Brief postnatal exposure to bisphenol A affects apoptosis and gene expression in the medial prefrontal cortex and social behavior in rats with sex specificity.

[1]  Ruoyu Sun,et al.  Prenatal Progestin Exposure-Mediated Oxytocin Suppression Contributes to Social Deficits in Mouse Offspring , 2022, Frontiers in Endocrinology.

[2]  C. Zeiss Comparative Milestones in Rodent and Human Postnatal Central Nervous System Development , 2021, Toxicologic pathology.

[3]  Y. Yanagawa,et al.  FoxG1 regulates the formation of cortical GABAergic circuit during an early postnatal critical period resulting in autism spectrum disorder-like phenotypes , 2021, Nature communications.

[4]  J. Willing,et al.  Cell death in the male and female rat medial prefrontal cortex during early postnatal development , 2021, IBRO neuroscience reports.

[5]  Ruifang Fan,et al.  Sex-specific oxidative damage effects induced by BPA and its analogs on primary hippocampal neurons attenuated by EGCG. , 2020, Chemosphere.

[6]  Zhen Yan,et al.  A standardized social preference protocol for measuring social deficits in mouse models of autism , 2020, Nature Protocols.

[7]  Ruifang Fan,et al.  Maternal exposure to environmental bisphenol A impairs the neurons in hippocampus across generations. , 2020, Toxicology.

[8]  A. Chocyk,et al.  Maternal separation disturbs postnatal development of the medial prefrontal cortex and affects the number of neurons and glial cells in adolescent rats , 2019, Neuroscience.

[9]  A. Henriksen,et al.  Transgenerational Bisphenol A causes deficits in social recognition and alters post-synaptic density genes in mice. , 2019, Endocrinology.

[10]  G. Feng,et al.  Anterior cingulate cortex dysfunction underlies social deficits in Shank3 mutant mice , 2019, Nature Neuroscience.

[11]  M. Frankfurt,et al.  Effects of adolescent Bisphenol-A exposure on memory and spine density in ovariectomized female rats: Adolescence vs adulthood , 2019, Hormones and Behavior.

[12]  J. Juraska,et al.  Perinatal High-Fat Diet and Bisphenol A: Effects on Behavior and Gene Expression in the Medial Prefrontal Cortex , 2018, Developmental Neuroscience.

[13]  Daniel G Kougias,et al.  Perinatal Exposure to an Environmentally Relevant Mixture of Phthalates Results in a Lower Number of Neurons and Synapses in the Medial Prefrontal Cortex and Decreased Cognitive Flexibility in Adult Male and Female Rats , 2018, The Journal of Neuroscience.

[14]  Tewarit Sarachana,et al.  Are endocrine disrupting compounds environmental risk factors for autism spectrum disorder? , 2017, Hormones and Behavior.

[15]  H. Hashimoto,et al.  Environmental enrichment attenuates behavioral abnormalities in valproic acid-exposed autism model mice , 2017, Behavioural Brain Research.

[16]  Bung-Nyun Kim,et al.  Prenatal and postnatal bisphenol A exposure and social impairment in 4-year-old children , 2017, Environmental Health.

[17]  B. Lanphear,et al.  Associations of Prenatal Urinary Bisphenol A Concentrations with Child Behaviors and Cognitive Abilities , 2017, Environmental health perspectives.

[18]  J. DiPietro,et al.  The gestational foundation of sex differences in development and vulnerability , 2017, Neuroscience.

[19]  Min Whan Jung,et al.  Enhanced Neuronal Activity in the Medial Prefrontal Cortex during Social Approach Behavior , 2016, The Journal of Neuroscience.

[20]  R. Weinberg,et al.  Altered mGluR5-Homer scaffolds and corticostriatal connectivity in a Shank3 complete knockout model of autism , 2016, Nature Communications.

[21]  J. Willing,et al.  Long-term effects of adolescent exposure to bisphenol A on neuron and glia number in the rat prefrontal cortex: Differences between the sexes and cell type. , 2016, Neurotoxicology.

[22]  J. Ploski,et al.  Overexpression of Homer1a in the basal and lateral amygdala impairs fear conditioning and induces an autism-like social impairment , 2016, Molecular Autism.

[23]  J. Willing,et al.  The timing of neuronal loss across adolescence in the medial prefrontal cortex of male and female rats , 2015, Neuroscience.

[24]  Mariana F. Fernández,et al.  Bisphenol A: Human exposure and neurobehavior. , 2015, Neurotoxicology.

[25]  Yuji Kajiwara,et al.  Autism-like Deficits in Shank3-Deficient Mice Are Rescued by Targeting Actin Regulators. , 2015, Cell reports.

[26]  J. Juraska,et al.  Gonadectomy before puberty increases the number of neurons and glia in the medial prefrontal cortex of female, but not male, rats. , 2015, Developmental psychobiology.

[27]  C. Rosenfeld,et al.  Bisphenol A and phthalate endocrine disruption of parental and social behaviors , 2015, Front. Neurosci..

[28]  J. Nyengaard,et al.  Corrigendum to “Long-term valproic acid exposure increases the number of neocortical neurons in the developing rat brain” [Neurosci. Lett. 580 (2014) 12–16] A possible new animal model of autism , 2015, Neuroscience Letters.

[29]  S. Swan,et al.  Prenatal bisphenol A exposure and maternally reported behavior in boys and girls. , 2014, Neurotoxicology.

[30]  S. Schantz,et al.  Early exposure to bisphenol A alters neuron and glia number in the rat prefrontal cortex of adult males, but not females , 2014, Neuroscience.

[31]  Mariana F. Fernández,et al.  Endocrine Disruptors: Time to Act , 2014, Current Environmental Health Reports.

