Polyphenol (-)-epigallocatechin gallate inhibits apoptosis induced by irradiation in human HaCaT keratinocytes.

[1]  Ji Hyun Han,et al.  Dual mechanisms of green tea extract‐induced cell survival in human epidermal keratinocytes , 2003, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[2]  Chung S. Yang,et al.  Mechanisms of cancer prevention by tea constituents. , 2003, The Journal of nutrition.

[3]  Z. Fuks,et al.  Radiation and ceramide-induced apoptosis , 2003, Oncogene.

[4]  A. Kong,et al.  Epigallocatechin-3-gallate-induced stress signals in HT-29 human colon adenocarcinoma cells. , 2003, Carcinogenesis.

[5]  H. Mukhtar,et al.  Role of p53 and NF-κB in epigallocatechin-3-gallate-induced apoptosis of LNCaP cells , 2003, Oncogene.

[6]  C. Hofmann,et al.  Green tea polyphenol epigallocatechin‐3 gallate induces apoptosis of proliferating vascular smooth muscle cells via activation of p53 , 2003, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[7]  O. Baffa,et al.  EVALUATION OF A HIGH DOSE TO A FINGER FROM A 60Co ACCIDENT , 2003, Health physics.

[8]  M. Akashi,et al.  Initial medical management of patients severely irradiated in the Tokai-mura criticality accident. , 2003, The British journal of radiology.

[9]  T. McMillan,et al.  The green tea polyphenol, epigallocatechin‐3‐gallate, protects against the oxidative cellular and genotoxic damage of UVA radiation , 2002, International journal of cancer.

[10]  J. Lewin,et al.  Inhibition of prostate carcinogenesis in TRAMP mice by oral infusion of green tea polyphenols , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[11]  G. Yang,et al.  Effect of black and green tea polyphenols on c-jun phosphorylation and H(2)O(2) production in transformed and non-transformed human bronchial cell lines: possible mechanisms of cell growth inhibition and apoptosis induction. , 2000, Carcinogenesis.

[12]  S. Nagata.  Apoptotic DNA fragmentation. , 2000, Experimental cell research.

[13]  S. Gupta,et al.  Growth inhibition, cell-cycle dysregulation, and induction of apoptosis by green tea constituent (-)-epigallocatechin-3-gallate in androgen-sensitive and androgen-insensitive human prostate carcinoma cells. , 2000, Toxicology and applied pharmacology.

[14]  T. Miyase,et al.  Importance of a pyrogallol-type structure in catechin compounds for apoptosis-inducing activity. , 2000, Phytochemistry.

[15]  G. Yang,et al.  Tea and tea polyphenols in cancer prevention. , 2000, Advances in experimental medicine and biology.

[16]  Z. Dong,et al.  Inhibition of activator protein 1 activity and cell growth by purified green tea and black tea polyphenols in H-ras-transformed cells: structure-activity relationship and mechanisms involved. , 1999, Cancer research.

[17]  J. Lin,et al.  Cancer chemoprevention by tea polyphenols through mitotic signal transduction blockade. , 1999, Biochemical pharmacology.

[18]  M. Isemura,et al.  Inhibition of Matrix Metalloproteinases by Tea Catechins and Related Polyphenols , 1999, Annals of the New York Academy of Sciences.

[19]  M. Raff,et al.  Caspase activation in the terminal differentiation of human epidermal keratinocytes , 1999, Current Biology.

[20]  H. Mukhtar,et al.  Mechanism of cancer chemopreventive activity of green Tea. , 1999, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

[21]  V. Steele,et al.  Preclinical efficacy studies of green and black tea extracts. , 1999, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

[22]  M MacFarlane,et al.  Distinct Caspase Cascades Are Initiated in Receptor-mediated and Chemical-induced Apoptosis* , 1999, The Journal of Biological Chemistry.

[23]  Hideyuki Ito,et al.  Wide distribution of [3H](-)-epigallocatechin gallate, a cancer preventive tea polyphenol, in mouse tissue. , 1998, Carcinogenesis.

[24]  Chi-Tang Ho,et al.  Green tea epigallocatechin gallate shows a pronounced growth inhibitory effect on cancerous cells but not on their normal counterparts. , 1998, Cancer letters.

[25]  S. Nagata,et al.  A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD , 1998, Nature.

[26]  R. Agarwal,et al.  Green tea constituent epigallocatechin-3-gallate and induction of apoptosis and cell cycle arrest in human carcinoma cells. , 1997, Journal of the National Cancer Institute.

[27]  M. Akashi,et al.  Endogenous production of tumour necrosis factor is required for manganese superoxide dismutase expression by irradiation in the human monocytic cell line THP-1. , 1997, The Biochemical journal.

[28]  G M Cohen,et al.  Caspases: the executioners of apoptosis. , 1997, The Biochemical journal.

[29]  C. Elmets,et al.  PROTECTION AGAINST ULTRAVIOLET‐B RADIATION‐INDUCED LOCAL and SYSTEMIC SUPPRESSION OF CONTACT HYPERSENSITIVITY and EDEMA RESPONSES IN C3H/HeN MICE BY GREEN TEA POLYPHENOLS , 1995, Photochemistry and photobiology.

[30]  M. Akashi,et al.  Irradiation Induces WAF1 Expression through a p53-independent Pathway in KG-1 Cells (*) , 1995, The Journal of Biological Chemistry.

[31]  M. Akashi,et al.  Irradiation Increases Manganese Superoxide Dismutase mRNA Levels in Human Fibroblasts. , 1995, The Journal of Biological Chemistry.

[32]  H. Fujiki,et al.  Inhibition of N-methyl-N'-nitro-N-nitrosoguanidine-induced carcinogenesis by (-)-epigallocatechin gallate in the rat glandular stomach. , 1995, Cancer research.

[33]  G. Wahl,et al.  DNA damage triggers a prolonged p53-dependent G1 arrest and long-term induction of Cip1 in normal human fibroblasts. , 1994, Genes & development.

[34]  Z. Y. Wang,et al.  Tea and cancer. , 1993, Journal of the National Cancer Institute.

[35]  R. Metcalf,et al.  p53 mutations in human immortalized epithelial cell lines. , 1993, Carcinogenesis.

[36]  S. J. Lee,et al.  Adaptive response is differently induced depending on the sensitivity to radiation-induced cell death in mouse epidermal cells , 2004, Cell Biology and Toxicology.

[37]  W. Wohlrab,et al.  Relevance of reactive oxygen species in the induction of 8-oxo-2'-deoxyguanosine in HaCaT keratinocytes. , 2002, Acta dermato-venereologica.

[38]  K. Ohtomo,et al.  Radiological findings of accidental radiation injury of the fingers: a case report. , 2001, Health physics.

[39]  Laishun Chen,et al.  Human salivary tea catechin levels and catechin esterase activities: implication in human cancer prevention studies. , 1999, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.

[40]  J. Bushman.  Green tea and cancer in humans: a review of the literature. , 1998, Nutrition and cancer.

[41]  F. Nanjo,et al.  Scavenging effects of tea catechins and their derivatives on 1,1-diphenyl-2-picrylhydrazyl radical. , 1996, Free radical biology & medicine.