The 5′-flanking cis-acting elements of the human ɛ-globin gene associates with the nuclear matrix and binds to the nuclear matrix proteins

[1]  R. Qian,et al.  Trans-acting factors from the human fetal liver binding to the human ɛ-globin gene silencer , 1997, Cell Research.

[2]  R. Qian,et al.  Studies on DNA-protein interactions in the upstream regulatory region of the human ɛ-globin gene promoter1 , 1996, Cell Research.

[3]  B. Weintraub,et al.  A soluble transcription factor, Oct-1, is also found in the insoluble nuclear matrix and possesses silencing activity in its alanine-rich domain , 1996, Molecular and cellular biology.

[4]  R. Getzenberg,et al.  Association of transcription factors with the nuclear matrix , 1996, Journal of cellular biochemistry.

[5]  J. Hi.  Studies on DNA-protein interactions in the upstream regulatory region of the human ε-globin gene promoter , 1996 .

[6]  R. Scheuermann,et al.  Mutually exclusive interaction of a novel matrix attachment region binding protein and the NF-muNR enhancer repressor. Implications for regulation of immunoglobulin heavy chain expression. , 1995, The Journal of biological chemistry.

[7]  刘建华.  α-Al_2O_3晶体中3d~2离子基态能级零场分裂的S-S耦合 , 1995 .

[8]  S. Jane,et al.  The regulatory element 3' to the A gamma-globin gene binds to the nuclear matrix and interacts with special A-T-rich binding protein 1 (SATB1), an SAR/MAR-associating region DNA binding protein. , 1994, Blood.

[9]  R. Getzenberg,et al.  Nuclear matrix and the regulation of gene expression: Tissue specificity , 1994, Journal of cellular biochemistry.

[10]  M. Dyer,et al.  Developmental regulation of the human embryonic beta-like globin gene is mediated by synergistic interactions among multiple tissue- and stage-specific elements , 1993, Molecular and cellular biology.

[11]  C. Mendelson,et al.  Identification of enhancers in the 5'-flanking region of the rabbit surfactant protein A (SP-A) gene and characterization of their binding proteins. , 1993, The Journal of biological chemistry.

[12]  G. Stein,et al.  Nuclear matrix association of multiple sequence-specific DNA binding activities related to SP-1, ATF, CCAAT, C/EBP, OCT-1, and AP-1. , 1993, Biochemistry.

[13]  Y. Kohwi,et al.  A tissue-specific MAR SAR DNA-binding protein with unusual binding site recognition , 1992, Cell.

[14]  U. K. Laemmli,et al.  Scaffold-associated regions: cis-acting determinants of chromatin structural loops and functional domains. , 1992, Current opinion in genetics & development.

[15]  W. Garrard,et al.  Immunoglobulin kappa gene expression after stable integration. I. Role of the intronic MAR and enhancer in plasmacytoma cells. , 1989, The Journal of biological chemistry.

[16]  A. E. Sippel,et al.  A nuclear DNA attachment element mediates elevated and position-independent gene activity , 1989, Nature.

[17]  A. Schechter,et al.  Identification of a transcriptional silencer in the 5'-flanking region of the human epsilon-globin gene. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[18]  A. Brand,et al.  RAP-1 factor is necessary for DNA loop formation in vitro at the silent mating type locus HML , 1989, Cell.

[19]  D. Higgs,et al.  Nuclear scaffold attachment sites in the human globin gene complexes. , 1988, The EMBO journal.

[20]  C. Noguchi,et al.  In vitro differential expression of human globin genes. , 1988, The Journal of biological chemistry.

[21]  S. Penman,et al.  Nuclear matrix proteins reflect cell type of origin in cultured human cells. , 1988, Proceedings of the National Academy of Sciences of the United States of America.

[22]  P. Cockerill,et al.  Chromosomal loop anchorage of the kappa immunoglobulin gene occurs next to the enhancer in a region containing topoisomerase II sites , 1986, Cell.

[23]  A. Varshavsky,et al.  A protein binds to a satellite DNA repeat at three specific sites that would be brought into mutual proximity by DNA folding in the nucleosome , 1984, Cell.

[24]  T. Papayannopoulou,et al.  HEL cells: a new human erythroleukemia cell line with spontaneous and induced globin expression. , 1982, Science.