[32]  D. Pfaff,et al.  Implications of prenatal steroid perturbations for neurodevelopment, behavior, and autism. , 2014, Endocrine reviews.

[33]  Xiaohong Li,et al.  The apoptotic perspective of autism , 2014, International Journal of Developmental Neuroscience.

[34]  V. Laudet,et al.  Estrogen‐related receptor γ is an in vivo receptor of bisphenol A , 2014, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[35]  A. Calafat,et al.  Prenatal and early childhood bisphenol A concentrations and behavior in school-aged children. , 2013, Environmental research.

[36]  F. Perera,et al.  Sex-specific epigenetic disruption and behavioral changes following low-dose in utero bisphenol A exposure , 2013, Proceedings of the National Academy of Sciences.

[37]  S. Swan,et al.  Sex specific impact of perinatal bisphenol A (BPA) exposure over a range of orally administered doses on rat hypothalamic sexual differentiation. , 2013, Neurotoxicology.

[38]  Mogens Vestergaard,et al.  Prenatal valproate exposure and risk of autism spectrum disorders and childhood autism. , 2013, JAMA.

[39]  Yuta Hara,et al.  Autism-like behaviours with transient histone hyperacetylation in mice treated prenatally with valproic acid. , 2013, The international journal of neuropsychopharmacology.

[40]  L. Vanderschuren,et al.  Within-litter variation in maternal care received by individual pups correlates with adolescent social play behavior in male rats , 2012, Physiology & Behavior.

[41]  F. Perera,et al.  Prenatal Bisphenol A Exposure and Child Behavior in an Inner-City Cohort , 2012, Environmental health perspectives.

[42]  E. Courchesne,et al.  Neuron number and size in prefrontal cortex of children with autism. , 2011, JAMA.

[43]  D. Doerge,et al.  Lactational transfer of bisphenol A in Sprague-Dawley rats. , 2010, Toxicology letters.

[44]  F. Champagne Epigenetic influence of social experiences across the lifespan. , 2010, Developmental psychobiology.

[45]  R. Hornung,et al.  Prenatal Bisphenol A Exposure and Early Childhood Behavior , 2009, Environmental health perspectives.

[46]  A. Fowden,et al.  Hormones as epigenetic signals in developmental programming , 2009, Experimental physiology.

[47]  Antonia M. Calafat,et al.  Exposure of the U.S. Population to Bisphenol A and 4-tertiary-Octylphenol: 2003–2004 , 2007, Environmental health perspectives.

[48]  Nihal Ahmad,et al.  Dose translation from animal to human studies revisited , 2007, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[49]  Y. Shimohigashi,et al.  Endocrine disruptor bisphenol A strongly binds to human estrogen-related receptor γ (ERRγ) with high constitutive activity , 2006 .

[50]  Laura N. Vandenberg,et al.  Evidence of altered brain sexual differentiation in mice exposed perinatally to low, environmentally relevant levels of bisphenol A. , 2006, Endocrinology.

[51]  Woong Sun,et al.  Adaptive roles of programmed cell death during nervous system development. , 2006, Annual review of neuroscience.

[52]  F. Dessì-Fulgheri,et al.  Bisphenol-A exposure during pregnancy and lactation affects maternal behavior in rats , 2005, Brain Research Bulletin.

[53]  F. Dessì-Fulgheri,et al.  Early exposure to a low dose of bisphenol A affects socio-sexual behavior of juvenile female rats , 2005, Brain Research Bulletin.

[54]  R. Zoeller,et al.  Bisphenol-A, an environmental contaminant that acts as a thyroid hormone receptor antagonist in vitro, increases serum thyroxine, and alters RC3/neurogranin expression in the developing rat brain. , 2005, Endocrinology.

[55]  Eric Courchesne,et al.  Localized enlargement of the frontal cortex in early autism , 2005, Biological Psychiatry.

[56]  J. Piven,et al.  Sociability and preference for social novelty in five inbred strains: an approach to assess autistic‐like behavior in mice , 2004, Genes, brain, and behavior.

[57]  J. Juraska,et al.  Ovarian hormones after postnatal day 20 reduce neuron number in the rat primary visual cortex. , 2002, Journal of neurobiology.

[58]  F. Dessì-Fulgheri,et al.  Effects of perinatal exposure to bisphenol A on play behavior of female and male juvenile rats. , 2002, Environmental health perspectives.

[59]  Guosong Liu,et al.  Regulation of Dendritic Spine Morphology and Synaptic Function by Shank and Homer , 2001, Neuron.

[60]  B. Kerr,et al.  Fetal valproate syndrome and autism: additional evidence of an association , 2001, Developmental medicine and child neurology.

[61]  J. Juraska,et al.  Androgens reduce cell death in the developing rat visual cortex. , 2000, Brain research. Developmental brain research.

[62]  D. Rice,et al.  Critical periods of vulnerability for the developing nervous system: evidence from humans and animal models. , 2000, Environmental health perspectives.

[63]  J. Corton,et al.  Interaction of Estrogenic Chemicals and Phytoestrogens with Estrogen Receptor β. , 1998, Endocrinology.

[64]  J. Sumpter,et al.  Several environmental oestrogens are also anti-androgens. , 1998, The Journal of endocrinology.

[65]  K. Grandien,et al.  Printed in U.S.A. Copyright © 1997 by The Endocrine Society Comparison of the Ligand Binding Specificity and Transcript Tissue Distribution of Estrogen Receptors � and � , 2022 .

[66]  G. Xi,et al.  Human Cortex Development: Estimates of Neuronal Numbers Indicate Major Loss Late During Gestation , 1996, Journal of neuropathology and experimental neurology.