Autoamplification of NFATc1 expression determines its essential role in bone homeostasis
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Tak W. Mak | Hiroshi Takayanagi | Ikuo Morita | Erwin F. Wagner | Edgar Serfling | E. Wagner | T. Mak | H. Nishina | H. Takayanagi | I. Morita | E. Serfling | T. Usami | Kojiro Sato | H. Yoshida | M. Asagiri | Masataka Asagiri | Takako Usami | Hiroshi Nishina | Hiroki Yoshida | Kojiro Sato | Sae Ochi | S. Ochi
[1] Tak W. Mak,et al. Role of the NF-ATc transcription factor in morphogenesis of cardiac valves and septum , 1998, Nature.
[2] A. Kerstan,et al. Alternative polyadenylation events contribute to the induction of NF-ATc in effector T cells. , 1999, Immunity.
[3] L. Glimcher,et al. NFATc1 and NFATc2 together control both T and B cell activation and differentiation. , 2001, Immunity.
[4] Hiroshi Takayanagi,et al. Induction and activation of the transcription factor NFATc1 (NFAT2) integrate RANKL signaling in terminal differentiation of osteoclasts. , 2002, Developmental cell.
[5] Y. Nogi,et al. Essential Role of p38 Mitogen-activated Protein Kinase in Cathepsin K Gene Expression during Osteoclastogenesis through Association of NFATc1 and PU.1* , 2004, Journal of Biological Chemistry.
[6] J. D. Engel,et al. GATA factor transgenes under GATA-1 locus control rescue germline GATA-1 mutant deficiencies. , 2000, Blood.
[7] Hiroshi Takayanagi,et al. Mechanistic insight into osteoclast differentiation in osteoimmunology , 2005, Journal of Molecular Medicine.
[8] Lin Chen,et al. Transcriptional regulation by calcium, calcineurin, and NFAT. , 2003, Genes & development.
[9] Daryl J. Thomas,et al. NFATc3, a Lymphoid-specific NFATc Family Member That Is Calcium-regulated and Exhibits Distinct DNA Binding Specificity (*) , 1995, The Journal of Biological Chemistry.
[10] T. Kodama,et al. NFAT and Osterix cooperatively regulate bone formation , 2005, Nature Medicine.
[11] V. Stewart,et al. RAG-2-deficient mice lack mature lymphocytes owing to inability to initiate V(D)J rearrangement , 1992, Cell.
[12] Taichiro Tomida,et al. NFAT functions as a working memory of Ca2+ signals in decoding Ca2+ oscillation , 2003, The EMBO journal.
[13] E. Wagner,et al. Bone and haematopoietic defects in mice lacking c-fos , 1992, Nature.
[14] Susumu Tonegawa,et al. RAG-1-deficient mice have no mature B and T lymphocytes , 1992, Cell.
[15] M. Glimcher,et al. The Nuclear Factor of Activated T Cells (Nfat) Transcription Factor Nfatp (Nfatc2) Is a Repressor of Chondrogenesis , 2000, The Journal of experimental medicine.
[16] S. Teitelbaum,et al. Genetic regulation of osteoclast development and function , 2003, Nature Reviews Genetics.
[17] G. Crabtree,et al. The transcription factor NF-ATc1 regulates lymphocyte proliferation and Th2 cytokine production. , 1998, Immunity.
[18] Kazuo Umezawa,et al. Inhibition of RANKL‐Induced Osteoclastogenesis by (−)‐DHMEQ, a Novel NF‐κB Inhibitor, Through Downregulation of NFATc1 , 2004 .
[19] F. Berberich-Siebelt,et al. Autoregulation of NFATc1/A expression facilitates effector T cells to escape from rapid apoptosis. , 2002, Immunity.
[20] H. Takayanagi,et al. Contribution of Nuclear Factor of Activated T Cells c1 to the Transcriptional Control of Immunoreceptor Osteoclast-associated Receptor but Not Triggering Receptor Expressed by Myeloid Cells-2 during Osteoclastogenesis* , 2005, Journal of Biological Chemistry.
[21] J. Penninger,et al. RANK-L and RANK: T cells, bone loss, and mammalian evolution. , 2002, Annual review of immunology.
[22] A Radbruch,et al. Stat6-independent GATA-3 autoactivation directs IL-4-independent Th2 development and commitment. , 2000, Immunity.
[23] Kozo Nakamura,et al. RANKL maintains bone homeostasis through c-Fos-dependent induction of interferon-β , 2002, Nature.
[24] H. Aburatani,et al. Nuclear Factor of Activated T-cells (NFAT) Rescues Osteoclastogenesis in Precursors Lacking c-Fos* , 2004, Journal of Biological Chemistry.
[25] I. Graef,et al. Evolutionary relationships among Rel domains indicate functional diversification by recombination , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[26] E. Wagner,et al. c-Fos: a key regulator of osteoclast-macrophage lineage determination and bone remodeling. , 1994, Science.
[27] G. Crabtree,et al. NFAT Signaling Choreographing the Social Lives of Cells , 2002, Cell.
[28] V. Stewart,et al. RAG-2-deficient blastocyst complementation: an assay of gene function in lymphocyte development. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[29] P. Robson,et al. Regulation of the Murine Nfatc1 Gene by NFATc2* , 2002, The Journal of Biological Chemistry.
[30] Sakae Tanaka,et al. Critical roles of c-Jun signaling in regulation of NFAT family and RANKL-regulated osteoclast differentiation. , 2004, The Journal of clinical investigation.
[31] Hiroshi Takayanagi,et al. T-cell-mediated regulation of osteoclastogenesis by signalling cross-talk between RANKL and IFN-γ , 2000, Nature.
[32] L. Glimcher,et al. Hyperproliferation and dysregulation of IL-4 expression in NF-ATp-deficient mice. , 1996, Immunity.
[33] T. Taniguchi,et al. Costimulatory signals mediated by the ITAM motif cooperate with RANKL for bone homeostasis , 2004, Nature.
[34] Michael J. Grusby,et al. The transcription factor NF-ATc is essential for cardiac valve formation , 1998, Nature.