Weak platelet agonists and U46619 induce apoptosis-like events in platelets, in the absence of phosphatidylserine exposure.

[1]  Joseph Loscalzo,et al.  Thrombosis and Hemorrhage , 2002 .

[2]  I. Palomo,et al.  Platelet Aging In Vivo Is Associated with Activation of Apoptotic Pathways: Studies in a Model of Suppressed Thrombopoiesis in Dogs , 2002, Thrombosis and Haemostasis.

[3]  C. Vesin,et al.  Modulation of platelet caspases and life‐span by anti‐platelet antibodies in mice , 2002, European journal of haematology.

[4]  D. Seiffert,et al.  Both the High Affinity Thrombin Receptor (GPIb-IX-V) and GPIIb/IIIa Are Implicated in Expression of Thrombin-induced Platelet Procoagulant Activity , 2001, Thrombosis and Haemostasis.

[5]  G. Tonon,et al.  Novel structurally altered P(2X1) receptor is preferentially activated by adenosine diphosphate in platelets and megakaryocytic cells. , 2001, Blood.

[6]  C. Kunzelmann-Marche,et al.  Regulation of Phosphatidylserine Transbilayer Redistribution by Store-operated Ca2+ Entry , 2001, The Journal of Biological Chemistry.

[7]  D. Kuter,et al.  The mechanism of apoptosis in human platelets during storage , 2000, Transfusion.

[8]  M. Hengartner.  The biochemistry of apoptosis , 2000, Nature.

[9]  P. Sims,et al.  Identification of three new members of the phospholipid scramblase gene family. , 2000, Biochimica et biophysica acta.

[10]  J. Savill,et al.  Constitutive Death of Platelets Leading to Scavenger Receptor-mediated Phagocytosis , 2000, The Journal of Biological Chemistry.

[11]  S. Orrenius,et al.  Phosphatidylserine exposure during apoptosis is a cell-type-specific event and does not correlate with plasma membrane phospholipid scramblase expression. , 1999, Biochemical and biophysical research communications.

[12]  I. Palomo,et al.  Platelet Aging In Vivo Is Associated with Loss of Membrane Phospholipid Asymmetry , 1999, Thrombosis and Haemostasis.

[13]  D. Green,et al.  Calpain functions in a caspase-independent manner to promote apoptosis-like events during platelet activation. , 1999, Blood.

[14]  E. Remold-O’Donnell,et al.  HEMOSTASIS , THROMBOSIS , AND VASCULAR BIOLOGY Role of Caspase in a Subset of Human Platelet Activation Responses , 1999 .

[15]  D. Kwiatkowski,et al.  Functions of gelsolin: motility, signaling, apoptosis, cancer. , 1999, Current opinion in cell biology.

[16]  G. Cohen,et al.  Dissociation of Phagocyte Recognition of Cells Undergoing Apoptosis from Other Features of the Apoptotic Program* , 1998, The Journal of Biological Chemistry.

[17]  P. Sims,et al.  Level of Expression of Phospholipid Scramblase Regulates Induced Movement of Phosphatidylserine to the Cell Surface* , 1998, The Journal of Biological Chemistry.

[18]  S. Orrenius,et al.  Alterations in Bcl‐2/Bax protein levels in platelets form part of an ionomycin‐induced process that resembles apoptosis , 1997, British journal of haematology.

[19]  V. Fadok,et al.  Appearance of Phosphatidylserine on Apoptotic Cells Requires Calcium-mediated Nonspecific Flip-Flop and Is Enhanced by Loss of the Aminophospholipid Translocase* , 1997, The Journal of Biological Chemistry.

[20]  Jay X. Tang,et al.  Caspase-3-generated fragment of gelsolin: effector of morphological change in apoptosis. , 1997, Science.

[21]  A. Schroit,et al.  Pathophysiologic implications of membrane phospholipid asymmetry in blood cells. , 1997, Blood.

[22]  Seamus J. Martin,et al.  Phosphatidylserine Externalization during CD95-induced Apoptosis of Cells and Cytoplasts Requires ICE/CED-3 Protease Activity* , 1996, The Journal of Biological Chemistry.

[23]  M. Raff,et al.  Constitutive expression of the machinery for programmed cell death , 1996, The Journal of cell biology.

[24]  N. Takeyama,et al.  Hyperammonemia inhibits platelet aggregation in rats. , 1996, Thrombosis research.

[25]  K. Schulze-Osthoff,et al.  Cell nucleus and DNA fragmentation are not required for apoptosis , 1994, The Journal of cell biology.

[26]  C. Reutelingsperger,et al.  Expression on B Cells Undergoing Apoptosis Annexin V for Flow Cytometric Detection of Phosphatidylserine , 2022 .

[27]  M. Raff,et al.  Programmed cell death and Bcl‐2 protection in the absence of a nucleus. , 1994, The EMBO journal.

[28]  A. Schroit,et al.  Loss of membrane phospholipid asymmetry in platelets and red cells may be associated with calcium-induced shedding of plasma membrane and inhibition of aminophospholipid translocase. , 1990, Biochimica et biophysica acta.

[29]  P. Comfurius,et al.  Changes in membrane phospholipid distribution during platelet activation. , 1983, Biochimica et biophysica acta.

[30]  H. Hemker,et al.  Generation of prothrombin-converting activity and the exposure of phosphatidylserine at the outer surface of platelets. , 1982, European journal of biochemistry.