Pregnancy swimming causes short- and long-term neuroprotection against hypoxia–ischemia in very immature rats

[1]  Mette Jensen Stochkendahl,et al.  Effectiveness and Economic Evaluation of Chiropractic Care for the Treatment of Low Back Pain: A Systematic Review of Pragmatic Studies , 2016, PloS one.

[2]  R. Lent,et al.  Maternal Exercise during Pregnancy Increases BDNF Levels and Cell Numbers in the Hippocampal Formation but Not in the Cerebral Cortex of Adult Rat Offspring , 2016, PloS one.

[3]  C. Matté,et al.  Effect of maternal exercise on biochemical parameters in rats submitted to neonatal hypoxia-ischemia , 2015, Brain Research.

[4]  T. Bale,et al.  Epigenetic and transgenerational reprogramming of brain development , 2015, Nature Reviews Neuroscience.

[5]  C. Netto,et al.  Sexual dimorphism and brain lateralization impact behavioral and histological outcomes following hypoxia–ischemia in P3 and P7 rats , 2015, Neuroscience.

[6]  Amanda L. Smith,et al.  Sex differences in behavioral outcome following neonatal hypoxia ischemia: Insights from a clinical meta-analysis and a rodent model of induced hypoxic ischemic brain injury , 2014, Experimental Neurology.

[7]  F. Northington,et al.  Sexual dimorphism in BDNF signaling after neonatal hypoxia–ischemia and treatment with necrostatin-1 , 2014, Neuroscience.

[8]  Darran Yates,et al.  Sensory neurons: The sense in reprogramming , 2014, Nature Reviews Neuroscience.

[9]  G. Leipnitz,et al.  Evidences that maternal swimming exercise improves antioxidant defenses and induces mitochondrial biogenesis in the brain of young Wistar rats , 2013, Neuroscience.

[10]  C. Netto,et al.  Early hypoxia–ischemia causes hemisphere and sex-dependent cognitive impairment and histological damage , 2013, Neuroscience.

[11]  A. Wyse,et al.  Are the consequences of neonatal hypoxia–ischemia dependent on animals' sex and brain lateralization? , 2013, Brain Research.

[12]  Yun Wang,et al.  Changes of Inflammatory Cytokines and Neurotrophins Emphasized Their Roles in Hypoxic–Ischemic Brain Damage , 2013, The International journal of neuroscience.

[13]  V. Lemos,et al.  Swim training attenuates oxidative damage and promotes neuroprotection in cerebral cortical slices submitted to oxygen glucose deprivation , 2012, Journal of neurochemistry.

[14]  M. Safari,et al.  Prenatal exposure to maternal voluntary exercise during pregnancy provides protection against mild chronic postnatal hypoxia in rat offspring. , 2012, Pakistan journal of pharmaceutical sciences.

[15]  M. Huntsman,et al.  Chronic Perinatal Hypoxia Reduces Glutamate–Aspartate Transporter Function in Astrocytes through the Janus Kinase/Signal Transducer and Activator of Transcription Pathway , 2011, The Journal of Neuroscience.

[16]  E. Hutchinson,et al.  Chronic Neurological Deficits in Mice after Perinatal Hypoxia and Ischemia Correlate with Hemispheric Tissue Loss and White Matter Injury Detected by MRI , 2011, Developmental Neuroscience.

[17]  E. Huang,et al.  Towards improved animal models of neonatal white matter injury associated with cerebral palsy , 2010, Disease Models & Mechanisms.

[18]  Dezhi Tian,et al.  Low-dose cardiotonic steroids increase sodium–potassium ATPase activity that protects hippocampal slice cultures from experimental ischemia , 2010, Neuroscience Letters.

[19]  J. Volpe,et al.  The encephalopathy of prematurity--brain injury and impaired brain development inextricably intertwined. , 2009, Seminars in pediatric neurology.

[20]  M. Johnston,et al.  Hypoxic-ischemic encephalopathy in the term infant. , 2009, Clinics in perinatology.

[21]  P. Cheung,et al.  Long-term cognitive impairment and myelination deficiency in a rat model of perinatal hypoxic-ischemic brain injury , 2009, Brain Research.

[22]  K. Burke,et al.  Growth deficits in a postnatal day 3 rat model of hypoxic-ischemic brain injury , 2009, Behavioural Brain Research.

[23]  Protein kinase M zeta regulation of Na/K ATPase: A persistent neuroprotective mechanism of ischemic preconditioning in hippocampal slice cultures , 2008, Brain Research.

[24]  M. Safari,et al.  Serotonergic and noradrenergic lesions suppress the enhancing effect of maternal exercise during pregnancy on learning and memory in rat pups , 2008, Neuroscience.

[25]  J. Volpe,et al.  Pathogenesis of cerebral white matter injury of prematurity , 2007, Archives of Disease in Childhood Fetal and Neonatal Edition.

[26]  Samantha Johnson,et al.  Cognitive and behavioural outcomes following very preterm birth. , 2007, Seminars in fetal & neonatal medicine.

[27]  Hong Kim,et al.  Maternal swimming during pregnancy enhances short-term memory and neurogenesis in the hippocampus of rat pups , 2006, Brain and Development.

[28]  P. Gluckman,et al.  Distinctive Neuropathologic Alterations in the Deep Layers of the Parietal Cortex after Moderate Ischemic-Hypoxic Injury in the P3 Immature Rat Brain , 2005, Pediatric Research.

[29]  A. Tamas,et al.  Neurological reflexes and early motor behavior in rats subjected to neonatal hypoxic–ischemic injury , 2005, Behavioural Brain Research.

[30]  Dong Hyun Kim,et al.  BDNF protects neurons following injury by modulation of caspase activity , 2005, Neurocritical care.

[31]  C. Netto,et al.  Preconditioning Prevents the Inhibition of Na+,K+-ATPase Activity after Brain Ischemia , 2000, Neurochemical Research.

[32]  N. Kotchabhakdi,et al.  Exercise during pregnancy increases hippocampal brain-derived neurotrophic factor mRNA expression and spatial learning in neonatal rat pup , 2003, Neuroscience Letters.

[33]  J. Gingrich,et al.  Brain injury and neurofunctional deficit in neonatal mice with hypoxic-ischemic encephalopathy , 2003, Behavioural Brain Research.

[34]  S. Yu.  Na(+), K(+)-ATPase: the new face of an old player in pathogenesis and apoptotic/hybrid cell death. , 2003, Biochemical pharmacology.

[35]  P. Gluckman,et al.  Selective Cortical Alteration after Hypoxic-Ischemic Injury in the Very Immature Rat Brain , 2003, Pediatric Research.

[36]  V. Katz.  Exercise in Water During Pregnancy , 2003, Clinical obstetrics and gynecology.

[37]  K. Nicolay,et al.  In Vivo Excitotoxicity Induced by Ouabain, a Na+/K+-ATPase Inhibitor , 2003, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[38]  D. Holtzman,et al.  BDNF Protects the Neonatal Brain from Hypoxic-Ischemic InjuryIn Vivo via the ERK Pathway , 2000, The Journal of Neuroscience.

[39]  A. Hamberger,et al.  The neurotoxicity of ouabain, a sodium-potassium ATPase inhibitor, in the rat hippocampus , 1990, Neuroscience Letters.

[40]  B. Stanfield,et al.  The recovery of forelimb-placing behavior in rats with neonatal unilateral cortical damage involves the remaining hemisphere , 1990, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[41]  T. Grisar.  Glial and neuronal Na+‐K+ pump in epilepsy , 1984, Annals of neurology.