Inhibitory effect of antidepressant drugs on contact hypersensitivity reaction

[1]  T. Honda,et al.  The role of regulatory T cells in contact hypersensitivity. , 2010, Recent Patents on Inflammation & Allergy Drug Discovery.

[2]  M. Grez,et al.  Dendritic cells lentivirally engineered to overexpress interleukin‐10 inhibit contact hypersensitivity responses, despite their partial activation induced by transduction‐associated physical stress , 2010, The journal of gene medicine.

[3]  M. Maes,et al.  Age-dependent stimulatory effect of desipramine and fluoxetine pretreatment on metastasis formation by B16F10 melanoma in male C57BL/6 mice , 2009, Pharmacological reports : PR.

[4]  M. Maes,et al.  Inhibitory effects of amantadine on the production of pro-inflammatory cytokines by stimulated in vitro human blood , 2009, Pharmacological reports : PR.

[5]  J. Koo,et al.  Evidence for IL-1 receptor blockade as a therapeutic strategy for the treatment of depression. , 2009, Current opinion in investigational drugs.

[6]  M. González-Peteiro,et al.  Effects of fluoxetine on cellular immune response in stressed mice , 2006, Neuroscience Letters.

[7]  Qiang Xu,et al.  Astilbin inhibits contact hypersensitivity through negative cytokine regulation distinct from cyclosporin A. , 2005, The Journal of allergy and clinical immunology.

[8]  P. Askenase,et al.  An Hour after Immunization Peritoneal B-1 Cells Are Activated to Migrate to Lymphoid Organs Where within 1 Day They Produce IgM Antibodies That Initiate Elicitation of Contact Sensitivity1 , 2005, The Journal of Immunology.

[9]  Qiang Xu,et al.  Aqueous extract from aerial parts of Artemisia vestita, a traditional Tibetan medicine, reduces contact sensitivity in mice by down-regulating the activation, adhesion and metalloproteinase production of T lymphocytes. , 2005, International immunopharmacology.

[10]  P. Askenase,et al.  Extravascular T-cell recruitment requires initiation begun by Valpha14+ NKT cells and B-1 B cells. , 2004, Trends in immunology.

[11]  P. Askenase,et al.  B Cell–dependent T Cell Responses , 2002, The Journal of experimental medicine.

[12]  E. Bosmans,et al.  Anti-Inflammatory Effects of Antidepressants Through Suppression of the Interferon-γ/Interleukin-10 Production Ratio , 2001, Journal of clinical psychopharmacology.

[13]  M. Maes,et al.  Prolonged desipramine treatment increases the production of interleukin-10, an anti-inflammatory cytokine, in C57BL/6 mice subjected to the chronic mild stress model of depression. , 2001, Journal of affective disorders.

[14]  M. Maes,et al.  The effect of repeated amitriptyline and desipramine administration on cytokine release in C57BL/6 mice , 2000, Psychoneuroendocrinology.

[15]  C. Albanesi,et al.  Human CD4+ T lymphocytes with remarkable regulatory functions on dendritic cells and nickel-specific Th1 immune responses. , 2000, The Journal of investigative dermatology.

[16]  E. Bosmans,et al.  Negative Immunoregulatory Effects of Antidepressants: Inhibition of Interferon-γ and Stimulation of Interleukin-10 Secretion , 1999, Neuropsychopharmacology.

[17]  Y. Kobayashi Langerhans’ cells produce type IV collagenase (MMP‐9) following epicutaneous stimulation with haptens , 1997, Immunology.

[18]  G F Gerberick,et al.  Phenotypic analysis of lymphocyte subpopulations in lymph nodes draining the ear following exposure to contact allergens and irritants. , 1996, Fundamental and applied toxicology : official journal of the Society of Toxicology.

[19]  J. Depierre,et al.  Tricyclic antidepressants inhibit IL-6, IL-1β and TNF-α release in human blood monocytes and IL-2 and interferon-γ in T cells , 1996 .

[20]  P. Askenase,et al.  Possible involvement of C5/C5a in the efferent and elicitation phases of contact sensitivity. , 1996, Journal of immunology.

[21]  M. Udey,et al.  Contact allergens and epidermal proinflammatory cytokines modulate Langerhans cell E-cadherin expression in situ. , 1996, The Journal of investigative dermatology.

[22]  A. Enk,et al.  Contact sensitivity as a model for T-cell activation in skin. , 1995, Journal of Investigative Dermatology.

[23]  P. Chambon,et al.  All-trans retinoic acid induces cellular retinol-binding protein in human skin in vivo. , 1995, The Journal of investigative dermatology.

[24]  W. Herzog,et al.  An antigen-specific DTH-initiating cell clone. Functional, phenotypical, and partial molecular characterization. , 1990, Journal of Immunology.

[25]  D. Green,et al.  Characterization of two different Ly-1+ T cell populations that mediate delayed-type hypersensitivity. , 1984, Journal of immunology.