Interplay between sympathetic nervous system and inflammation in aseptic loosening of hip joint replacement
暂无分享,去创建一个
M. Lamghari | D. Linhares | N. Neves | I. Alencastre | C. J. Alves | D. Vasconcelos | G. Costa | M. Ribeiro-da-Silva | R. Henrique | M. J. Oliveira
[1] Brian T. Maurer. Regression. , 2020, JAAPA : official journal of the American Academy of Physician Assistants.
[2] M. Lamghari,et al. Neuroimmune expression in hip osteoarthritis: a systematic review , 2017, BMC Musculoskeletal Disorders.
[3] I. Elenkov,et al. Neuropeptide Y as regulator of macrophage phenotype and functions: a neuroimmune CUE in atherosclerosis regression? , 2017, Atherosclerosis.
[4] M. Schellings,et al. Sema3A promotes the resolution of cardiac inflammation after myocardial infarction , 2017, Basic Research in Cardiology.
[5] M. Lamghari,et al. Immune response and innervation signatures in aseptic hip implant loosening , 2016, Journal of Translational Medicine.
[6] N. Athanasou,et al. The pathobiology and pathology of aseptic implant failure , 2016, Bone & joint research.
[7] P. Wooley,et al. Macrophage Polarization in IL-10 Treatment of Particle-Induced Inflammation and Osteolysis. , 2016, The American journal of pathology.
[8] Marco Cosentino,et al. Adrenergic regulation of innate immunity: a review , 2015, Front. Pharmacol..
[9] M. Mareel,et al. Matrix metalloproteases as maestros for the dual role of LPS- and IL-10-stimulated macrophages in cancer cell behaviour , 2015, BMC Cancer.
[10] Y. Saeki,et al. Semaphorin 4D Contributes to Rheumatoid Arthritis by Inducing Inflammatory Cytokine Production: Pathogenic and Therapeutic Implications , 2015, Arthritis & rheumatology.
[11] Y. Goshima,et al. Semaphorin 3A controls allergic and inflammatory responses in experimental allergic conjunctivitis. , 2015, International journal of ophthalmology.
[12] R. Straub,et al. The sympathetic nervous response in inflammation , 2014, Arthritis Research & Therapy.
[13] Fan Yang,et al. Novel biological strategies for treatment of wear particle-induced periprosthetic osteolysis of orthopaedic implants for joint replacement , 2014, Journal of The Royal Society Interface.
[14] J. V. Sarma,et al. Induction of M2 Regulatory Macrophages through the β2-Adrenergic Receptor with Protection during Endotoxemia and Acute Lung Injury , 2014, Journal of Innate Immunity.
[15] S. Goodman,et al. Role of macrophages in the biological reaction to wear debris from joint replacements. , 2014, Journal of long-term effects of medical implants.
[16] S. Goodman,et al. Macrophage polarization in response to wear particles in vitro , 2013, Cellular and Molecular Immunology.
[17] H. Schaible,et al. The innervation of synovium of human osteoarthritic joints in comparison with normal rat and sheep synovium. , 2013, Osteoarthritis and cartilage.
[18] Y. Goshima,et al. Decreased Semaphorin3A expression correlates with disease activity and histological features of rheumatoid arthritis , 2013, BMC Musculoskeletal Disorders.
[19] S. Goodman,et al. Revision joint replacement, wear particles, and macrophage polarization. , 2012, Acta biomaterialia.
[20] V. Sanders. The beta2-adrenergic receptor on T and B lymphocytes: Do we understand it yet? , 2012, Brain, Behavior, and Immunity.
[21] M. Dimitrijević,et al. The intriguing mission of neuropeptide Y in the immune system , 2011, Amino Acids.
[22] D. Perez,et al. Modulation of immune cell function by α(1)-adrenergic receptor activation. , 2011, Current topics in membranes.
[23] A. Catalano. The Neuroimmune Semaphorin-3A Reduces Inflammation and Progression of Experimental Autoimmune Arthritis , 2010, The Journal of Immunology.
