzels,et al.  Angiotensin II type 1-receptor activating antibodies in renal-allograft rejection. , 2005, The New England journal of medicine.

[17]  G. Wallukat,et al.  Agonistic Autoantibodies to the AT1 Receptor in a Transgenic Rat Model of Preeclampsia , 2005, Hypertension.

[18]  J. Ingelfinger.  Agonistic autoantibodies and rejection of renal allografts. , 2005, The New England journal of medicine.

[19]  J. Ernerudh,et al.  Immunology of preeclampsia. , 2005, Chemical immunology and allergy.

[20]  L. Poston,et al.  Oxidative stress and preeclampsia: rationale for antioxidant clinical trials. , 2004, Hypertension.

[21]  L. Gilstrap,et al.  Antibodies From Preeclamptic Patients Stimulate Increased Intracellular Ca2+ Mobilization Through Angiotensin Receptor Activation , 2004, Circulation.

[22]  N. Mackman.  Role of tissue factor in hemostasis, thrombosis, and vascular development. , 2004, Arteriosclerosis, thrombosis, and vascular biology.

[23]  Martin J. Lohse,et al.  Direct evidence for a β1-adrenergic receptor–directed autoimmune attack as a cause of idiopathic dilated cardiomyopathy , 2004 .

[24]  R. Pijnenborg,et al.  Trophoblast and the endometrium--a workshop report. , 2004, Placenta.

[25]  K. Lim,et al.  Circulating angiogenic factors and the risk of preeclampsia. , 2004, The New England journal of medicine.

[26]  Roland Jahns,et al.  Direct evidence for a beta 1-adrenergic receptor-directed autoimmune attack as a cause of idiopathic dilated cardiomyopathy. , 2004, The Journal of clinical investigation.

[27]  T. Libermann,et al.  Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia. , 2003, The Journal of clinical investigation.

[28]  G. Wallukat,et al.  AT1 Receptor Agonistic Antibodies From Preeclamptic Patients Stimulate NADPH Oxidase , 2003, Circulation.

[29]  S. Bobst,et al.  Maternal Autoantibodies From Preeclamptic Patients Active Angiotensin Receptors on Human Trophoblast Cells , 2003, The Journal of the Society for Gynecologic Investigation: JSGI.

[30]  J. Cutler,et al.  Summary of the NHLBI Working Group on Research on Hypertension During Pregnancy , 2003, Hypertension in pregnancy.

[31]  H. Lother,et al.  Increased AT1 receptor heterodimers in preeclampsia mediate enhanced angiotensin II responsiveness , 2001, Nature Medicine.

[32]  J. Granger,et al.  Pathophysiology of hypertension during preeclampsia linking placental ischemia with endothelial dysfunction. , 2001, Hypertension.

[33]  D. Ganten,et al.  Rats transgenic for human renin and human angiotensinogen as a model for gestational hypertension. , 2000, Journal of the American Society of Nephrology : JASN.

[34]  James J. Walker,et al.  Pre-eclampsia , 2000, The Lancet.

[35]  R. Ansari,et al.  Definitive molecular evidence of renin-angiotensin system in human uterine decidual cells. , 2000, Hypertension.

[36]  G. Wallukat,et al.  Autoantibodies against the angiotensin receptor (AT1) in patients with hypertension , 2000, Journal of hypertension.

[37]  G. Wallukat,et al.  AT(1) receptor agonistic antibodies from preeclamptic patients cause vascular cells to express tissue factor. , 2000, Circulation.

[38]  J. Chirgwin,et al.  REPRODUCTIVE TISSUE RENIN GENE EXPRESSION IN PREECLAMPSIA , 2000, Hypertension in pregnancy.

[39]  M. Faas,et al.  Etiology and pathogenesis of preeclampsia: current concepts. , 1999, American journal of obstetrics and gynecology.

[40]  G. Wallukat,et al.  Patients with preeclampsia develop agonistic autoantibodies against the angiotensin AT1 receptor. , 1999, The Journal of clinical investigation.

[41]  I. Sargent,et al.  Preeclampsia: an excessive maternal inflammatory response to pregnancy. , 1999, American journal of obstetrics and gynecology.

[42]  J. Lalouel,et al.  Angiotensinogen T235 expression is elevated in decidual spiral arteries. , 1997, The Journal of clinical investigation.

[43]  R. Schmieder,et al.  Preeclampsia -- a state of sympathetic overactivity. , 1996, The New England journal of medicine.

[44]  A. Fukamizu,et al.  Hypertension Induced in Pregnant Mice by Placental Renin and Maternal Angiotensinogen , 1996, Science.

[45]  M. Faas,et al.  A new animal model for human preeclampsia: ultra-low-dose endotoxin infusion in pregnant rats. , 1994, American journal of obstetrics and gynecology.

[46]  J. Lalouel,et al.  A molecular variant of angiotensinogen associated with preeclampsia , 1993, Nature Genetics.