Review article: anti TNF‐α induced psoriasis in patients with inflammatory bowel disease
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[1] W. Kirch,et al. Efficacy and tolerability of biologic and nonbiologic systemic treatments for moderate‐to‐severe psoriasis: meta‐analysis of randomized controlled trials , 2008, The British journal of dermatology.
[2] J. S. Henning,et al. Psoriatic skin lesions induced by tumor necrosis factor antagonist therapy: a literature review and potential mechanisms of action. , 2008, Arthritis and rheumatism.
[3] A. Alonso-Ruiz,et al. Tumor necrosis factor alpha drugs in rheumatoid arthritis: systematic review and metaanalysis of efficacy and safety , 2008, BMC musculoskeletal disorders.
[4] F. Kerdel,et al. From conventional to cutting edge: the new era of biologics in treatment of psoriasis , 2008, Dermatologic therapy.
[5] A. Triantafyllou,et al. Tumor necrosis factor-a antagonist-induced psoriasis: yet another paradox in medicine , 2008, Clinical Rheumatology.
[6] P. Tak,et al. Tumor necrosis factor antagonist mechanisms of action: a comprehensive review. , 2008, Pharmacology & therapeutics.
[7] P. Gionchetti,et al. Cutaneous Manifestations in Inflammatory Bowel Diseases: Eight Cases of Psoriasis Induced by Anti-Tumor-Necrosis-Factor Antibody Therapy , 2007, Dermatology.
[8] R. Caprilli,et al. Another paradox in Crohn's disease: new onset of psoriasis in a patient receiving tumor necrosis factor-alpha antagonist. , 2007, Inflammatory bowel diseases.
[9] A. Ishida-Yamamoto,et al. Psoriasiform and pustular eruption induced by infliximab , 2007, The Journal of dermatology.
[10] M. Bagot,et al. Psoriasis induced by tumor necrosis factor-alpha antagonist therapy: a case series. , 2007, The Journal of rheumatology.
[11] M. Martinka,et al. Psoriasis and pustular dermatitis triggered by TNF-{alpha} inhibitors in patients with rheumatologic conditions. , 2007, Archives of dermatology.
[12] U. Kajermo,et al. Infliximab can precipitate as well as worsen palmoplantar pustulosis: possible linkage to the expression of tumour necrosis factor‐α in the normal palmar eccrine sweat duct? , 2005, The British journal of dermatology.
[13] F. Nestle,et al. Defining upstream elements of psoriasis pathogenesis: an emerging role for interferon alpha. , 2005, The Journal of investigative dermatology.
[14] E. Roé,et al. Onset of flexural psoriasis during infliximab treatment for Crohn's disease , 2005, Clinical and experimental dermatology.
[15] K. Çefle,et al. Two siblings with distal pachydermodactyly , 2005, Clinical and experimental dermatology.
[16] C. Geisler,et al. Increased sensitivity to interferon-α in psoriatic T cells , 2005 .
[17] V. Pascual,et al. Cross-regulation of TNF and IFN-¿ in autoimmune diseases , 2005 .
[18] P. Jüni,et al. Increase of peripheral CXCR3 positive T lymphocytes upon treatment of RA patients with TNF-alpha inhibitors. , 2005, Rheumatology.
[19] J. Stroehlein,et al. Pustular psoriasis induced by infliximab. , 2004, Journal of drugs in dermatology : JDD.
[20] J. Gudjonsson,et al. Immunopathogenic mechanisms in psoriasis , 2004, Clinical and experimental immunology.
[21] E. Fonseca,et al. Psoriasiform Eruption Induced by Infliximab , 2004, The Annals of pharmacotherapy.
[22] A. Gottlieb,et al. Etanercept as monotherapy in patients with psoriasis. , 2003, The New England journal of medicine.
[23] A. Gröne. Keratinocytes and cytokines. , 2002, Veterinary immunology and immunopathology.
[24] A. Zinsmeister,et al. Etanercept for active Crohn's disease: a randomized, double-blind, placebo-controlled trial. , 2001, Gastroenterology.
[25] Richard W. Martin,et al. A comparison of etanercept and methotrexate in patients with early rheumatoid arthritis. , 2000, The New England journal of medicine.
[26] K. Asadullah,et al. Inhibition of Keratinocyte Apoptosis by IL-15: A New Parameter in the Pathogenesis of Psoriasis?1 , 2000, The Journal of Immunology.
[27] M. Diaz,et al. Role of NF-κB in the Apoptotic-resistant Phenotype of Keratinocytes* , 1999, The Journal of Biological Chemistry.
[28] B. Nickoloff,et al. Injection of pre-psoriatic skin with CD4+ T cells induces psoriasis. , 1999, The American journal of pathology.
[29] C. Enerbäck,et al. Evidence that HLA-Cw6 determines early onset of psoriasis, obtained using sequence-specific primers (PCR-SSP). , 1997, Acta dermato-venereologica.
[30] B. Nickoloff,et al. Dermal injection of immunocytes induces psoriasis. , 1996, The Journal of clinical investigation.
[31] Irene M. Leigh,et al. Keratins (Kl6 and Kl7) as markers of keratinocyte hyperproliferation in psoriasis in vivo and in vitro , 1995 .
[32] Gary R. Grotendorst,et al. Fibroblast function in psoriatic arthritis. II. Increased expression of beta platelet derived growth factor receptors and increased production of growth factor and cytokines. , 1994, The Journal of rheumatology.
[33] G. Weinstein,et al. Cell kinetic basis for pathophysiology of psoriasis. , 1985, The Journal of investigative dermatology.
[34] S. Gelfant. On the existence of non‐cycling germinative cells in human epidermis in vivo and cell cycle aspects of psoriasis , 1982, Cell and tissue kinetics.
[35] G. Haroske,et al. Tumor Necrosis Factor-α Inhibitor-Induced Psoriasis or Psoriasiform Exanthemata , 2008 .
[36] G. Haroske,et al. Tumor necrosis factor-alpha inhibitor-induced psoriasis or psoriasiform exanthemata: first 120 cases from the literature including a series of six new patients. , 2008, American journal of clinical dermatology.
[37] S. Berney. Psoriasis Induced by Tumor Necrosis Factor-α Antagonist Therapy: A Case Series , 2008 .
[38] M. Diaz,et al. Role of NF-kappaB in the apoptotic-resistant phenotype of keratinocytes. , 1999, The Journal of biological chemistry.
[39] H. E. Hansen,et al. Psoriasis in monozygotic twins: variations in expression in individuals with identical genetic constitution. , 1982, Acta dermato-venereologica.
[40] W. Eyler,et al. PSORIATIC ARTHRITIS. , 1965, Henry Ford Hospital medical bulletin.