Genetic polymorphisms of the RGS4 and dorsolateral prefrontal cortex morphometry among first episode schizophrenia patients
暂无分享,去创建一个
M. Keshavan | M. Talkowski | K. Prasad | V. Nimgaonkar | K. Chowdari | D A Lewis | K M R Prasad | K V Chowdari | V L Nimgaonkar | M E Talkowski | M S Keshavan | D. Lewis
[1] Pat Levitt,et al. Molecular Characterization of Schizophrenia Viewed by Microarray Analysis of Gene Expression in Prefrontal Cortex , 2000, Neuron.
[2] Philip D. Harvey,et al. Cognitive deficits in schizophrenia. , 1993, The Psychiatric clinics of North America.
[3] H. Groenewegen,et al. Detailed localization of regulator of G protein signaling 2 messenger ribonucleic acid and protein in the rat brain , 2002, Neuroscience.
[4] W. Honer,et al. Location of a major susceptibility locus for familial schizophrenia on chromosome 1q21-q22. , 2000, Science.
[5] S. Olson,et al. Third and lateral ventricular volumes in schizophrenia: support for progressive enlargement of both structures. , 1990, Psychopharmacology bulletin.
[6] J. Coyle,et al. Glutamatergic neurotransmission involves structural and clinical deficits of schizophrenia , 1998, Biological Psychiatry.
[7] M. Farquhar,et al. The regulator of G protein signaling family. , 2000, Annual review of pharmacology and toxicology.
[8] R. McCarley,et al. A review of MRI findings in schizophrenia , 2001, Schizophrenia Research.
[9] R. Straub,et al. Genetic variation in the 6p22.3 gene DTNBP1, the human ortholog of the mouse dysbindin gene, is associated with schizophrenia. , 2002, American journal of human genetics.
[10] David A Lewis,et al. Schizophrenia as a disorder of neurodevelopment. , 2002, Annual review of neuroscience.
[11] J. Cohen,et al. Selective deficits in prefrontal cortex function in medication-naive patients with schizophrenia. , 2001, Archives of general psychiatry.
[12] Margot Albus,et al. Support for association of schizophrenia with genetic variation in the 6p22.3 gene, dysbindin, in sib-pair families with linkage and in an additional sample of triad families. , 2003, American journal of human genetics.
[13] M. Casanova,et al. Functional and anatomical aspects of prefrontal pathology in schizophrenia. , 1997, Schizophrenia bulletin.
[14] P. Goldman-Rakic,et al. The reduced neuropil hypothesis: a circuit based model of schizophrenia , 1999, Biological Psychiatry.
[15] N. Minshew,et al. Prefrontal membrane phospholipid metabolism of child and adolescent offspring at risk for schizophrenia or schizoaffective disorder: an in vivo 31P MRS study , 2003, Molecular Psychiatry.
[16] J. Pierri,et al. Alterations in chandelier neuron axon terminals in the prefrontal cortex of schizophrenic subjects. , 1999, The American journal of psychiatry.
[17] J. Beckmann,et al. A highly significant association between a COMT haplotype and schizophrenia. , 2002, American journal of human genetics.
[18] K. Ranga R. Krishnan,et al. A comparison of stereology and segmentation techniques for volumetric measurements of lateral ventricles in magnetic resonance imaging , 1995, Psychiatry Research: Neuroimaging.
[19] Michael Gill,et al. Confirming RGS4 as a susceptibility gene for schizophrenia , 2004, American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics.
[20] B. Devlin,et al. Association and linkage analyses of RGS4 polymorphisms in schizophrenia. , 2002, Human molecular genetics.
[21] J. Kleinman,et al. Abnormal excitatory neurotransmitter metabolism in schizophrenic brains. , 1995, Archives of general psychiatry.
[22] D. Kupfer,et al. Biological correlates of slow wave sleep deficits in functional psychoses: 31P-magnetic resonance spectroscopy , 1995, Psychiatry Research.
[23] D. Weinberger,et al. Physiologic dysfunction of dorsolateral prefrontal cortex in schizophrenia. I. Regional cerebral blood flow evidence. , 1986, Archives of general psychiatry.
[24] Michael C O'Donovan,et al. Support for RGS4 as a susceptibility gene for schizophrenia , 2004, Biological Psychiatry.
[25] S. Faraone,et al. Association between a functional catechol O-methyltransferase gene polymorphism and schizophrenia: meta-analysis of case-control and family-based studies. , 2003, The American journal of psychiatry.
[26] P. Goldman-Rakic,et al. Abnormally high neuronal density in the schizophrenic cortex. A morphometric analysis of prefrontal area 9 and occipital area 17. , 1995, Archives of general psychiatry.
[27] M. Keshavan,et al. Alterations in brain high-energy phosphate and membrane phospholipid metabolism in first-episode, drug-naive schizophrenics. A pilot study of the dorsal prefrontal cortex by in vivo phosphorus 31 nuclear magnetic resonance spectroscopy. , 1991, Archives of general psychiatry.
[28] D. Lewis,et al. Disease-specific changes in regulator of G-protein signaling 4 (RGS4) expression in schizophrenia , 2001, Molecular Psychiatry.
[29] N. Minshew,et al. Prefrontal membrane phospholipid metabolism of child and adolescent offspring at risk for schizophrenia or schizoaffective disorder: an in vivo 31P MRS study , 2003, Molecular Psychiatry.
[30] J. Sweeney,et al. Thalamic volumes in patients with first-episode schizophrenia. , 2001, The American journal of psychiatry.
[31] M. Keshavan,et al. Changes in caudate volume with neuroleptic treatment , 1994, The Lancet.
[32] J. Coyle,et al. Glutamatergic mechanisms in schizophrenia. , 2003, Annual review of pharmacology and toxicology.
[33] Daniel Auerbach,et al. Identification of novel ErbB3-interacting factors using the split-ubiquitin membrane yeast two-hybrid system. , 2003, Genome research.
[34] Paul J. Harrison,et al. For Personal Use. Only Reproduce with Permission from the Lancet Publishing Group. Genes for Schizophrenia? Recent Findings and Their Pathophysiological Implications , 2022 .