[24] Š. Podzimek,et al. Influence of metals on cytokines production in connection with successful implantation therapy in dentistry. , 2010, Neuro endocrinology letters.
[25] W. Falk,et al. Soluble neuropilin-2, a nerve repellent receptor, is increased in rheumatoid arthritis synovium and aggravates sympathetic fiber repulsion and arthritis. , 2009, Arthritis and rheumatism.
[26] A. Major,et al. Differences in the injury/sprouting response of splenic noradrenergic nerves in Lewis rats with adjuvant-induced arthritis compared with rats treated with 6-hydroxydopamine , 2009, Brain, Behavior, and Immunity.
[27] C. Jiang,et al. Neuropeptide Y promotes TGF-β1 production in RAW264.7 cells by activating PI3K pathway via Y1 receptor , 2008, Neuroscience Bulletin.
[28] W. Falk,et al. Anti-inflammatory role of sympathetic nerves in chronic intestinal inflammation , 2008, Gut.
[29] Herbert Herzog,et al. NPY and receptors in immune and inflammatory diseases. , 2007, Current topics in medicinal chemistry.
[30] D. Nance,et al. Autonomic innervation and regulation of the immune system (1987–2007) , 2007, Brain, Behavior, and Immunity.
[31] J. Clohisy,et al. Aseptic loosening of total joint replacements: mechanisms underlying osteolysis and potential therapies , 2007, Arthritis research & therapy.
[32] J. Schölmerich,et al. Increase of sympathetic outflow measured by neuropeptide Y and decrease of the hypothalamic-pituitary-adrenal axis tone in patients with systemic lupus erythematosus and rheumatoid arthritis: another example of uncoupling of response systems , 2005, Annals of the rheumatic diseases.
[33] M. Puerto,et al. Modulation of neuropeptide Y and norepinephrine on several leucocyte functions in adult, old and very old mice , 2005, Journal of Neuroimmunology.
[34] P. Ernfors,et al. Attenuation of acute experimental colitis by preventing NPY Y1 receptor signaling. , 2005, American journal of physiology. Gastrointestinal and liver physiology.
[35] R. Straub,et al. Sympathetic neurotransmitters in joint inflammation. , 2005, Rheumatic diseases clinics of North America.
[36] A. Beck‐Sickinger,et al. Neuropeptide Y and its receptor subtypes specifically modulate rat peritoneal macrophage functions in vitro: counter regulation through Y1 and Y2/5 receptors , 2005, Regulatory Peptides.
[37] R. Geenen,et al. Elevated sympathetic nervous system activity in patients with recently diagnosed rheumatoid arthritis with active disease. , 2004, Clinical and experimental rheumatology.
[38] D. Bellinger,et al. Potential use of drugs that target neural-immune pathways in the treatment of rheumatoid arthritis and other autoimmune diseases. , 2003, Current drug targets. Inflammation and allergy.
[39] J. Schölmerich,et al. Inadequately low serum levels of steroid hormones in relation to interleukin-6 and tumor necrosis factor in untreated patients with early rheumatoid arthritis and reactive arthritis. , 2002, Arthritis and rheumatism.
[40] D. Moura,et al. Vascular adrenoceptors: an update. , 2001, Pharmacological reviews.
[41] R. Straub,et al. Involvement of the hypothalamic--pituitary--adrenal/gonadal axis and the peripheral nervous system in rheumatoid arthritis: viewpoint based on a systemic pathogenetic role. , 2001, Arthritis and rheumatism.
[42] R. Straub,et al. The loss of sympathetic nerve fibers in the synovial tissue of patients with rheumatoid arthritis is accompanied by increased norepinephrine release from synovial macrophages , 2000, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[43] H. Raspe,et al. Knee replacement surgery for osteoarthritis: effectiveness, practice variations, indications and possible determinants of utilization. , 1999, Rheumatology.
[44] C. Ranawat,et al. Indications for total hip and total knee arthroplasties. Results of orthopaedic surveys. , 1996, The Journal of arthroplasty.
[45] P. Mapp. Innervation of the synovium. , 1995, Annals of the rheumatic diseases.