Expression of Interferon Lambda 4 Is Associated with Reduced Proliferation and Increased Cell Death in Human Hepatic Cells
暂无分享,去创建一个
L. Prokunina-Olsson | Wei Tang | O. Onabajo | A. Paquin | P. Porter-Gill | Luyang Liu | N. Brand | N. Rao
[1] S. Kakinuma,et al. Impaired induction of interleukin 28B and expression of interferon λ 4 associated with nonresponse to interferon‐based therapy in chronic hepatitis C , 2015, Journal of gastroenterology and hepatology.
[2] T. O'Brien,et al. Reply: Subgroup Differences in Response to 8 Weeks of Ledipasvir/Sofosbuvir for Chronic Hepatitis C , 2015, Open forum infectious diseases.
[3] V. Wong,et al. Interferon-λ rs12979860 genotype and liver fibrosis in viral and non-viral chronic liver disease , 2015, Nature Communications.
[4] D. Goldstein,et al. Interferon-λ4 is a cell-autonomous type III interferon associated with pre-treatment hepatitis C virus burden. , 2015, Virology.
[5] Z. Kutalik,et al. Correction: Corrigendum: Reduced IFNλ4 activity is associated with improved HCV clearance and reduced expression of interferon-stimulated genes , 2014, Nature Communications.
[6] D. Booth,et al. Interferon- l rs12979860 genotype and liver fibrosis in viral and non-viral chronic liver disease , 2015 .
[7] Z. Kutalik,et al. Reduced IFNλ4 activity is associated with improved HCV clearance and reduced expression of interferon-stimulated genes , 2014, Nature Communications.
[8] T. O'Brien,et al. Subgroup Differences in Response to 8 Weeks of Ledipasvir/Sofosbuvir for Chronic Hepatitis C , 2014, Open forum infectious diseases.
[9] L. Prokunina-Olsson,et al. IFN-λ4: the paradoxical new member of the interferon lambda family. , 2014, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.
[10] M. Dennis,et al. Selection on a Variant Associated with Improved Viral Clearance Drives Local, Adaptive Pseudogenization of Interferon Lambda 4 (IFNL4) , 2014, PLoS genetics.
[11] L. Prokunina-Olsson,et al. Endogenous intrahepatic IFNs and association with IFN-free HCV treatment outcome. , 2014, The Journal of clinical investigation.
[12] L. Prokunina-Olsson,et al. IFNL4-ΔG genotype is associated with slower viral clearance in hepatitis C, genotype-1 patients treated with sofosbuvir and ribavirin. , 2014, The Journal of infectious diseases.
[13] Y. Maehara,et al. Interferon‐lambda4 genetic polymorphism is associated with the therapy response for hepatitis C virus recurrence after a living donor liver transplant , 2014, Journal of viral hepatitis.
[14] C. Rice,et al. Interferon lambda alleles predict innate antiviral immune responses and hepatitis C virus permissiveness. , 2014, Cell host & microbe.
[15] L. Prokunina-Olsson,et al. Association of the IFNL4-ΔG Allele With Impaired Spontaneous Clearance of Hepatitis C Virus. , 2014, The Journal of infectious diseases.
[16] Brien,et al. Association of the IFNL 4-Δ G Allele With Impaired Spontaneous Clearance of Hepatitis C Virus , 2014 .
[17] M. Imamura,et al. Predictive value of the IFNL4 polymorphism on outcome of telaprevir, peginterferon, and ribavirin therapy for older patients with genotype 1b chronic hepatitis C , 2014, Journal of Gastroenterology.
[18] U. Spengler,et al. Interferon-λ4 (IFNL4) Transcript Expression in Human Liver Tissue Samples , 2013, PloS one.
[19] T. Pietschmann,et al. Interferon lambda 4 signals via the IFNλ receptor to regulate antiviral activity against HCV and coronaviruses , 2013, The EMBO journal.
[20] T. Liang,et al. Current and future therapies for hepatitis C virus infection. , 2013, The New England journal of medicine.
[21] A. Flaxman,et al. Global epidemiology of hepatitis C virus infection: New estimates of age‐specific antibody to HCV seroprevalence , 2013, Hepatology.
[22] Barbara Rehermann,et al. A variant upstream of IFNL3 (IL28B) creating a new interferon gene IFNL4 is associated with impaired clearance of hepatitis C virus , 2013, Nature Genetics.
[23] F. Marincola,et al. HCV RNA levels in a multiethnic cohort of injection drug users: Human genetic, viral and demographic associations , 2012, Hepatology.
