Inhibitory effect of antidepressant drugs on contact hypersensitivity reaction
暂无分享,去创建一个
M. Maes | M. Leśkiewicz | B. Budziszewska | A. Basta-Kaim | M. Kubera | W. Lasoń | W. Ptak | K. Curzytek | M. Szczepanik | Katarzyna Marcińska | M. Majewska-Szczepanik | J. Detka | W. Duda
[1] T. Honda,et al. The role of regulatory T cells in contact hypersensitivity. , 2010, Recent Patents on Inflammation & Allergy Drug Discovery.
[2] M. Grez,et al. Dendritic cells lentivirally engineered to overexpress interleukin‐10 inhibit contact hypersensitivity responses, despite their partial activation induced by transduction‐associated physical stress , 2010, The journal of gene medicine.
[3] M. Maes,et al. Age-dependent stimulatory effect of desipramine and fluoxetine pretreatment on metastasis formation by B16F10 melanoma in male C57BL/6 mice , 2009, Pharmacological reports : PR.
[4] M. Maes,et al. Inhibitory effects of amantadine on the production of pro-inflammatory cytokines by stimulated in vitro human blood , 2009, Pharmacological reports : PR.
[5] J. Koo,et al. Evidence for IL-1 receptor blockade as a therapeutic strategy for the treatment of depression. , 2009, Current opinion in investigational drugs.
[6] M. González-Peteiro,et al. Effects of fluoxetine on cellular immune response in stressed mice , 2006, Neuroscience Letters.
[7] Qiang Xu,et al. Astilbin inhibits contact hypersensitivity through negative cytokine regulation distinct from cyclosporin A. , 2005, The Journal of allergy and clinical immunology.
[8] P. Askenase,et al. An Hour after Immunization Peritoneal B-1 Cells Are Activated to Migrate to Lymphoid Organs Where within 1 Day They Produce IgM Antibodies That Initiate Elicitation of Contact Sensitivity1 , 2005, The Journal of Immunology.
[9] Qiang Xu,et al. Aqueous extract from aerial parts of Artemisia vestita, a traditional Tibetan medicine, reduces contact sensitivity in mice by down-regulating the activation, adhesion and metalloproteinase production of T lymphocytes. , 2005, International immunopharmacology.
[10] P. Askenase,et al. Extravascular T-cell recruitment requires initiation begun by Valpha14+ NKT cells and B-1 B cells. , 2004, Trends in immunology.
[11] P. Askenase,et al. B Cell–dependent T Cell Responses , 2002, The Journal of experimental medicine.
[12] E. Bosmans,et al. Anti-Inflammatory Effects of Antidepressants Through Suppression of the Interferon-γ/Interleukin-10 Production Ratio , 2001, Journal of clinical psychopharmacology.
[13] M. Maes,et al. Prolonged desipramine treatment increases the production of interleukin-10, an anti-inflammatory cytokine, in C57BL/6 mice subjected to the chronic mild stress model of depression. , 2001, Journal of affective disorders.
[14] M. Maes,et al. The effect of repeated amitriptyline and desipramine administration on cytokine release in C57BL/6 mice , 2000, Psychoneuroendocrinology.
[15] C. Albanesi,et al. Human CD4+ T lymphocytes with remarkable regulatory functions on dendritic cells and nickel-specific Th1 immune responses. , 2000, The Journal of investigative dermatology.
[16] E. Bosmans,et al. Negative Immunoregulatory Effects of Antidepressants: Inhibition of Interferon-γ and Stimulation of Interleukin-10 Secretion , 1999, Neuropsychopharmacology.
[17] Y. Kobayashi. Langerhans’ cells produce type IV collagenase (MMP‐9) following epicutaneous stimulation with haptens , 1997, Immunology.
[18] G F Gerberick,et al. Phenotypic analysis of lymphocyte subpopulations in lymph nodes draining the ear following exposure to contact allergens and irritants. , 1996, Fundamental and applied toxicology : official journal of the Society of Toxicology.
[19] J. Depierre,et al. Tricyclic antidepressants inhibit IL-6, IL-1β and TNF-α release in human blood monocytes and IL-2 and interferon-γ in T cells , 1996 .
[20] P. Askenase,et al. Possible involvement of C5/C5a in the efferent and elicitation phases of contact sensitivity. , 1996, Journal of immunology.
[21] M. Udey,et al. Contact allergens and epidermal proinflammatory cytokines modulate Langerhans cell E-cadherin expression in situ. , 1996, The Journal of investigative dermatology.
[22] A. Enk,et al. Contact sensitivity as a model for T-cell activation in skin. , 1995, Journal of Investigative Dermatology.
[23] P. Chambon,et al. All-trans retinoic acid induces cellular retinol-binding protein in human skin in vivo. , 1995, The Journal of investigative dermatology.
[24] W. Herzog,et al. An antigen-specific DTH-initiating cell clone. Functional, phenotypical, and partial molecular characterization. , 1990, Journal of Immunology.
[25] D. Green,et al. Characterization of two different Ly-1+ T cell populations that mediate delayed-type hypersensitivity. , 1984, Journal of immunology.