[24] R A Knight,et al. Molecular definitions of cell death subroutines: recommendations of the Nomenclature Committee on Cell Death 2012 , 2011, Cell Death and Differentiation.
[25] L. Prokunina-Olsson,et al. A variant upstream of IFNL 3 ( IL 28 B ) creating a novel interferon gene IFNL 4 is associated with impaired clearance of hepatitis C virus , 2012 .
[26] E. Fish,et al. Interferon: current status and future prospects in cancer therapy. , 2011, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.
[27] Mingli Liu,et al. The emerging role of CXCL10 in cancer (Review). , 2011, Oncology letters.
[28] Julia E. Vogt,et al. Interferon-induced gene expression is a stronger predictor of treatment response than IL28B genotype in patients with hepatitis C. , 2011, Gastroenterology.
[29] Shanchun Guo,et al. The emerging role of CXCL 10 in cancer ( Review ) , 2011 .
[30] Jacques Fellay,et al. IL28B genotype is associated with differential expression of intrahepatic interferon‐stimulated genes in patients with chronic hepatitis C , 2010, Hepatology.
[31] G. Los,et al. Peroxisome Dynamics in Cultured Mammalian Cells , 2009, Traffic.
[32] David B. Goldstein,et al. Genetic variation in IL28B and spontaneous clearance of hepatitis C virus , 2009, Nature.
[33] Jacques Fellay,et al. Genetic variation in IL28B predicts hepatitis C treatment-induced viral clearance , 2009, Nature.
[34] A. Neumann,et al. IP‐10 predicts viral response and therapeutic outcome in difficult‐to‐treat patients with HCV genotype 1 infection , 2006, Hepatology.
[35] Peter Vandenabeele,et al. Necrosis, a well-orchestrated form of cell demise: signalling cascades, important mediators and concomitant immune response. , 2006, Biochimica et biophysica acta.
[36] J. Olynyk,et al. Antiproliferative effects of interferon alpha on hepatic progenitor cells in vitro and in vivo , 2006, Hepatology.
[37] S. Kelsen,et al. CXCR3 surface expression in human airway epithelial cells: cell cycle dependence and effect on cell proliferation. , 2006, American journal of physiology. Lung cellular and molecular physiology.
[38] J. Hayashi,et al. A comparison of the antitumor effects of interferon-α and β on human hepatocellular carcinoma cell lines , 2006 .
[39] J. Hayashi,et al. A comparison of the antitumor effects of interferon-alpha and beta on human hepatocellular carcinoma cell lines. , 2006, Cytokine.
[40] Rakesh K. Kumar,et al. Expression of the chemokine IP‐10 (CXCL10) by hepatocytes in chronic hepatitis C virus infection correlates with histological severity and lobular inflammation , 2003, Journal of leukocyte biology.
[41] J. Lippincott-Schwartz,et al. Measuring Protein Mobility by Photobleaching GFP Chimeras in Living Cells , 2003, Current protocols in cell biology.
[42] T. Taniguchi,et al. New aspects of IFN‐α/β signalling in immunity, oncogenesis and bone metabolism , 2003 .
[43] T. Taniguchi,et al. New aspects of IFN-alpha/beta signalling in immunity, oncogenesis and bone metabolism. , 2003, Cancer science.
[44] K. Honda,et al. Integration of interferon-alpha/beta signalling to p53 responses in tumour suppression and antiviral defence. , 2003, Nature.
[45] K. Schulze-Osthoff,et al. Apoptosis in hepatitis C virus infection , 2003, Cell Death and Differentiation.
[46] J. Lippincott-Schwartz,et al. Studying protein dynamics in living cells , 2001, Nature Reviews Molecular Cell Biology.
[47] A. Levine,et al. Surfing the p53 network , 2000, Nature.
[48] W. Fiers,et al. More than one way to die: apoptosis, necrosis and reactive oxygen damage , 1999, Oncogene.
[49] R. Silverman,et al. A dominant negative mutant of 2‐5A‐dependent RNase suppresses antiproliferative and antiviral effects of interferon. , 1993, The EMBO journal.
[50] K. Orita,et al. A comparison of the antitumor effects of natural human tumor necrosis factors alpha and beta: the roles of arachidonic acid metabolism and intracellular cAMP. , 1990, Japanese journal of clinical oncology.
[51] B. Williams,et al. Constitutive expression of a 2',5'-oligoadenylate synthetase cDNA results in increased antiviral activity and growth suppression. , 1989, Journal of interferon research.